Biblio

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Filters: Keyword is Disease Models, Animal  [Clear All Filters]
2022
T. June Yoo, Anti-Inflammatory Gene Therapy Improves Spatial Memory Performance in a Mouse Model of Alzheimer's Disease., J Alzheimers Dis, vol. 85, no. 3, pp. 1001-1008, 2022.
X. Sun, Suo, X., Xia, X., Yu, C., and Dou, Y., Dimethyl Fumarate is a Potential Therapeutic Option for Alzheimer's Disease., J Alzheimers Dis, vol. 85, no. 1, pp. 443-456, 2022.
D. Cutuli, Landolfo, E., Petrosini, L., and Gelfo, F., Environmental Enrichment Effects on the Brain-Derived Neurotrophic Factor Expression in Healthy Condition, Alzheimer's Disease, and Other Neurodegenerative Disorders., J Alzheimers Dis, vol. 85, no. 3, pp. 975-992, 2022.
M. Abdelhamid, Zhou, C., Ohno, K., Kuhara, T., Taslima, F., Abdullah, M., Jung, C. - G., and Michikawa, M., Probiotic Bifidobacterium breve Prevents Memory Impairment Through the Reduction of Both Amyloid-β Production and Microglia Activation in APP Knock-In Mouse., J Alzheimers Dis, vol. 85, no. 4, pp. 1555-1571, 2022.
A. Sil, Erfani, A., Lamb, N., Copland, R., Riedel, G., and Platt, B., Sex Differences in Behavior and Molecular Pathology in the 5XFAD Model., J Alzheimers Dis, vol. 85, no. 2, pp. 755-778, 2022.
K. Metsla, Kirss, S., Laks, K., Sildnik, G., Palgi, M., Palumaa, T., Tõugu, V., and Palumaa, P., α-Lipoic Acid Has the Potential to Normalize Copper Metabolism, Which Is Dysregulated in Alzheimer's Disease., J Alzheimers Dis, vol. 85, no. 2, pp. 715-728, 2022.
2018
J. L. King, Wong, A. A., and Brown, R. E., Age-Related Changes in the Spatial Frequency Threshold of Male and Female 3xTg-AD Mice Using OptoMotry., J Alzheimers Dis, vol. 62, no. 2, pp. 591-596, 2018.
D. Zádori, Veres, G., Szalárdy, L., Klivenyi, P., and Vecsei, L., Alzheimer's Disease: Recent Concepts on the Relation of Mitochondrial Disturbances, Excitotoxicity, Neuroinflammation, and Kynurenines., J Alzheimers Dis, vol. 62, no. 2, pp. 523-547, 2018.
J. A. Snir, Suchy, M., Bindseil, G. A., Kovacs, M., Chronik, B. A., Hudson, R. H. E., Pasternak, S. H., and Bartha, R., An Aspartyl Cathepsin Targeted PET Agent: Application in an Alzheimer's Disease Mouse Model., J Alzheimers Dis, vol. 61, no. 3, pp. 1241-1252, 2018.
C. M. Huber, Yee, C., May, T., Dhanala, A., and Mitchell, C. S., Cognitive Decline in Preclinical Alzheimer's Disease: Amyloid-Beta versus Tauopathy., J Alzheimers Dis, vol. 61, no. 1, pp. 265-281, 2018.
S. Gelman, Palma, J., Tombaugh, G., and Ghavami, A., Differences in Synaptic Dysfunction Between rTg4510 and APP/PS1 Mouse Models of Alzheimer's Disease., J Alzheimers Dis, vol. 61, no. 1, pp. 195-208, 2018.
Y. Fan, Luo, R., Su, L. - Y., Xiang, Q., Yu, D., Xu, L., Chen, J. - Q., Bi, R., Wu, D. - D., Zheng, P., and Yao, Y. - G., Does the Genetic Feature of the Chinese Tree Shrew (Tupaia belangeri chinensis) Support Its Potential as a Viable Model for Alzheimer's Disease Research?, J Alzheimers Dis, vol. 61, no. 3, pp. 1015-1028, 2018.
J. Paulo Alme Santos, Vizuete, A., Hansen, F., Biasibetti, R., and Gonçalves, C. - A., Early and Persistent O-GlcNAc Protein Modification in the Streptozotocin Model of Alzheimer's Disease., J Alzheimers Dis, vol. 61, no. 1, pp. 237-249, 2018.
B. Gurel, Cansev, M., Sevinc, C., Kelestemur, S., Ocalan, B., Cakir, A., Aydin, S., Kahveci, N., Ozansoy, M., Taskapilioglu, O., Ulus, I. Hakki, Başar, M. Karayel, Sahin, B., Tuzuner, M. Bora, and Baykal, A. Tarik, Early Stage Alterations in CA1 Extracellular Region Proteins Indicate Dysregulation of IL6 and Iron Homeostasis in the 5XFAD Alzheimer's Disease Mouse Model., J Alzheimers Dis, vol. 61, no. 4, pp. 1399-1410, 2018.
B. Ahlemeyer, Halupczok, S., Rodenberg-Frank, E., Valerius, K. - P., and Baumgart-Vogt, E., Endogenous Murine Amyloid-β Peptide Assembles into Aggregates in the Aged C57BL/6J Mouse Suggesting These Animals as a Model to Study Pathogenesis of Amyloid-β Plaque Formation., J Alzheimers Dis, vol. 61, no. 4, pp. 1425-1450, 2018.
E. Chiziane, Telemann, H., Krueger, M., Adler, J., Arnhold, J., Alia, A., and Flemmig, J., Free Heme and Amyloid-β: A Fatal Liaison in Alzheimer's Disease., J Alzheimers Dis, vol. 61, no. 3, pp. 963-984, 2018.
W. Zhang, Guo, Y., Li, B., Zhang, Q., Liu, J. - H., Gu, G. - J., Wang, J. - H., Bao, R. - K., Chen, Y. - J., and Xu, J. - R., GDF11 Rejuvenates Cerebrovascular Structure and Function in an Animal Model of Alzheimer's Disease., J Alzheimers Dis, vol. 62, no. 2, pp. 807-819, 2018.
J. Mehla, Lacoursiere, S., Stuart, E., McDonald, R. J., and Mohajerani, M. H., Gradual Cerebral Hypoperfusion Impairs Fear Conditioning and Object Recognition Learning and Memory in Mice: Potential Roles of Neurodegeneration and Cholinergic Dysfunction., J Alzheimers Dis, vol. 61, no. 1, pp. 283-293, 2018.
T. Saksida, Koprivica, I., Vujičić, M., Stošić-Grujičić, S., Perović, M., Kanazir, S., and Stojanović, I., Impaired IL-17 Production in Gut-Residing Immune Cells of 5xFAD Mice with Alzheimer's Disease Pathology., J Alzheimers Dis, vol. 61, no. 2, pp. 619-630, 2018.
A. Ishikawa, Tokunaga, M., Maeda, J., Minamihisamatsu, T., Shimojo, M., Takuwa, H., Ono, M., Ni, R., Hirano, S., Kuwabara, S., Ji, B., Zhang, M. - R., Aoki, I., Suhara, T., Higuchi, M., and Sahara, N., In Vivo Visualization of Tau Accumulation, Microglial Activation, and Brain Atrophy in a Mouse Model of Tauopathy rTg4510., J Alzheimers Dis, vol. 61, no. 3, pp. 1037-1052, 2018.
B. M. Law, Guest, A. L., Pullen, M. W. J., Perkinton, M. S., and Williams, R. J., Increased Foxo3a Nuclear Translocation and Activity is an Early Neuronal Response to βγ-Secretase-Mediated Processing of the Amyloid-β Protein Precursor: Utility of an AβPP-GAL4 Reporter Assay., J Alzheimers Dis, vol. 61, no. 2, pp. 673-688, 2018.
A. Arner, Rockenstein, E., Mante, M., Florio, J., Masliah, D., Salehi, B., Adame, A., Overk, C., Masliah, E., and Rissman, R. A., Increased Vulnerability of the Hippocampus in Transgenic Mice Overexpressing APP and Triple Repeat Tau., J Alzheimers Dis, vol. 61, no. 3, pp. 1201-1219, 2018.
L. Yu, Liu, Y., Jin, Y., Cao, X., Chen, J., Jin, J., Gu, Y., Bao, X., Ren, Z., Xu, Y., and Zhu, X., Lentivirus-Mediated HDAC3 Inhibition Attenuates Oxidative Stress in APPswe/PS1dE9 Mice., J Alzheimers Dis, vol. 61, no. 4, pp. 1411-1424, 2018.

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