Biblio

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2018
J. L. King, Wong, A. A., and Brown, R. E., Age-Related Changes in the Spatial Frequency Threshold of Male and Female 3xTg-AD Mice Using OptoMotry., J Alzheimers Dis, vol. 62, no. 2, pp. 591-596, 2018.
B. Ahlemeyer, Halupczok, S., Rodenberg-Frank, E., Valerius, K. - P., and Baumgart-Vogt, E., Endogenous Murine Amyloid-β Peptide Assembles into Aggregates in the Aged C57BL/6J Mouse Suggesting These Animals as a Model to Study Pathogenesis of Amyloid-β Plaque Formation., J Alzheimers Dis, vol. 61, no. 4, pp. 1425-1450, 2018.
J. Mehla, Lacoursiere, S., Stuart, E., McDonald, R. J., and Mohajerani, M. H., Gradual Cerebral Hypoperfusion Impairs Fear Conditioning and Object Recognition Learning and Memory in Mice: Potential Roles of Neurodegeneration and Cholinergic Dysfunction., J Alzheimers Dis, vol. 61, no. 1, pp. 283-293, 2018.
L. Yu, Liu, Y., Jin, Y., Cao, X., Chen, J., Jin, J., Gu, Y., Bao, X., Ren, Z., Xu, Y., and Zhu, X., Lentivirus-Mediated HDAC3 Inhibition Attenuates Oxidative Stress in APPswe/PS1dE9 Mice., J Alzheimers Dis, vol. 61, no. 4, pp. 1411-1424, 2018.
S. Beggiato, Borelli, A. Celeste, Ferraro, L., Tanganelli, S., Antonelli, T., and Tomasini, M. Cristina, Palmitoylethanolamide Blunts Amyloid-β42-Induced Astrocyte Activation and Improves Neuronal Survival in Primary Mouse Cortical Astrocyte-Neuron Co-Cultures., J Alzheimers Dis, vol. 61, no. 1, pp. 389-399, 2018.
Y. Hasegawa, Toyama, K., Uekawa, K., Ichijo, H., and Kim-Mitsuyama, S., Role of ASK1/p38 Cascade in a Mouse Model of Alzheimer's Disease and Brain Aging., J Alzheimers Dis, vol. 61, no. 1, pp. 259-263, 2018.
2016
A. Tramutola, Pupo, G., Di Domenico, F., Barone, E., Arena, A., Lanzillotta, C., Broekaart, D., Blarzino, C., Head, E., D Butterfield, A., and Perluigi, M., Activation of p53 in Down Syndrome and in the Ts65Dn Mouse Brain is Associated with a Pro-Apoptotic Phenotype., J Alzheimers Dis, vol. 52, no. 1, pp. 359-71, 2016.
Y. Shan, Wang, D. - D., Xu, Y. - X., Wang, C., Cao, L., Liu, Y. - S., and Zhu, C. - Q., Aging as a Precipitating Factor in Chronic Restraint Stress-Induced Tau Aggregation Pathology, and the Protective Effects of Rosmarinic Acid., J Alzheimers Dis, vol. 49, no. 3, pp. 829-44, 2016.
S. Eketjäll, Janson, J., Kaspersson, K., Bogstedt, A., Jeppsson, F., Fälting, J., Haeberlein, S. Budd, Kugler, A. R., Alexander, R. C., and Cebers, G., AZD3293: A Novel, Orally Active BACE1 Inhibitor with High Potency and Permeability and Markedly Slow Off-Rate Kinetics., J Alzheimers Dis, vol. 50, no. 4, pp. 1109-23, 2016.
F. Guillot, Kemppainen, S., Lavasseur, G., Miettinen, P. O., Laroche, S., Tanila, H., and Davis, S., Brain-Specific Basal and Novelty-Induced Alternations in PI3K-Akt and MAPK/ERK Signaling in a Middle-Aged AβPP/PS1 Mouse Model of Alzheimer's Disease., J Alzheimers Dis, vol. 51, no. 4, pp. 1157-73, 2016.
E. Aso, Andrés-Benito, P., Carmona, M., Maldonado, R., and Ferrer, I., Cannabinoid Receptor 2 Participates in Amyloid-β Processing in a Mouse Model of Alzheimer's Disease but Plays a Minor Role in the Therapeutic Properties of a Cannabis-Based Medicine., J Alzheimers Dis, vol. 51, no. 2, pp. 489-500, 2016.
V. Bonet-Costa, Herranz-Pérez, V., Blanco-Gandía, M. C., Mas-Bargues, C., Inglés, M., Garcia-Tarraga, P., Rodriguez-Arias, M., Miñarro, J., Borras, C., Garcia-Verdugo, J. Manuel, and Viña, J., Clearing Amyloid-β through PPARγ/ApoE Activation by Genistein is a Treatment of Experimental Alzheimer's Disease., J Alzheimers Dis, vol. 51, no. 3, pp. 701-11, 2016.
M. Bolós, Llorens-Martín, M., Jurado-Arjona, J., Hernández, F., Rábano, A., and Avila, J., Direct Evidence of Internalization of Tau by Microglia In Vitro and In Vivo., J Alzheimers Dis, vol. 50, no. 1, pp. 77-87, 2016.
Z. Rusznák, Kim, W. Scott, Hsiao, J. - H. T., Halliday, G. M., Paxinos, G., and Fu, Y. H., Early in vivo Effects of the Human Mutant Amyloid-β Protein Precursor (hAβPPSwInd) on the Mouse Olfactory Bulb., J Alzheimers Dis, vol. 49, no. 2, pp. 443-57, 2016.
A. M. Masoud, Bihaqi, S. W., Machan, J. T., Zawia, N. H., and Renehan, W. E., Early-Life Exposure to Lead (Pb) Alters the Expression of microRNA that Target Proteins Associated with Alzheimer's Disease., J Alzheimers Dis, vol. 51, no. 4, pp. 1257-64, 2016.
M. Ángel Ontiveros-Torres, Labra-Barrios, M. Luisa, Díaz-Cintra, S., Aguilar-Vázquez, A. Ruth, Moreno-Campuzano, S., Flores-Rodríguez, P., Luna-Herrera, C., Mena, R., Perry, G., Florán-Garduño, B., Luna-Muñoz, J., and Luna-Arias, J. Pedro, Fibrillar Amyloid-β Accumulation Triggers an Inflammatory Mechanism Leading to Hyperphosphorylation of the Carboxyl-Terminal End of Tau Polypeptide in the Hippocampal Formation of the 3×Tg-AD Transgenic Mouse., J Alzheimers Dis, vol. 52, no. 1, pp. 243-69, 2016.
A. S. R. Sierksma, de Nijs, L., Hoogland, G., Vanmierlo, T., van Leeuwen, F. W., Rutten, B. P. F., Steinbusch, H. W. M., Prickaerts, J., and van den Hove, D. L. A., Fluoxetine Treatment Induces Seizure Behavior and Premature Death in APPswe/PS1dE9 Mice., J Alzheimers Dis, vol. 51, no. 3, pp. 677-82, 2016.
G. Lou, Zhang, Q., Xiao, F., Xiang, Q., Su, Z., and Huang, Y., Intranasal TAT-haFGF Improves Cognition and Amyloid-β Pathology in an AβPP/PS1 Mouse Model of Alzheimer's Disease., J Alzheimers Dis, vol. 51, no. 4, pp. 985-90, 2016.
H. Xu, Perreau, V. M., Dent, K. A., Bush, A. I., Finkelstein, D. I., and Adlard, P. A., Iron Regulates Apolipoprotein E Expression and Secretion in Neurons and Astrocytes., J Alzheimers Dis, vol. 51, no. 2, pp. 471-87, 2016.
M. Rodriguez-Perdigon, Solas, M., and Ramirez, M. Javier, JNK: A Putative Link Between Insulin Signaling and VGLUT1 in Alzheimer's Disease., J Alzheimers Dis, vol. 50, no. 4, pp. 963-7, 2016.
J. Min Song, Sung, Y. Me, Nam, J. Han, Yoon, H., Chung, A., Moffat, E., Jung, M., Pak, D. T. S., Kim, J., and Hoe, H. - S., A Mercaptoacetamide-Based Class II Histone Deacetylase Inhibitor Increases Dendritic Spine Density via RasGRF1/ERK Pathway., J Alzheimers Dis, vol. 51, no. 2, pp. 591-604, 2016.
S. Min Son, Shin, H. - J., Byun, J., Kook, S. Young, Moon, M., Chang, Y. Jin, and Mook-Jung, I., Metformin Facilitates Amyloid-β Generation by β- and γ-Secretases via Autophagy Activation., J Alzheimers Dis, vol. 51, no. 4, pp. 1197-208, 2016.

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