Biblio
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“Staging Alzheimer's Disease Risk by Sequencing Brain Function and Structure, Cerebrospinal Fluid, and Cognition Biomarkers.”, J Alzheimers Dis, vol. 54, no. 3, pp. 983-993, 2016.
, “Sporadic Cases with Novel Mutations and Pedigree in Hereditary Leukoencephalopathy with Axonal Spheroids.”, J Alzheimers Dis, vol. 56, no. 3, pp. 893-898, 2017.
, “The Spectrum of Alzheimer-Type Pathology in Cognitively Normal Individuals.”, J Alzheimers Dis, vol. 91, no. 2, pp. 683-695, 2022.
, “Specific Verbal Memory Measures May Distinguish Alzheimer's Disease from Dementia with Lewy Bodies.”, J Alzheimers Dis, vol. 59, no. 3, pp. 1009-1015, 2017.
, “Specific Triazine Herbicides Induce Amyloid-β42 Production.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1593-1605, 2016.
, “Specific Triazine Herbicides Induce Amyloid-β42 Production.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1593-1605, 2016.
, “Specific Neuropsychiatric Symptoms Are Associated with Faster Progression in Alzheimer's Disease: Results of the Prospective Dementia Registry (PRODEM-Austria).”, J Alzheimers Dis, vol. 73, no. 1, pp. 125-133, 2020.
, “Specific Binding of Cu(II) Ions to Amyloid-Beta Peptides Bound to Aggregation-Inhibiting Molecules or SDS Micelles Creates Complexes that Generate Radical Oxygen Species.”, J Alzheimers Dis, vol. 54, no. 3, pp. 971-982, 2016.
, “SPARCL1 Accelerates Symptom Onset in Alzheimer's Disease and Influences Brain Structure and Function During Aging.”, J Alzheimers Dis, vol. 61, no. 1, pp. 401-414, 2018.
, “Space-Like Irradiation Exacerbated Cognitive Deficits and Amyloid Pathology in CRND8 Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 100, no. s1, pp. S327-S339, 2024.
, “SORL1 Variants Show Different Association with Early-Onset and Late-Onset Alzheimer's Disease Risk.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1121-1128, 2017.
, “SORL1 Variants Show Different Association with Early-Onset and Late-Onset Alzheimer's Disease Risk.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1121-1128, 2017.
, “SORL1 Variants in Familial Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1275-1281, 2018.
, “Soluble TREM2 and Inflammatory Proteins in Alzheimer's Disease Cerebrospinal Fluid.”, J Alzheimers Dis, vol. 73, no. 4, pp. 1615-1626, 2020.
, “Soluble Oligomers Require a Ganglioside to Trigger Neuronal Calcium Overload.”, J Alzheimers Dis, vol. 60, no. 3, pp. 923-938, 2017.
, “Soluble Amyloid-β Consumption in Alzheimer's Disease.”, J Alzheimers Dis, vol. 82, no. 4, pp. 1403-1415, 2021.
, “Soluble Amyloid-β Consumption in Alzheimer's Disease.”, J Alzheimers Dis, vol. 82, no. 4, pp. 1403-1415, 2021.
, “Soluble amyloid beta-protein dimers isolated from Alzheimer cortex directly induce Tau hyperphosphorylation and neuritic degeneration.”, Proc Natl Acad Sci U S A, vol. 108, no. 14, pp. 5819-24, 2011.
, “Soluble amyloid beta-protein dimers isolated from Alzheimer cortex directly induce Tau hyperphosphorylation and neuritic degeneration.”, Proc Natl Acad Sci U S A, vol. 108, no. 14, pp. 5819-24, 2011.
, “Socioeconomic Status and Dementia Risk Among Intensive Care Unit Survivors: Using National Health Insurance Cohort in Korea.”, J Alzheimers Dis, vol. 97, no. 1, pp. 273-281, 2024.
, “The Societal Cost of Dementia in Singapore: Results from the WiSE Study.”, J Alzheimers Dis, vol. 51, no. 2, pp. 439-49, 2016.
, “The Societal Cost of Dementia in Singapore: Results from the WiSE Study.”, J Alzheimers Dis, vol. 51, no. 2, pp. 439-49, 2016.
, “Social support may buffer the effect of ε4 allele on cognition in older adults.”, J Alzheimers Dis, vol. 102, no. 4, pp. 967-974, 2024.
, “Social Representation of Dementia: An Analysis of 5,792 Consecutive Cases Evaluated in a Memory Clinic.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1099-1108, 2017.
, “Social Representation of Dementia: An Analysis of 5,792 Consecutive Cases Evaluated in a Memory Clinic.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1099-1108, 2017.
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