Biblio
Export 436 results:
[ Author
Filters: First Letter Of Keyword is S [Clear All Filters]
“Differential Diagnosis of Dementia with High Levels of Cerebrospinal Fluid Tau Protein.”, J Alzheimers Dis, vol. 51, no. 3, pp. 905-13, 2016.
, “SORL1 Variants in Familial Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1275-1281, 2018.
, “SORL1 Variants in Familial Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1275-1281, 2018.
, “Diversity of Cognitive Phenotypes Associated with C9ORF72 Hexanucleotide Expansion.”, J Alzheimers Dis, vol. 52, no. 1, pp. 25-31, 2016.
, “Anti-Correlated Cerebrospinal Fluid Biomarker Trajectories in Preclinical Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1085-97, 2016.
, “COVID-19 as a Risk Factor for Alzheimer's Disease.”, J Alzheimers Dis, vol. 91, no. 1, pp. 1-23, 2023.
, “Sildenafil as a Candidate Drug for Alzheimer's Disease: Real-World Patient Data Observation and Mechanistic Observations from Patient-Induced Pluripotent Stem Cell-Derived Neurons.”, J Alzheimers Dis, vol. 98, no. 2, pp. 643-657, 2024.
, “Sildenafil as a Candidate Drug for Alzheimer's Disease: Real-World Patient Data Observation and Mechanistic Observations from Patient-Induced Pluripotent Stem Cell-Derived Neurons.”, J Alzheimers Dis, vol. 98, no. 2, pp. 643-657, 2024.
, “Characteristics of Alzheimer's Disease Patients with Severe Executive Disorders.”, J Alzheimers Dis, vol. 51, no. 3, pp. 815-25, 2016.
, “Cognitive Variability Predicts Incident Alzheimer's Disease and Mild Cognitive Impairment Comparable to a Cerebrospinal Fluid Biomarker.”, J Alzheimers Dis, vol. 61, no. 1, pp. 79-89, 2018.
, “Current and Future Prevalence Estimates of Mild Cognitive Impairment, Dementia, and Its Subtypes in a Population-Based Sample of People 70 Years and Older in Norway: The HUNT Study.”, J Alzheimers Dis, vol. 79, no. 3, pp. 1213-1226, 2021.
, “Current and Future Prevalence Estimates of Mild Cognitive Impairment, Dementia, and Its Subtypes in a Population-Based Sample of People 70 Years and Older in Norway: The HUNT Study.”, J Alzheimers Dis, vol. 79, no. 3, pp. 1213-1226, 2021.
, “Memory impairment in transgenic Alzheimer mice requires cellular prion protein.”, J Neurosci, vol. 30, no. 18, pp. 6367-74, 2010.
, “Memory impairment in transgenic Alzheimer mice requires cellular prion protein.”, J Neurosci, vol. 30, no. 18, pp. 6367-74, 2010.
, “Impact of a Virtual Dementia Experience on Medical and Pharmacy Students' Knowledge and Attitudes Toward People with Dementia: A Controlled Study.”, J Alzheimers Dis, vol. 62, no. 2, pp. 867-876, 2018.
, “Impact of a Virtual Dementia Experience on Medical and Pharmacy Students' Knowledge and Attitudes Toward People with Dementia: A Controlled Study.”, J Alzheimers Dis, vol. 62, no. 2, pp. 867-876, 2018.
, “Differences in Synaptic Dysfunction Between rTg4510 and APP/PS1 Mouse Models of Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 195-208, 2018.
, “Differences in Synaptic Dysfunction Between rTg4510 and APP/PS1 Mouse Models of Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 195-208, 2018.
, “Association of Cerebrospinal Fluid (CSF) Insulin with Cognitive Performance and CSF Biomarkers of Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 309-320, 2018.
, “The Efficacy of Emotion Recognition Rehabilitation for People with Alzheimer's Disease.”, J Alzheimers Dis, vol. 57, no. 3, pp. 937-951, 2017.
, “The Efficacy of Emotion Recognition Rehabilitation for People with Alzheimer's Disease.”, J Alzheimers Dis, vol. 57, no. 3, pp. 937-951, 2017.
, “Linguistic Features Identify Alzheimer's Disease in Narrative Speech.”, J Alzheimers Dis, vol. 49, no. 2, pp. 407-22, 2016.
, “Neurobiology of COVID-19.”, J Alzheimers Dis, vol. 76, no. 1, pp. 3-19, 2020.
, “Lewy Bodies, Vascular Risk Factors, and Subcortical Arteriosclerotic Leukoencephalopathy, but not Alzheimer Pathology, are Associated with Development of Psychosis in Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 1, pp. 283-95, 2016.
, “Lewy Bodies, Vascular Risk Factors, and Subcortical Arteriosclerotic Leukoencephalopathy, but not Alzheimer Pathology, are Associated with Development of Psychosis in Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 1, pp. 283-95, 2016.
,