Biblio
“Is the Frontal Lobe the Primary Target of SARS-CoV-2?”, J Alzheimers Dis, vol. 81, no. 1, pp. 75-81, 2021.
, “Family History of Alzheimer's Disease is Associated with Impaired Perceptual Discrimination of Novel Objects.”, J Alzheimers Dis, vol. 57, no. 3, pp. 735-745, 2017.
, “The First Historically Reported Italian Family with FTD/ALS Teaches a Lesson on C9orf72 RE: Clinical Heterogeneity and Oligogenic Inheritance.”, J Alzheimers Dis, vol. 62, no. 2, pp. 687-697, 2018.
, “Future Dementia Severity is Almost Entirely Explained by the Latent Variable δ's Intercept and Slope.”, J Alzheimers Dis, vol. 49, no. 2, pp. 521-9, 2016.
, “Functional Connectivity of Ventral and Dorsal Visual Streams in Posterior Cortical Atrophy.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1119-30, 2016.
, “Formal Psychiatric Disorders are not Overrepresented in Behavioral Variant Frontotemporal Dementia.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1249-56, 2016.
, “Fibrillar Amyloid-β Accumulation Triggers an Inflammatory Mechanism Leading to Hyperphosphorylation of the Carboxyl-Terminal End of Tau Polypeptide in the Hippocampal Formation of the 3×Tg-AD Transgenic Mouse.”, J Alzheimers Dis, vol. 52, no. 1, pp. 243-69, 2016.
, “Frequency of Cardiovascular Genetic Risk Factors in a Calabrian Population and Their Effects on Dementia.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1179-1187, 2018.
, “Future Dementia Severity is Almost Entirely Explained by the Latent Variable δ's Intercept and Slope.”, J Alzheimers Dis, vol. 49, no. 2, pp. 521-9, 2016.
, “Fibrillar Amyloid-β Accumulation Triggers an Inflammatory Mechanism Leading to Hyperphosphorylation of the Carboxyl-Terminal End of Tau Polypeptide in the Hippocampal Formation of the 3×Tg-AD Transgenic Mouse.”, J Alzheimers Dis, vol. 52, no. 1, pp. 243-69, 2016.
, “Frequency of Cardiovascular Genetic Risk Factors in a Calabrian Population and Their Effects on Dementia.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1179-1187, 2018.
, “Fibrillar Amyloid-β Accumulation Triggers an Inflammatory Mechanism Leading to Hyperphosphorylation of the Carboxyl-Terminal End of Tau Polypeptide in the Hippocampal Formation of the 3×Tg-AD Transgenic Mouse.”, J Alzheimers Dis, vol. 52, no. 1, pp. 243-69, 2016.
, “Future Dementia Severity is Almost Entirely Explained by the Latent Variable δ's Intercept and Slope.”, J Alzheimers Dis, vol. 49, no. 2, pp. 521-9, 2016.
, “Familial Presenilin Mutations and Sporadic Alzheimer's Disease Pathology: Is the Assumption of Biochemical Equivalence Justified?”, J Alzheimers Dis, vol. 50, no. 3, pp. 645-58, 2016.
, “Fluoxetine Treatment Induces Seizure Behavior and Premature Death in APPswe/PS1dE9 Mice.”, J Alzheimers Dis, vol. 51, no. 3, pp. 677-82, 2016.
, “Functional Connectivity of Ventral and Dorsal Visual Streams in Posterior Cortical Atrophy.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1119-30, 2016.
, “Fibrillar Amyloid-β Accumulation Triggers an Inflammatory Mechanism Leading to Hyperphosphorylation of the Carboxyl-Terminal End of Tau Polypeptide in the Hippocampal Formation of the 3×Tg-AD Transgenic Mouse.”, J Alzheimers Dis, vol. 52, no. 1, pp. 243-69, 2016.
, “Free Heme and Amyloid-β: A Fatal Liaison in Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 3, pp. 963-984, 2018.
, “Family History of Alzheimer's Disease is Associated with Impaired Perceptual Discrimination of Novel Objects.”, J Alzheimers Dis, vol. 57, no. 3, pp. 735-745, 2017.
, “Frequency of Cardiovascular Genetic Risk Factors in a Calabrian Population and Their Effects on Dementia.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1179-1187, 2018.
, “Functional Neurophysiological Biomarkers of Early-Stage Alzheimer's Disease: A Perspective of Network Hyperexcitability in Disease Progression.”, J Alzheimers Dis, vol. 88, no. 3, pp. 809-836, 2022.
, “From Association to Intervention: The Alzheimer's Disease-Associated Processes and Targets (ADAPT) Ontology.”, J Alzheimers Dis, vol. 94, no. s1, pp. S87-S96, 2023.
, “Free Heme and Amyloid-β: A Fatal Liaison in Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 3, pp. 963-984, 2018.
, “Free Heme and Amyloid-β: A Fatal Liaison in Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 3, pp. 963-984, 2018.
, “Familial Presenilin Mutations and Sporadic Alzheimer's Disease Pathology: Is the Assumption of Biochemical Equivalence Justified?”, J Alzheimers Dis, vol. 50, no. 3, pp. 645-58, 2016.
,