Biblio
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“Investigating Causal Relations Between Circulating Metabolites and Alzheimer's Disease: A Mendelian Randomization Study.”, J Alzheimers Dis, vol. 87, no. 1, pp. 463-477, 2022.
, “Late-Onset Alcohol Abuse as a Presenting Symptom of Neurodegenerative Diseases.”, J Alzheimers Dis, vol. 86, no. 3, pp. 1073-1080, 2022.
, “Late-Onset Alcohol Abuse as a Presenting Symptom of Neurodegenerative Diseases.”, J Alzheimers Dis, vol. 86, no. 3, pp. 1073-1080, 2022.
, “Lipid Peroxidation as a Marker of Apathy and Executive Dysfunction in Patients at Risk for Vascular Cognitive Impairment.”, J Alzheimers Dis, vol. 89, no. 2, pp. 733-743, 2022.
, “Lipid Rafts Act as a Common Platform for Amyloid-β Oligomer-Induced Alzheimer's Disease Pathology.”, J Alzheimers Dis, vol. 87, no. 3, pp. 1189-1203, 2022.
, “Lipid Rafts Act as a Common Platform for Amyloid-β Oligomer-Induced Alzheimer's Disease Pathology.”, J Alzheimers Dis, vol. 87, no. 3, pp. 1189-1203, 2022.
, “Lipid Rafts Act as a Common Platform for Amyloid-β Oligomer-Induced Alzheimer's Disease Pathology.”, J Alzheimers Dis, vol. 87, no. 3, pp. 1189-1203, 2022.
, “Lipid Rafts Act as a Common Platform for Amyloid-β Oligomer-Induced Alzheimer's Disease Pathology.”, J Alzheimers Dis, vol. 87, no. 3, pp. 1189-1203, 2022.
, “Mild Cognitive Impairment, Reversion Rates, and Associated Factors: Comparison of Two Diagnostic Approaches.”, J Alzheimers Dis, vol. 91, no. 2, pp. 585-601, 2022.
, “Mild Traumatic Brain Injury is Related to Elevated Cerebrospinal Fluid Tau in Alzheimer's Disease Dementia.”, J Alzheimers Dis, vol. 87, no. 4, pp. 1491-1496, 2022.
, “A Mitochondrial DNA Haplogroup Defines Patterns of Five-Year Cognitive Change.”, J Alzheimers Dis, vol. 89, no. 3, pp. 913-922, 2022.
, “Mitochondrial Targeting of Amyloid-β Protein Precursor Intracellular Domain Induces Hippocampal Cell Death via a Mechanism Distinct from Amyloid-β.”, J Alzheimers Dis, vol. 86, no. 4, pp. 1727-1744, 2022.
, “Mitochondrial Targeting of Amyloid-β Protein Precursor Intracellular Domain Induces Hippocampal Cell Death via a Mechanism Distinct from Amyloid-β.”, J Alzheimers Dis, vol. 86, no. 4, pp. 1727-1744, 2022.
, “Non-Alcoholic Fatty Liver Disease, Liver Fibrosis, and Regional Amyloid-β and Tau Pathology in Middle-Aged Adults: The Framingham Study.”, J Alzheimers Dis, vol. 86, no. 3, pp. 1371-1383, 2022.
, “Non-Alcoholic Fatty Liver Disease, Liver Fibrosis, and Regional Amyloid-β and Tau Pathology in Middle-Aged Adults: The Framingham Study.”, J Alzheimers Dis, vol. 86, no. 3, pp. 1371-1383, 2022.
, “Noninvasive Antemortem Detection of Retinal Prions by a Fluorescent Tracer.”, J Alzheimers Dis, vol. 88, no. 3, pp. 1137-1145, 2022.
, “Noninvasive Antemortem Detection of Retinal Prions by a Fluorescent Tracer.”, J Alzheimers Dis, vol. 88, no. 3, pp. 1137-1145, 2022.
, “Noninvasive Antemortem Detection of Retinal Prions by a Fluorescent Tracer.”, J Alzheimers Dis, vol. 88, no. 3, pp. 1137-1145, 2022.
, “The Outcome of Patients with Amyloid-Negative Amnestic Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 86, no. 2, pp. 629-640, 2022.
, “Pelargonidin and Berry Intake Association with Alzheimer's Disease Neuropathology: A Community-Based Study.”, J Alzheimers Dis, vol. 88, no. 2, pp. 653-661, 2022.
, “Perivascular Space Predicts Brain Hypometabolism of Individuals with Underlying Amyloid Pathology.”, J Alzheimers Dis, vol. 90, no. 3, pp. 1329-1337, 2022.
, “Perivascular Space Predicts Brain Hypometabolism of Individuals with Underlying Amyloid Pathology.”, J Alzheimers Dis, vol. 90, no. 3, pp. 1329-1337, 2022.
, “Plasma Amyloid and in vivo Brain Amyloid in Late Middle-Aged Hispanics.”, J Alzheimers Dis, vol. 87, no. 3, pp. 1229-1238, 2022.
, “Plasma Amyloid and in vivo Brain Amyloid in Late Middle-Aged Hispanics.”, J Alzheimers Dis, vol. 87, no. 3, pp. 1229-1238, 2022.
, “Plasma Myeloperoxidase as a Potential Biomarker of Patient Response to Anti-Dementia Treatment in Alzheimer's Disease.”, J Alzheimers Dis, vol. 89, no. 4, pp. 1483-1492, 2022.
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