Biblio

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2016
J. Haworth, Phillips, M., Newson, M., Rogers, P. J., Torrens-Burton, A., and Tales, A., “Measuring Information Processing Speed in Mild Cognitive Impairment: Clinical Versus Research Dichotomy.”, J Alzheimers Dis, vol. 51, no. 1, pp. 263-75, 2016.
S. Min Son, Shin, H. - J., Byun, J., Kook, S. Young, Moon, M., Chang, Y. Jin, and Mook-Jung, I., “Metformin Facilitates Amyloid-β Generation by β- and γ-Secretases via Autophagy Activation.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1197-208, 2016.
J. A. Luchsinger, Perez, T., Chang, H., Mehta, P., Steffener, J., Pradabhan, G., Ichise, M., Manly, J., Devanand, D. P., and Bagiella, E., “Metformin in Amnestic Mild Cognitive Impairment: Results of a Pilot Randomized Placebo Controlled Clinical Trial.”, J Alzheimers Dis, vol. 51, no. 2, pp. 501-14, 2016.
D. S Han, Boyle, P. A., James, B. D., Yu, L., and Bennett, D. A., “Mild Cognitive Impairment and Susceptibility to Scams in Old Age.”, J Alzheimers Dis, vol. 49, no. 3, pp. 845-51, 2016.
D. J. Quintana-Hernández, Miró-Barrachina, M. T., Ibáñez-Fernández, I. J., Del Pino, A. Santana-, Quintana-Montesdeoca, M. P., de Vera, B. Rodríguez, Morales-Casanova, D., Pérez-Vieitez, M. Del Carmen, Rodríguez-García, J., and Bravo-Caraduje, N., “Mindfulness in the Maintenance of Cognitive Capacities in Alzheimer's Disease: A Randomized Clinical Trial.”, J Alzheimers Dis, vol. 50, no. 1, pp. 217-32, 2016.
H. - H. Li, Lin, S. - L., Huang, C. - N., Lu, F. - J., Chiu, P. - Y., Huang, W. - N., Lai, T. - J., and Lin, C. - L., “miR-302 Attenuates Amyloid-β-Induced Neurotoxicity through Activation of Akt Signaling.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1083-98, 2016.
H. - H. Li, Lin, S. - L., Huang, C. - N., Lu, F. - J., Chiu, P. - Y., Huang, W. - N., Lai, T. - J., and Lin, C. - L., “miR-302 Attenuates Amyloid-β-Induced Neurotoxicity through Activation of Akt Signaling.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1083-98, 2016.
C. Stockburger, Miano, D., Baeumlisberger, M., Pallas, T., Arrey, T. N., Karas, M., Friedland, K., and Müller, W. E., “A Mitochondrial Role of SV2a Protein in Aging and Alzheimer's Disease: Studies with Levetiracetam.”, J Alzheimers Dis, vol. 50, no. 1, pp. 201-15, 2016.
C. Stockburger, Miano, D., Baeumlisberger, M., Pallas, T., Arrey, T. N., Karas, M., Friedland, K., and Müller, W. E., “A Mitochondrial Role of SV2a Protein in Aging and Alzheimer's Disease: Studies with Levetiracetam.”, J Alzheimers Dis, vol. 50, no. 1, pp. 201-15, 2016.
C. Stockburger, Miano, D., Baeumlisberger, M., Pallas, T., Arrey, T. N., Karas, M., Friedland, K., and Müller, W. E., “A Mitochondrial Role of SV2a Protein in Aging and Alzheimer's Disease: Studies with Levetiracetam.”, J Alzheimers Dis, vol. 50, no. 1, pp. 201-15, 2016.
M. Zhu, Huang, C., Ma, X., Wu, R., Zhu, W., Li, X., Liang, Z., Deng, F., Zhu, J., Xie, W., Yang, X., Jiang, Y., Wang, S., Wu, J., Geng, S., Xie, C., and Zhong, C., “Modulation of miR-19 in Aluminum-Induced Neural Cell Apoptosis.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1149-62, 2016.
M. Edwards, Hall, J., Williams, B., Johnson, L., and O'Bryant, S., “Molecular markers of amnestic mild cognitive impairment among Mexican Americans.”, J Alzheimers Dis, vol. 49, no. 1, pp. 221-8, 2016.
M. Chakrabarti, McDonald, A. J., J Reed, W., Moss, M. A., Das, B. C., and Ray, S. K., “Molecular Signaling Mechanisms of Natural and Synthetic Retinoids for Inhibition of Pathogenesis in Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 2, pp. 335-52, 2016.
C. -cui Yang, Kuai, X. -xian, bin Gao, W. -, Yu, J. -chun, Wang, Q., Li, L., and Zhang, L., “Morroniside-Induced PP2A Activation Antagonizes Tau Hyperphosphorylation in a Cellular Model of Neurodegeneration.”, J Alzheimers Dis, vol. 51, no. 1, pp. 33-44, 2016.
A. Ezra, Rabinovich-Nikitin, I., Rabinovich-Toidman, P., and Solomon, B., “Multifunctional Effect of Human Serum Albumin Reduces Alzheimer's Disease Related Pathologies in the 3xTg Mouse Model.”, J Alzheimers Dis, vol. 50, no. 1, pp. 175-88, 2016.
A. Ezra, Rabinovich-Nikitin, I., Rabinovich-Toidman, P., and Solomon, B., “Multifunctional Effect of Human Serum Albumin Reduces Alzheimer's Disease Related Pathologies in the 3xTg Mouse Model.”, J Alzheimers Dis, vol. 50, no. 1, pp. 175-88, 2016.
P. Eustache, Nemmi, F., Saint-Aubert, L., Pariente, J., and Péran, P., “Multimodal Magnetic Resonance Imaging in Alzheimer's Disease Patients at Prodromal Stage.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1035-50, 2016.
M. Frenkel-Pinter, Tal, S., Scherzer-Attali, R., Abu-Hussien, M., Alyagor, I., Eisenbaum, T., Gazit, E., and Segal, D., “Naphthoquinone-Tryptophan Hybrid Inhibits Aggregation of the Tau-Derived Peptide PHF6 and Reduces Neurotoxicity.”, J Alzheimers Dis, vol. 51, no. 1, pp. 165-78, 2016.
M. Frenkel-Pinter, Tal, S., Scherzer-Attali, R., Abu-Hussien, M., Alyagor, I., Eisenbaum, T., Gazit, E., and Segal, D., “Naphthoquinone-Tryptophan Hybrid Inhibits Aggregation of the Tau-Derived Peptide PHF6 and Reduces Neurotoxicity.”, J Alzheimers Dis, vol. 51, no. 1, pp. 165-78, 2016.
S. V. Ovsepian and O'Leary, V. B., “Neuronal activity and amyloid plaque pathology: an update.”, J Alzheimers Dis, vol. 49, no. 1, pp. 13-9, 2016.
S. Ramanan, Flanagan, E., Leyton, C. E., Villemagne, V. L., Rowe, C. C., Hodges, J. R., and Hornberger, M., “Non-Verbal Episodic Memory Deficits in Primary Progressive Aphasias are Highly Predictive of Underlying Amyloid Pathology.”, J Alzheimers Dis, vol. 51, no. 2, pp. 367-76, 2016.
C. Sanders, Behrens, S., Schwartz, S., Wengreen, H., Corcoran, C. D., Lyketsos, C. G., and Tschanz, J. A. T., “Nutritional Status is Associated with Faster Cognitive Decline and Worse Functional Impairment in the Progression of Dementia: The Cache County Dementia Progression Study1.”, J Alzheimers Dis, vol. 52, no. 1, pp. 33-42, 2016.
A. Oulhaj, Jernerén, F., Refsum, H., A Smith, D., and de Jager, C. A., “Omega-3 Fatty Acid Status Enhances the Prevention of Cognitive Decline by B Vitamins in Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 50, no. 2, pp. 547-57, 2016.
P. Banerjee, Sahoo, A., Anand, S., Bir, A., and Chakrabarti, S., “The Oral Iron Chelator, Deferasirox, Reverses the Age-Dependent Alterations in Iron and Amyloid-β Homeostasis in Rat Brain: Implications in the Therapy of Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 3, pp. 681-93, 2016.
P. Banerjee, Sahoo, A., Anand, S., Bir, A., and Chakrabarti, S., “The Oral Iron Chelator, Deferasirox, Reverses the Age-Dependent Alterations in Iron and Amyloid-β Homeostasis in Rat Brain: Implications in the Therapy of Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 3, pp. 681-93, 2016.

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