Biblio

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2018
L. Wu, Liu, J., Feng, X., Dong, J., Qin, W., Liu, Y., Wang, J., Lu, J., Chen, K., Wang, Y., and Jia, J., 11C-CFT-PET in Presymptomatic FTDP-17: A Potential Biomarker Predicting Onset., J Alzheimers Dis, vol. 61, no. 2, pp. 613-618, 2018.
I. Piaceri, Bessi, V., Matà, S., Polito, C., Tedde, A., Berti, V., Bagnoli, S., Braccia, A., Del Mastio, M., Pignone, A. Moggi, Pupi, A., Sorbi, S., and Nacmias, B., Association of the New Variant Tyr424Asp at TBK1 Gene with Amyotrophic Lateral Sclerosis and Cognitive Decline., J Alzheimers Dis, vol. 61, no. 1, pp. 41-46, 2018.
C. Alexander, Zeithamova, D., Hsiung, G. - Y. R., Mackenzie, I. R., and Jacova, C., Decreased Prefrontal Activation during Matrix Reasoning in Predementia Progranulin Mutation Carriers., J Alzheimers Dis, vol. 62, no. 2, pp. 583-589, 2018.
S. Gelman, Palma, J., Tombaugh, G., and Ghavami, A., Differences in Synaptic Dysfunction Between rTg4510 and APP/PS1 Mouse Models of Alzheimer's Disease., J Alzheimers Dis, vol. 61, no. 1, pp. 195-208, 2018.
J. Gustavo Villamil-Ortiz, Barrera-Ocampo, A., Arias-Londoño, J. David, Villegas, A., Lopera, F., and Cardona-Gómez, G. Patricia, Differential Pattern of Phospholipid Profile in the Temporal Cortex from E280A-Familiar and Sporadic Alzheimer's Disease Brains., J Alzheimers Dis, vol. 61, no. 1, pp. 209-219, 2018.
M. Pia Giannoccaro, Bartoletti-Stella, A., Piras, S., Casalena, A., Oppi, F., Ambrosetto, G., Montagna, P., Liguori, R., Parchi, P., and Capellari, S., The First Historically Reported Italian Family with FTD/ALS Teaches a Lesson on C9orf72 RE: Clinical Heterogeneity and Oligogenic Inheritance., J Alzheimers Dis, vol. 62, no. 2, pp. 687-697, 2018.
S. Fostinelli, Ciani, M., Zanardini, R., Zanetti, O., Binetti, G., Ghidoni, R., and Benussi, L., The Heritability of Frontotemporal Lobar Degeneration: Validation of Pedigree Classification Criteria in a Northern Italy Cohort., J Alzheimers Dis, vol. 61, no. 2, pp. 753-760, 2018.
B. M. Law, Guest, A. L., Pullen, M. W. J., Perkinton, M. S., and Williams, R. J., Increased Foxo3a Nuclear Translocation and Activity is an Early Neuronal Response to βγ-Secretase-Mediated Processing of the Amyloid-β Protein Precursor: Utility of an AβPP-GAL4 Reporter Assay., J Alzheimers Dis, vol. 61, no. 2, pp. 673-688, 2018.
X. - Q. Che, Zhao, Q. - H., Huang, Y., Li, X., Ren, R. - J., Di Chen, S. -, Guo, Q. - H., and Wang, G., Mutation Screening of the CHCHD2 Gene for Alzheimer's Disease and Frontotemporal Dementia in Chinese Mainland Population., J Alzheimers Dis, vol. 61, no. 4, pp. 1283-1288, 2018.
R. Zanardini, Benussi, L., Fostinelli, S., Saraceno, C., Ciani, M., Borroni, B., Padovani, A., Binetti, G., and Ghidoni, R., Serum C-Peptide, Visfatin, Resistin, and Ghrelin are Altered in Sporadic and GRN-Associated Frontotemporal Lobar Degeneration., J Alzheimers Dis, vol. 61, no. 3, pp. 1053-1060, 2018.
E. Gómez-Tortosa, Ruggiero, M., Sainz, M. José, Villarejo-Galende, A., Prieto-Jurczynska, C., Pérez, B. Venegas, Ordás, C., Agüero, P., Guerrero-López, R., and Pérez-Pérez, J., SORL1 Variants in Familial Alzheimer's Disease., J Alzheimers Dis, vol. 61, no. 4, pp. 1275-1281, 2018.
K. Muthukumaran, Kanwar, A., Vegh, C., Marginean, A., Elliott, A., Guilbeault, N., Badour, A., Sikorska, M., Cohen, J., and Pandey, S., Ubisol-Q10 (a Nanomicellar Water-Soluble Formulation of CoQ10) Treatment Inhibits Alzheimer-Type Behavioral and Pathological Symptoms in a Double Transgenic Mouse (TgAPEswe, PSEN1dE9) Model of Alzheimer's Disease., J Alzheimers Dis, vol. 61, no. 1, pp. 221-236, 2018.
2016
F. Guillot, Kemppainen, S., Lavasseur, G., Miettinen, P. O., Laroche, S., Tanila, H., and Davis, S., Brain-Specific Basal and Novelty-Induced Alternations in PI3K-Akt and MAPK/ERK Signaling in a Middle-Aged AβPP/PS1 Mouse Model of Alzheimer's Disease., J Alzheimers Dis, vol. 51, no. 4, pp. 1157-73, 2016.
H. Qiu, Zhong, R., Liu, H., Zhang, F., Li, S., and Le, W., Chronic Sleep Deprivation Exacerbates Learning-Memory Disability and Alzheimer's Disease-Like Pathologies in AβPP(swe)/PS1(ΔE9) Mice., J Alzheimers Dis, vol. 50, no. 3, pp. 669-85, 2016.
K. R. LeVault, Tischkau, S. A., and Brewer, G. J., Circadian Disruption Reveals a Correlation of an Oxidative GSH/GSSG Redox Shift with Learning and Impaired Memory in an Alzheimer's Disease Mouse Model., J Alzheimers Dis, vol. 49, no. 2, pp. 301-16, 2016.
Z. - Q. Xu, Huang, H., Chen, Y. - L., Gao, Y. - Y., Xu, J., Marshall, C., Cai, Z. - Y., and Xiao, M., Different Expression Patterns of Amyloid-β Protein Precursor Secretases in Human and Mouse Hippocampal Neurons: A Potential Contribution to Species Differences in Neuronal Susceptibility to Amyloid-β Pathogenesis., J Alzheimers Dis, vol. 51, no. 1, pp. 179-95, 2016.
Z. Rusznák, Kim, W. Scott, Hsiao, J. - H. T., Halliday, G. M., Paxinos, G., and Fu, Y. H., Early in vivo Effects of the Human Mutant Amyloid-β Protein Precursor (hAβPPSwInd) on the Mouse Olfactory Bulb., J Alzheimers Dis, vol. 49, no. 2, pp. 443-57, 2016.
B. Ongali, Nicolakakis, N., Tong, X. - K., Aboulkassim, T., Imboden, H., and Hamel, E., Enalapril Alone or Co-Administered with Losartan Rescues Cerebrovascular Dysfunction, but not Mnemonic Deficits or Amyloidosis in a Mouse Model of Alzheimer's Disease., J Alzheimers Dis, vol. 51, no. 4, pp. 1183-95, 2016.
A. E. Roher, Maarouf, C. L., and Kokjohn, T. A., Familial Presenilin Mutations and Sporadic Alzheimer's Disease Pathology: Is the Assumption of Biochemical Equivalence Justified?, J Alzheimers Dis, vol. 50, no. 3, pp. 645-58, 2016.
M. Díaz, Fabelo, N., Casañas-Sánchez, V., Marin, R., Gómez, T., Quinto-Alemany, D., and Pérez, J. A., Hippocampal Lipid Homeostasis in APP/PS1 Mice is Modulated by a Complex Interplay Between Dietary DHA and Estrogens: Relevance for Alzheimer's Disease., J Alzheimers Dis, vol. 49, no. 2, pp. 459-81, 2016.
P. Jul, Volbracht, C., de Jong, I. E. M., Helboe, L., Elvang, A. Brandt, and Pedersen, J. Torleif, Hyperactivity with Agitative-Like Behavior in a Mouse Tauopathy Model., J Alzheimers Dis, vol. 49, no. 3, pp. 783-95, 2016.
Y. Kim, Kim, C., Jang, H. Young, and Mook-Jung, I., Inhibition of Cholesterol Biosynthesis Reduces γ-Secretase Activity and Amyloid-β Generation., J Alzheimers Dis, vol. 51, no. 4, pp. 1057-68, 2016.
G. Lou, Zhang, Q., Xiao, F., Xiang, Q., Su, Z., and Huang, Y., Intranasal TAT-haFGF Improves Cognition and Amyloid-β Pathology in an AβPP/PS1 Mouse Model of Alzheimer's Disease., J Alzheimers Dis, vol. 51, no. 4, pp. 985-90, 2016.

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