Biblio

Export 3284 results:
Author Title [ Type(Desc)] Year
Journal Article
E. Del Valle, Navarro, A., Martínez-Pinilla, E., Torices, S., and Tolivia, J., “Apo J and Apo D: Complementary or Antagonistic Roles in Alzheimer's Disease?”, J Alzheimers Dis, vol. 53, no. 2, pp. 639-50, 2016.
X. Tang, Holland, D., Dale, A. M., and Miller, M. I., “APOE Affects the Volume and Shape of the Amygdala and the Hippocampus in Mild Cognitive Impairment and Alzheimer's Disease: Age Matters.”, J Alzheimers Dis, vol. 47, no. 3, pp. 645-60, 2015.
J. B. Toledo, Habes, M., Sotiras, A., Bjerke, M., Fan, Y., Weiner, M. W., Shaw, L. M., Davatzikos, C., and Trojanowski, J. Q., “APOE Effect on Amyloid-β PET Spatial Distribution, Deposition Rate, and Cut-Points.”, J Alzheimers Dis, vol. 69, no. 3, pp. 783-793, 2019.
J. Foraker, Millard, S. P., Leong, L., Thomson, Z., Chen, S., C Keene, D., Bekris, L. M., and Yu, C. - E., “The APOE Gene is Differentially Methylated in Alzheimer's Disease.”, J Alzheimers Dis, 2015.
J. C. Morris, Roe, C. M., Xiong, C., Fagan, A. M., Goate, A. M., Holtzman, D. M., and Mintun, M. A., “APOE predicts amyloid-beta but not tau Alzheimer pathology in cognitively normal aging.”, Ann Neurol, vol. 67, no. 1, pp. 122-31, 2010.
E. Werden, Khlif, M. Salah, Bird, L. J., Cumming, T., Bradshaw, J., Khan, W., Pase, M., Restrepo, C., Veldsman, M., Egorova, N., Patel, S. K., Gottlieb, E., and Brodtmann, A., “APOE ɛ4 Carriers Show Delayed Recovery of Verbal Memory and Smaller Entorhinal Volume in the First Year After Ischemic Stroke.”, J Alzheimers Dis, vol. 71, no. 1, pp. 245-259, 2019.
R. López-Higes, Rodríguez-Rojo, I. C., Prados, J. M., Montejo, P., Del-Río, D., Delgado-Losada, M. Luisa, Montenegro, M., López-Sanz, D., and Barabash, A., “APOE ε4 Modulation of Training Outcomes in Several Cognitive Domains in a Sample of Cognitively Intact Older Adults.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1201-1215, 2017.
D. Hu, Chen, M., Li, X., Daley, S., Morin, P., Han, Y., Hemberg, M., Weiner, H. L., and Xia, W., “ApoE ε4-dependent Alteration of CXCR3  CD127 CD4 T cells associates with elevated plasma neurofilament light chain in Alzheimer's disease.”, J Alzheimers Dis, p. 13872877251320123, 2025.
M. Berger, Cooter, M., Roesler, A. S., Chung, S., Park, J., Modliszewski, J. L., VanDusen, K. W., J Thompson, W., Moseley, A., Devinney, M. J., Smani, S., Hall, A., Cai, V., Browndyke, J. N., Lutz, M. W., and Corcoran, D. L., “APOE4 Copy Number-Dependent Proteomic Changes in the Cerebrospinal Fluid.”, J Alzheimers Dis, vol. 79, no. 2, pp. 511-530, 2021.
S. Rabipour, Rajagopal, S., Yu, E., Pasvanis, S., Lafaille-Magnan, M. - E., Breitner, J., and M Rajah, N., “APOE4 Status is Related to Differences in Memory-Related Brain Function in Asymptomatic Older Adults with Family History of Alzheimer's Disease: Baseline Analysis of the PREVENT-AD Task Functional MRI Dataset.”, J Alzheimers Dis, vol. 76, no. 1, pp. 97-119, 2020.
O. Lerch, Pařízková, M., Vyhnálek, M., Nedelska, Z., Hort, J., and Laczó, J., “APOEɛ4 Allele Moderates the Association Between Basal Forebrain Nuclei Volumes and Allocentric Navigation in Older Adults Without Dementia.”, J Alzheimers Dis, vol. 86, no. 1, pp. 155-171, 2022.
S. Hollands, Lim, Y. Ying, Laws, S. M., Villemagne, V. L., Pietrzak, R. H., Harrington, K., Porter, T., Snyder, P., Ames, D., Fowler, C., Rainey-Smith, S. R., Martins, R. N., Salvado, O., Robertson, J., Rowe, C. C., Masters, C. L., and Maruff, P., “APOEɛ4 Genotype, Amyloid, and Clinical Disease Progression in Cognitively Normal Older Adults.”, J Alzheimers Dis, vol. 57, no. 2, 2017.
R. Cardoso, Lemos, C., Oliveiros, B., Almeida, M. Rosário, Baldeiras, I., Pereira, C. Fragão, Santos, A., Duro, D., Vieira, D., Santana, I., and Oliveira, C. Resende, “APOEɛ4-TOMM40L Haplotype Increases the Risk of Mild Cognitive Impairment Conversion to Alzheimer's Disease.”, J Alzheimers Dis, vol. 78, no. 2, pp. 587-601, 2020.
R. E. R. Slot, Van Harten, A. C., Kester, M. I., Jongbloed, W., Bouwman, F. H., Teunissen, C. E., Scheltens, P., Veerhuis, R., and van der Flier, W. M., “Apolipoprotein A1 in Cerebrospinal Fluid and Plasma and Progression to Alzheimer's Disease in Non-Demented Elderly.”, J Alzheimers Dis, vol. 56, no. 2, pp. 687-697, 2017.
E. Dorey, Bamji-Mirza, M., Najem, D., Li, Y., Liu, H., Callaghan, D., Walker, D., Lue, L. - F., Stanimirovic, D., and Zhang, W., “Apolipoprotein E Isoforms Differentially Regulate Alzheimer's Disease and Amyloid-β-Induced Inflammatory Response in vivo and in vitro.”, J Alzheimers Dis, vol. 57, no. 4, pp. 1265-1279, 2017.
S. K. Singhrao, Harding, A., Chukkapalli, S., Olsen, I., Kesavalu, L., and Crean, S. J., “Apolipoprotein E Related Co-Morbidities and Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 4, pp. 935-48, 2016.
M. Basta, Zaganas, I., Simos, P., Koutentaki, E., Dimovasili, C., Mathioudakis, L., Bourbouli, M., Panagiotakis, S., Kapetanaki, S., and Vgontzas, A., “Apolipoprotein E ɛ4 (APOE ɛ4) Allele is Associated with Long Sleep Duration Among Elderly with Cognitive Impairment.”, J Alzheimers Dis, vol. 79, no. 2, pp. 763-771, 2021.
T. Persson, Lattanzio, F., Calvo-Garrido, J., Rimondini, R., Rubio-Rodrigo, M., Sundström, E., Maioli, S., Sandebring-Matton, A., and Cedazo-Minguez, A., “Apolipoprotein E4 Elicits Lysosomal Cathepsin D Release, Decreased Thioredoxin-1 Levels, and Apoptosis.”, J Alzheimers Dis, vol. 56, no. 2, pp. 601-617, 2017.
L. Calderón-Garcidueñas and de la Monte, S. M., “Apolipoprotein E4, Gender, Body Mass Index, Inflammation, Insulin Resistance, and Air Pollution Interactions: Recipe for Alzheimer's Disease Development in Mexico City Young Females.”, J Alzheimers Dis, vol. 58, no. 3, pp. 613-630, 2017.
Y. Ling Chai, Yeo, H. Kai- Hui, Wang, J., Hilal, S., Ikram, M. Kamran, Venketasubramanian, N., Wong, B. - S., and Chen, C. Li- Hsian, “Apolipoprotein ɛ4 is Associated with Dementia and Cognitive Impairment Predominantly Due to Alzheimer's Disease and Not with Vascular Cognitive Impairment: A Singapore-Based Cohort.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1111-8, 2016.
C. J. Smith and J Ashford, W., “Apolipoprotein ɛ4-Associated Protection Against Pediatric Enteric Infections is a Survival Advantage in Pre-Industrial Populations.”, J Alzheimers Dis, vol. 93, no. 3, pp. 907-918, 2023.
N. Nakamura, Ohyagi, Y., Imamura, T., Yanagihara, Y. T., Iinuma, K. M., Soejima, N., Murai, H., Yamasaki, R., and Kira, J. - I., “Apomorphine Therapy for Neuronal Insulin Resistance in a Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1151-1161, 2017.
R. Pluta, Bogucka-Kocka, A., Bogucki, J., Kocki, J., and Czuczwar, S. J., “Apoptosis, Autophagy, and Mitophagy Genes in the CA3 Area in an Ischemic Model of Alzheimer's Disease with 2-Year Survival.”, J Alzheimers Dis, vol. 99, no. 4, pp. 1375-1383, 2024.
F. Sellal, Wallon, D., Martinez-Almoyna, L., Marelli, C., Dhar, A., Oesterlé, H., Rovelet-Lecrux, A., Rousseau, S., Kourkoulis, C. E., Rosand, J., DiPucchio, Z. Y., Frosch, M., Gombert, C., Audoin, B., Miné, M., Riant, F., Frebourg, T., Hannequin, D., Campion, D., Greenberg, S. M., Tournier-Lasserve, E., and Nicolas, G., “APP Mutations in Cerebral Amyloid Angiopathy with or without Cortical Calcifications: Report of Three Families and a Literature Review.”, J Alzheimers Dis, vol. 56, no. 1, pp. 37-46, 2017.
T. B. Shea and Remington, R., “Apparent Cognitive Decline as Revealed by an Executive Function Test within a Cohort of Elderly Individuals Self-Reporting Normal Cognitive Performance.”, J Alzheimers Dis, vol. 61, no. 3, pp. 913-915, 2018.

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