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“Cognitive Composites Domain Scores Related to Neuroimaging Biomarkers within Probable-Amnestic Mild Cognitive Impairment-Storage Subtype.”, J Alzheimers Dis, vol. 57, no. 2, pp. 447-459, 2017.
, “Cognitive and Neuropsychological Profiles in Alzheimer's Disease and Primary Age-Related Tauopathy and the Influence of Comorbid Neuropathologies.”, J Alzheimers Dis, vol. 92, no. 3, pp. 1037-1049, 2023.
, “Co-Expression of Glia Maturation Factor and Apolipoprotein E4 in Alzheimer's Disease Brain.”, J Alzheimers Dis, vol. 61, no. 2, pp. 553-560, 2018.
, “On the Clinimetrics of the Montreal Cognitive Assessment: Cutoff Analysis in Patients with Mild Cognitive Impairment due to Alzheimer's Disease.”, J Alzheimers Dis, vol. 101, no. 1, pp. 293-308, 2024.
, “On the Clinimetrics of the Montreal Cognitive Assessment: Cutoff Analysis in Patients with Mild Cognitive Impairment due to Alzheimer's Disease.”, J Alzheimers Dis, vol. 101, no. 1, pp. 293-308, 2024.
, “Clinical Subtypes of Dementia with Lewy Bodies Based on the Initial Clinical Presentation.”, J Alzheimers Dis, vol. 64, no. 2, pp. 505-513, 2018.
, “Clinical Experience with Cerebrospinal Fluid Aβ42, Total and Phosphorylated Tau in the Evaluation of 1,016 Individuals for Suspected Dementia.”, J Alzheimers Dis, vol. 65, no. 4, pp. 1417-1425, 2018.
, “Clinical Effectiveness and Tolerability of Electroconvulsive Therapy in Patients with Neuropsychiatric Symptoms of Dementia.”, J Alzheimers Dis, vol. 57, no. 1, pp. 45-51, 2017.
, “Clearing Amyloid-β through PPARγ/ApoE Activation by Genistein is a Treatment of Experimental Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 3, pp. 701-11, 2016.
, “Chronic Lithium Treatment Increases Telomere Length in Parietal Cortex and Hippocampus of Triple-Transgenic Alzheimer's Disease Mice.”, J Alzheimers Dis, vol. 63, no. 1, pp. 93-101, 2018.
, “Change of Amyloid-β 1-42 Toxic Conformer Ratio After Cerebrospinal Fluid Diversion Predicts Long-Term Cognitive Outcome in Patients with Idiopathic Normal Pressure Hydrocephalus.”, J Alzheimers Dis, vol. 63, no. 3, pp. 989-1002, 2018.
, “Challenges in Screening and Recruitment for a Neuroimaging Study in Cognitively Impaired Geriatric Inpatients.”, J Alzheimers Dis, vol. 56, no. 1, pp. 197-204, 2017.
, “Challenges and Considerations Related to Studying Dementia in Blacks/African Americans.”, J Alzheimers Dis, vol. 60, no. 1, pp. 1-10, 2017.
, “Cerebrospinal Fluid Orexin Levels and Nocturnal Sleep Disruption in Alzheimer's Disease Patients Showing Neuropsychiatric Symptoms.”, J Alzheimers Dis, vol. 66, no. 3, pp. 993-999, 2018.
, “Cerebrospinal Fluid Metals and the Association with Cerebral Small Vessel Disease.”, J Alzheimers Dis, vol. 78, no. 3, pp. 1229-1236, 2020.
, “Cerebrospinal Fluid C-C Motif Chemokine Ligand 2 Correlates with Brain Atrophy and Cognitive Impairment in Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 2, pp. 581-588, 2018.
, “Cerebrospinal Fluid Biomarkers and Reserve Variables as Predictors of Future "Non-Cognitive" Outcomes of Alzheimer's Disease.”, J Alzheimers Dis, vol. 52, no. 3, pp. 1055-64, 2016.
, “Cerebrospinal Fluid Amyloid-β Oligomer Levels in Patients with Idiopathic Normal Pressure Hydrocephalus.”, J Alzheimers Dis, vol. 83, no. 1, pp. 179-190, 2021.
, “Cerebral Microbleeds Are Associated with Cerebral Hypoperfusion in Patients with Alzheimer's Disease.”, J Alzheimers Dis, vol. 71, no. 1, pp. 273-280, 2019.
, “Cerebral Microbleeds Are Associated with Cerebral Hypoperfusion in Patients with Alzheimer's Disease.”, J Alzheimers Dis, vol. 71, no. 1, pp. 273-280, 2019.
, “Cerebral Microbleeds and Cerebrovascular Reactivity in the General Population: The EDAN Study.”, J Alzheimers Dis, vol. 53, no. 2, pp. 497-503, 2016.
, “Cerebral Amyloid Angiopathy-Related Microbleeds and Cerebrospinal Fluid Biomarkers in Alzheimer's Disease.”, J Alzheimers Dis, vol. 55, no. 3, pp. 905-913, 2017.
, “Central Obesity, Cardiometabolic Risk, and Cognitive Change in the Study of Latinos: Investigation of Neurocognitive Aging.”, J Alzheimers Dis, vol. 82, no. 3, pp. 1203-1218, 2021.
, “CD14 and Toll-Like Receptor 4 Promote Fibrillar Aβ42 Uptake by Microglia Through A Clathrin-Mediated Pathway.”, J Alzheimers Dis, vol. 68, no. 1, pp. 323-337, 2019.
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