Biblio
“Noninvasive Antemortem Detection of Retinal Prions by a Fluorescent Tracer.”, J Alzheimers Dis, vol. 88, no. 3, pp. 1137-1145, 2022.
, “Ethnic Variations in Prognosis of Patients with Dementia: A Prospective Nationwide Registry Linkage Study in The Netherlands.”, J Alzheimers Dis, vol. 56, no. 1, pp. 205-213, 2017.
, “Quantitative Neuroimaging Software for Clinical Assessment of Hippocampal Volumes on MR Imaging.”, J Alzheimers Dis, vol. 49, no. 3, pp. 723-32, 2016.
, “Endogenous Murine Amyloid-β Peptide Assembles into Aggregates in the Aged C57BL/6J Mouse Suggesting These Animals as a Model to Study Pathogenesis of Amyloid-β Plaque Formation.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1425-1450, 2018.
, “Co-Localization of Glia Maturation Factor with NLRP3 Inflammasome and Autophagosome Markers in Human Alzheimer's Disease Brain.”, J Alzheimers Dis, vol. 60, no. 3, pp. 1143-1160, 2017.
, “Lipid Metabolism and Survival Across the Frontotemporal Dementia-Amyotrophic Lateral Sclerosis Spectrum: Relationships to Eating Behavior and Cognition.”, J Alzheimers Dis, vol. 61, no. 2, pp. 773-783, 2018.
, “Memory Impairment at Initial Clinical Presentation in Posterior Cortical Atrophy.”, J Alzheimers Dis, vol. 52, no. 4, pp. 1245-50, 2016.
, “Report of the task force on designing clinical trials in early (predementia) AD.”, Neurology, vol. 76, no. 3, pp. 280-6, 2011.
, “Neurodegenerative Disease-Related Proteins within the Epidermal Layer of the Human Skin.”, J Alzheimers Dis, vol. 69, no. 2, pp. 463-478, 2019.
, “A Small Molecule Inhibitor of Plasminogen Activator Inhibitor-1 Reduces Brain Amyloid-β Load and Improves Memory in an Animal Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 64, no. 2, pp. 447-457, 2018.
, “Change of Amyloid-β 1-42 Toxic Conformer Ratio After Cerebrospinal Fluid Diversion Predicts Long-Term Cognitive Outcome in Patients with Idiopathic Normal Pressure Hydrocephalus.”, J Alzheimers Dis, vol. 63, no. 3, pp. 989-1002, 2018.
, “Platelets: Peripheral Biomarkers of Dementia?”, J Alzheimers Dis, vol. 63, no. 4, pp. 1235-1259, 2018.
, “Cerebral Microbleeds and Cerebrovascular Reactivity in the General Population: The EDAN Study.”, J Alzheimers Dis, vol. 53, no. 2, pp. 497-503, 2016.
, “Time Trends in the Prevalence of Neurocognitive Disorders and Cognitive Impairment in the United States: The Effects of Disease Severity and Improved Ascertainment.”, J Alzheimers Dis, vol. 64, no. 1, pp. 137-148, 2018.
, “Distinct White Matter Changes Associated with Cerebrospinal Fluid Amyloid-β1-42 and Hypertension.”, J Alzheimers Dis, vol. 66, no. 3, pp. 1095-1104, 2018.
, “Mixed Brain Pathology Is the Most Common Cause of Cognitive Impairment in the Elderly.”, J Alzheimers Dis, vol. 78, no. 1, pp. 453-465, 2020.
, “Is Acculturation Associated with the Cognitive Performance of Older Hispanics?”, J Alzheimers Dis, vol. 85, no. 2, pp. 535-544, 2021.
, “The diagnosis of mild cognitive impairment due to Alzheimer's disease: recommendations from the National Institute on Aging-Alzheimer's Association workgroups on diagnostic guidelines for Alzheimer's disease.”, Alzheimers Dement, vol. 7, no. 3, pp. 270-9, 2011.
, “The Role of Verb Fluency in the Detection of Early Cognitive Impairment in Alzheimer's Disease.”, J Alzheimers Dis, vol. 62, no. 2, pp. 611-619, 2018.
, “Decreased Prefrontal Activation during Matrix Reasoning in Predementia Progranulin Mutation Carriers.”, J Alzheimers Dis, vol. 62, no. 2, pp. 583-589, 2018.
, “COVID-19 Crisis Effects on Caregiver Distress in Neurocognitive Disorder.”, J Alzheimers Dis, vol. 79, no. 1, pp. 459-466, 2021.
, “Reduction of RPT6/S8 (a Proteasome Component) and Proteasome Activity in the Cortex is associated with Cognitive Impairment in Lewy Body Dementia.”, J Alzheimers Dis, vol. 57, no. 2, pp. 373-386, 2017.
, “Subjective Cognitive Decline and APOE ɛ4: A Systematic Review.”, J Alzheimers Dis, vol. 65, no. 1, pp. 303-320, 2018.
, “Parkinsonism Differentiates Idiopathic Normal Pressure Hydrocephalus from Its Mimics.”, J Alzheimers Dis, vol. 54, no. 1, pp. 123-7, 2016.
, “Parkinsonism is a Phenotypical Signature of Amyloidopathy in Patients with Gait Disorders.”, J Alzheimers Dis, vol. 63, no. 4, pp. 1373-1381, 2018.
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