Biblio
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“tRNA-Derived Fragments in Alzheimer's Disease: Implications for New Disease Biomarkers and Neuropathological Mechanisms.”, J Alzheimers Dis, vol. 79, no. 2, pp. 793-806, 2021.
, “tRNA-Derived Fragments in Alzheimer's Disease: Implications for New Disease Biomarkers and Neuropathological Mechanisms.”, J Alzheimers Dis, vol. 79, no. 2, pp. 793-806, 2021.
, “Soluble TREM2, Alzheimer's Disease Pathology, and Risk for Progression of Cerebral Small Vessel Disease: A Longitudinal Study.”, J Alzheimers Dis, vol. 92, no. 1, pp. 311-322, 2023.
, “Distinct Patterns of Cognitive Aging Modified by Education Level and Gender among Adults with Limited or No Formal Education: A Normative Study of the Mini-Mental State Examination.”, J Alzheimers Dis, vol. 49, no. 4, pp. 961-9, 2016.
, “UCH-L1 Inhibition Decreases the Microtubule-Binding Function of Tau Protein.”, J Alzheimers Dis, vol. 49, no. 2, pp. 353-63, 2016.
, “Distinct Patterns of Cognitive Aging Modified by Education Level and Gender among Adults with Limited or No Formal Education: A Normative Study of the Mini-Mental State Examination.”, J Alzheimers Dis, vol. 49, no. 4, pp. 961-9, 2016.
, “Iron Regulates Apolipoprotein E Expression and Secretion in Neurons and Astrocytes.”, J Alzheimers Dis, vol. 51, no. 2, pp. 471-87, 2016.
, “Iron Regulates Apolipoprotein E Expression and Secretion in Neurons and Astrocytes.”, J Alzheimers Dis, vol. 51, no. 2, pp. 471-87, 2016.
, “Iron Regulates Apolipoprotein E Expression and Secretion in Neurons and Astrocytes.”, J Alzheimers Dis, vol. 51, no. 2, pp. 471-87, 2016.
, “The Retinal Vessel Density Can Reflect Cognitive Function in Patients with Alzheimer's Disease: Evidence from Optical Coherence Tomography Angiography.”, J Alzheimers Dis, vol. 79, no. 3, pp. 1307-1316, 2021.
, “Morroniside-Induced PP2A Activation Antagonizes Tau Hyperphosphorylation in a Cellular Model of Neurodegeneration.”, J Alzheimers Dis, vol. 51, no. 1, pp. 33-44, 2016.
, “Metabolic Syndrome and Mild Cognitive Impairment: A Case-Control Study among Elderly in a Shanghai Suburb.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1175-82, 2016.
, “Serotonin 2A Receptor Inverse Agonist as a Treatment for Parkinson's Disease Psychosis: A Systematic Review and Meta-analysis of Serotonin 2A Receptor Negative Modulators.”, J Alzheimers Dis, vol. 50, no. 3, pp. 733-40, 2016.
, “Serotonin 2A Receptor Inverse Agonist as a Treatment for Parkinson's Disease Psychosis: A Systematic Review and Meta-analysis of Serotonin 2A Receptor Negative Modulators.”, J Alzheimers Dis, vol. 50, no. 3, pp. 733-40, 2016.
, “Shared Genetic Risk Factors for Late-Life Depression and Alzheimer's Disease.”, J Alzheimers Dis, vol. 52, no. 1, pp. 1-15, 2016.
, “Unstable Body Mass Index and Progression to Probable Alzheimer's Disease Dementia in Patients with Amnestic Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 49, no. 2, pp. 483-91, 2016.
, “SIRT1 Deacetylates SC35 and Suppresses Its Function in Tau Exon 10 Inclusion.”, J Alzheimers Dis, vol. 61, no. 2, pp. 561-570, 2018.
, “The Korean Size/Weight Attribute Test: A Semantic Knowledge Test for Korean Older Adults and Brain-Imaging Evidence.”, J Alzheimers Dis, vol. 49, no. 2, pp. 377-86, 2016.
, “The Korean Size/Weight Attribute Test: A Semantic Knowledge Test for Korean Older Adults and Brain-Imaging Evidence.”, J Alzheimers Dis, vol. 49, no. 2, pp. 377-86, 2016.
, “Predictive factors for disease progression in patients with early-onset Alzheimer's disease.”, J Alzheimers Dis, vol. 49, no. 1, pp. 85-91, 2016.
, “Lentivirus-Mediated HDAC3 Inhibition Attenuates Oxidative Stress in APPswe/PS1dE9 Mice.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1411-1424, 2018.
, “Alzheimer's Disease: Recent Concepts on the Relation of Mitochondrial Disturbances, Excitotoxicity, Neuroinflammation, and Kynurenines.”, J Alzheimers Dis, vol. 62, no. 2, pp. 523-547, 2018.
, “Alzheimer's Disease: Recent Concepts on the Relation of Mitochondrial Disturbances, Excitotoxicity, Neuroinflammation, and Kynurenines.”, J Alzheimers Dis, vol. 62, no. 2, pp. 523-547, 2018.
, “Serum C-Peptide, Visfatin, Resistin, and Ghrelin are Altered in Sporadic and GRN-Associated Frontotemporal Lobar Degeneration.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1053-1060, 2018.
, “Chronic Obstructive Pulmonary Disease as a Risk Factor for Cognitive Dysfunction: A Meta-Analysis of Current Studies.”, J Alzheimers Dis, vol. 52, no. 1, pp. 101-11, 2016.
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