Biblio
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“Feasibility of Lumbar Puncture in the Study of Cerebrospinal Fluid Biomarkers for Alzheimer's Disease in Subjects with Down Syndrome.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1489-1496, 2017.
, “From Cerebrospinal Fluid to Blood: The Third Wave of Fluid Biomarkers for Alzheimer's Disease.”, J Alzheimers Dis, vol. 64, no. s1, pp. S271-S279, 2018.
, “A Genetic Variant of the Sortilin 1 Gene is Associated with Reduced Risk of Alzheimer's Disease.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1353-63, 2016.
, “Greater specificity for cerebrospinal fluid P-tau231 over P-tau181 in the differentiation of healthy controls from Alzheimer's disease.”, J Alzheimers Dis, vol. 49, no. 1, pp. 93-100, 2016.
, “Improved Cerebrospinal Fluid-Based Discrimination between Alzheimer's Disease Patients and Controls after Correction for Ventricular Volumes.”, J Alzheimers Dis, vol. 56, no. 2, pp. 543-555, 2017.
, “Induction of Amyloid-β42 Production by Fipronil and Other Pyrazole Insecticides.”, J Alzheimers Dis, vol. 62, no. 4, pp. 1663-1681, 2018.
, “Inflammation, Amyloid, and Atrophy in The Aging Brain: Relationships with Longitudinal Changes in Cognition.”, J Alzheimers Dis, vol. 58, no. 3, pp. 829-840, 2017.
, “Insulin Resistance is Associated with Increased Levels of Cerebrospinal Fluid Biomarkers of Alzheimer's Disease and Reduced Memory Function in At-Risk Healthy Middle-Aged Adults.”, J Alzheimers Dis, vol. 52, no. 4, pp. 1373-83, 2016.
, “Intracranial Arterial 4D Flow in Individuals with Mild Cognitive Impairment is Associated with Cognitive Performance and Amyloid Positivity.”, J Alzheimers Dis, vol. 60, no. 1, pp. 243-252, 2017.
, “Low Cerebrospinal Fluid Aβ42 and Aβ40 are Related to White Matter Lesions in Cognitively Normal Elderly.”, J Alzheimers Dis, vol. 62, no. 4, pp. 1877-1886, 2018.
, “Non-Phosphorylated Tau as a Potential Biomarker of Alzheimer's Disease: Analytical and Diagnostic Characterization.”, J Alzheimers Dis, vol. 55, no. 1, pp. 159-170, 2017.
, “The Past and the Future of Alzheimer's Disease Fluid Biomarkers.”, J Alzheimers Dis, vol. 62, no. 3, pp. 1125-1140, 2018.
, “Pathway-Specific Polygenic Risk Scores as Predictors of Amyloid-β Deposition and Cognitive Function in a Sample at Increased Risk for Alzheimer's Disease.”, J Alzheimers Dis, vol. 55, no. 2, pp. 473-484, 2017.
, “Plasma Amyloid-β (Aβ42) Correlates with Cerebrospinal Fluid Aβ42 in Alzheimer's Disease.”, J Alzheimers Dis, vol. 62, no. 4, pp. 1857-1863, 2018.
, “Plasma Tau Association with Brain Atrophy in Mild Cognitive Impairment and Alzheimer's Disease.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1245-1254, 2017.
, “Preclinical Amyloid-β and Axonal Degeneration Pathology in Delirium.”, J Alzheimers Dis, vol. 55, no. 1, pp. 371-379, 2017.
, “Reduced Cerebrospinal Fluid Concentration of Apolipoprotein A-I in Patients with Alzheimer's Disease.”, J Alzheimers Dis, vol. 59, no. 3, pp. 1017-1026, 2017.
, “S-[18F]THK-5117-PET and [11C]PIB-PET Imaging in Idiopathic Normal Pressure Hydrocephalus in Relation to Confirmed Amyloid-β Plaques and Tau in Brain Biopsies.”, J Alzheimers Dis, vol. 64, no. 1, pp. 171-179, 2018.
, “Serum Hepcidin Levels in Cognitively Normal Older Adults with High Neocortical Amyloid-β Load.”, J Alzheimers Dis, vol. 76, no. 1, pp. 291-301, 2020.
, “Specific Triazine Herbicides Induce Amyloid-β42 Production.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1593-1605, 2016.
, “Synaptic Molecular and Neurophysiological Markers Are Independent Predictors of Progression in Alzheimer's Disease.”, J Alzheimers Dis, vol. 83, no. 1, pp. 355-366, 2021.
, “Time Trends of Cerebrospinal Fluid Biomarkers of Neurodegeneration in Idiopathic Normal Pressure Hydrocephalus.”, J Alzheimers Dis, vol. 80, no. 4, pp. 1629-1642, 2021.
, “Two phase 3 trials of bapineuzumab in mild-to-moderate Alzheimer's disease.”, N Engl J Med, vol. 370, no. 4, pp. 322-33, 2014.
, “Ultrasensitive Detection of Plasma Amyloid-β as a Biomarker for Cognitively Normal Elderly Individuals at Risk of Alzheimer's Disease.”, J Alzheimers Dis, vol. 71, no. 3, pp. 775-783, 2019.
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