Biblio
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“Alzheimer's Disease Diagnosis Relies on a Twofold Clinical-Biological Algorithm: Three Memory Clinic Case Reports.”, J Alzheimers Dis, vol. 60, no. 2, pp. 577-583, 2017.
, “Alzheimer's Disease: Characterization of the Brain Sites of the Initial Tau Cytoskeletal Pathology Will Improve the Success of Novel Immunological Anti-Tau Treatment Approaches.”, J Alzheimers Dis, vol. 57, no. 3, pp. 683-696, 2017.
, “Alzheimer's Disease: Characterization of the Brain Sites of the Initial Tau Cytoskeletal Pathology Will Improve the Success of Novel Immunological Anti-Tau Treatment Approaches.”, J Alzheimers Dis, vol. 57, no. 3, pp. 683-696, 2017.
, “Alzheimer's Disease Biomarkers Have Distinct Associations with Specific Hippocampal Subfield Volumes.”, J Alzheimers Dis, vol. 66, no. 2, pp. 811-823, 2018.
, “Alzheimer's Disease Biomarkers and Future Decline in Cognitive Normal Older Adults.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1451-1459, 2017.
, “Alzheimer's Disease Biomarkers and Future Decline in Cognitive Normal Older Adults.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1451-1459, 2017.
, “The Alzheimer's Disease Assessment Scale-Cognitive Subscale (ADAS-Cog): Modifications and Responsiveness in Pre-Dementia Populations. A Narrative Review.”, J Alzheimers Dis, vol. 63, no. 2, pp. 423-444, 2018.
, “Alzheimer's Disease and Neurosyphilis: Meaningful Commonalities and Differences of Clinical Phenotype and Pathophysiological Biomarkers.”, J Alzheimers Dis, vol. 94, no. 2, pp. 611-625, 2023.
, “Alzheimer's Disease: A Journey from Amyloid Peptides and Oxidative Stress, to Biomarker Technologies and Disease Prevention Strategies-Gains from AIBL and DIAN Cohort Studies.”, J Alzheimers Dis, vol. 62, no. 3, pp. 965-992, 2018.
, “Alzheimer's Disease: A Journey from Amyloid Peptides and Oxidative Stress, to Biomarker Technologies and Disease Prevention Strategies-Gains from AIBL and DIAN Cohort Studies.”, J Alzheimers Dis, vol. 62, no. 3, pp. 965-992, 2018.
, “Alzheimer's Disease: A Journey from Amyloid Peptides and Oxidative Stress, to Biomarker Technologies and Disease Prevention Strategies-Gains from AIBL and DIAN Cohort Studies.”, J Alzheimers Dis, vol. 62, no. 3, pp. 965-992, 2018.
, “Alzheimer's and Parkinson's Diseases and the Risk of Cancer: A Cohort Study.”, J Alzheimers Dis, vol. 72, no. 4, pp. 1269-1277, 2019.
, “Alzheimer-Like Pattern of Hypometabolism Emerges with Elevated Amyloid-β Burden in Down Syndrome.”, J Alzheimers Dis, vol. 61, no. 2, pp. 631-644, 2018.
, “Alzheimer DataLENS: An Open Data Analytics Portal for Alzheimer's Disease Research.”, J Alzheimers Dis, vol. 99, no. s2, pp. S397-S407, 2024.
, “Altered Sphingolipid Balance in Capillary Cerebral Amyloid Angiopathy.”, J Alzheimers Dis, vol. 60, no. 3, pp. 795-807, 2017.
, “Altered sense of humor in dementia.”, J Alzheimers Dis, vol. 49, no. 1, pp. 111-9, 2016.
, “Altered sense of humor in dementia.”, J Alzheimers Dis, vol. 49, no. 1, pp. 111-9, 2016.
, “Altered Neuronal Activity Patterns of the Prefrontal Cortex in Alzheimer's Disease After Transcranial Alternating Current Stimulation: A Resting-State fMRI Study.”, J Alzheimers Dis, vol. 101, no. 3, pp. 901-912, 2024.
, “Altered microRNA, mRNA, and Protein Expression of Neurodegeneration-Related Biomarkers and Their Transcriptional and Epigenetic Modifiers in a Human Tau Transgenic Mouse Model in Response to Developmental Lead Exposure.”, J Alzheimers Dis, vol. 63, no. 1, pp. 273-282, 2018.
, “Altered Gut Microbiota in a Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1241-1257, 2017.
, “Altered Gut Microbiota in a Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1241-1257, 2017.
, “Altered Gut Microbiome Composition and Tryptic Activity of the 5xFAD Alzheimer's Mouse Model.”, J Alzheimers Dis, vol. 56, no. 2, pp. 775-788, 2017.
, “Altered Gut Microbiome Composition and Tryptic Activity of the 5xFAD Alzheimer's Mouse Model.”, J Alzheimers Dis, vol. 56, no. 2, pp. 775-788, 2017.
, “Altered Gut Microbiome Composition and Tryptic Activity of the 5xFAD Alzheimer's Mouse Model.”, J Alzheimers Dis, vol. 56, no. 2, pp. 775-788, 2017.
, “Altered Expression of Circulating Cdc42 in Frontotemporal Lobar Degeneration.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1477-1483, 2018.
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