Biblio
“A Tale of Two Diseases: Exploring Mechanisms Linking Diabetes Mellitus with Alzheimer's Disease.”, J Alzheimers Dis, vol. 85, no. 2, pp. 485-501, 2022.
, “α-Lipoic Acid Has the Potential to Normalize Copper Metabolism, Which Is Dysregulated in Alzheimer's Disease.”, J Alzheimers Dis, vol. 85, no. 2, pp. 715-728, 2022.
, “Anti-Aβ Antibody Aducanumab Regulates the Proteome of Senile Plaques and Closely Surrounding Tissue in a Transgenic Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 79, no. 1, pp. 249-265, 2021.
, “Cannabidiol Ameliorates Cognitive Function via Regulation of IL-33 and TREM2 Upregulation in a Murine Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 80, no. 3, pp. 973-977, 2021.
, “Clinical Approach of Low-Dose Whole-Brain Ionizing Radiation Treatment in Alzheimer's Disease Dementia Patients.”, J Alzheimers Dis, vol. 80, no. 3, pp. 941-947, 2021.
, “Gut Microbiota Alterations and Cognitive Impairment Are Sexually Dissociated in a Transgenic Mice Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 82, no. s1, pp. S195-S214, 2021.
, “Hypoxia, Oxidative Stress, and Inflammation: Three Faces of Neurodegenerative Diseases.”, J Alzheimers Dis, vol. 82, no. s1, pp. S109-S126, 2021.
, “The Locus Coeruleus in Aging and Alzheimer's Disease: A Postmortem and Brain Imaging Review.”, J Alzheimers Dis, vol. 83, no. 1, pp. 5-22, 2021.
, “NLRP3 Inflammasome: A Starring Role in Amyloid-β- and Tau-Driven Pathological Events in Alzheimer's Disease.”, J Alzheimers Dis, vol. 83, no. 3, pp. 939-961, 2021.
, “Pharmacological Strategies to Improve Dendritic Spines in Alzheimer's Disease.”, J Alzheimers Dis, vol. 82, no. s1, pp. S91-S107, 2021.
, “The Potential Role of Ferroptosis in Alzheimer's Disease.”, J Alzheimers Dis, vol. 80, no. 3, pp. 907-925, 2021.
, “Sleep Deprivation, a Link Between Post-Traumatic Stress Disorder and Alzheimer's Disease.”, J Alzheimers Dis, vol. 79, no. 4, pp. 1443-1449, 2021.
, “A Super-Resolved View of the Alzheimer's Disease-Related Amyloidogenic Pathway in Hippocampal Neurons.”, J Alzheimers Dis, vol. 83, no. 2, pp. 833-852, 2021.
, “ABC Transporters Are Key Players in Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 2, pp. 463-485, 2018.
, “Age-Related Changes in the Spatial Frequency Threshold of Male and Female 3xTg-AD Mice Using OptoMotry.”, J Alzheimers Dis, vol. 62, no. 2, pp. 591-596, 2018.
, “Alzheimer's Disease: Recent Concepts on the Relation of Mitochondrial Disturbances, Excitotoxicity, Neuroinflammation, and Kynurenines.”, J Alzheimers Dis, vol. 62, no. 2, pp. 523-547, 2018.
, “An Aspartyl Cathepsin Targeted PET Agent: Application in an Alzheimer's Disease Mouse Model.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1241-1252, 2018.
, “Clinical Trials for Disease-Modifying Therapies in Alzheimer's Disease: A Primer, Lessons Learned, and a Blueprint for the Future.”, J Alzheimers Dis, vol. 64, no. s1, pp. S3-S22, 2018.
, “Cognitive Decline in Preclinical Alzheimer's Disease: Amyloid-Beta versus Tauopathy.”, J Alzheimers Dis, vol. 61, no. 1, pp. 265-281, 2018.
, “Differences in Synaptic Dysfunction Between rTg4510 and APP/PS1 Mouse Models of Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 195-208, 2018.
, “Does the Genetic Feature of the Chinese Tree Shrew (Tupaia belangeri chinensis) Support Its Potential as a Viable Model for Alzheimer's Disease Research?”, J Alzheimers Dis, vol. 61, no. 3, pp. 1015-1028, 2018.
, “Dysregulation and Dislocation of SFPQ Disturbed DNA Organization in Alzheimer's Disease and Frontotemporal Dementia.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1311-1321, 2018.
, “Early and Persistent O-GlcNAc Protein Modification in the Streptozotocin Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 237-249, 2018.
, “Early Stage Alterations in CA1 Extracellular Region Proteins Indicate Dysregulation of IL6 and Iron Homeostasis in the 5XFAD Alzheimer's Disease Mouse Model.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1399-1410, 2018.
, “Endogenous Murine Amyloid-β Peptide Assembles into Aggregates in the Aged C57BL/6J Mouse Suggesting These Animals as a Model to Study Pathogenesis of Amyloid-β Plaque Formation.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1425-1450, 2018.
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