Biblio
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“Blood Inflammatory Mediators and Cognitive Decline in Alzheimer's Disease: A Two Years Longitudinal Study.”, J Alzheimers Dis, vol. 63, no. 1, pp. 87-92, 2018.
, “Blood Inflammatory Mediators and Cognitive Decline in Alzheimer's Disease: A Two Years Longitudinal Study.”, J Alzheimers Dis, vol. 63, no. 1, pp. 87-92, 2018.
, “Can 11C-PiB-PET Relative Delivery R1 or 11C-PiB-PET Perfusion Replace 18F-FDG-PET in the Assessment of Brain Neurodegeneration?”, J Alzheimers Dis, vol. 65, no. 1, pp. 89-97, 2018.
, “Cardiorespiratory Fitness and Cognitive Function are Positively Related Among Participants with Mild and Subjective Cognitive Impairment.”, J Alzheimers Dis, vol. 62, no. 4, pp. 1865-1875, 2018.
, “Cardiorespiratory Fitness and Cognitive Function are Positively Related Among Participants with Mild and Subjective Cognitive Impairment.”, J Alzheimers Dis, vol. 62, no. 4, pp. 1865-1875, 2018.
, “Cardiorespiratory Fitness and White Matter Neuronal Fiber Integrity in Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 61, no. 2, pp. 729-739, 2018.
, “Caregiving and Caregiver Burden in Dementia Home Care: Results from the Prospective Dementia Registry (PRODEM) of the Austrian Alzheimer Society.”, J Alzheimers Dis, vol. 63, no. 1, pp. 103-114, 2018.
, “Cerebral Amyloid Angiopathy and Cerebral Amyloid Angiopathy-Related Inflammation: Comparison of Hemorrhagic and DWI MRI Features.”, J Alzheimers Dis, vol. 64, no. 4, pp. 1113-1121, 2018.
, “Cerebrospinal Fluid Levels of Angiotensin-Converting Enzyme Are Associated with Amyloid-β42 Burden in Alzheimer's Disease.”, J Alzheimers Dis, vol. 64, no. 4, pp. 1085-1090, 2018.
, “Cerebrospinal Fluid Levels of Angiotensin-Converting Enzyme Are Associated with Amyloid-β42 Burden in Alzheimer's Disease.”, J Alzheimers Dis, vol. 64, no. 4, pp. 1085-1090, 2018.
, “Cerebrospinal Fluid, MRI, and Florbetaben-PET in Cerebral Amyloid Angiopathy-Related Inflammation.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1107-1117, 2018.
, “Cerebrovascular Disease and Neurodegeneration in Alzheimer's Disease with and without a Strong Family History: A Pilot Magnetic Resonance Imaging Study in Dominican Republic.”, J Alzheimers Dis, vol. 66, no. 4, pp. 1519-1528, 2018.
, “Cerebrovascular Disease and Neurodegeneration in Alzheimer's Disease with and without a Strong Family History: A Pilot Magnetic Resonance Imaging Study in Dominican Republic.”, J Alzheimers Dis, vol. 66, no. 4, pp. 1519-1528, 2018.
, “CFH and ARMS2 Polymorphisms Interact with Zinc Supplements in Cognitive Impairment in the Women's Health Initiative Hormone Trial.”, J Alzheimers Dis, vol. 66, no. 2, pp. 707-715, 2018.
, “Childhood Lead Exposure and Adult Neurodegenerative Disease.”, J Alzheimers Dis, vol. 64, no. 1, pp. 17-42, 2018.
, “Circulating, Cell-Free Micro-RNA Profiles Reflect Discordant Development of Dementia in Monozygotic Twins.”, J Alzheimers Dis, vol. 63, no. 2, pp. 591-601, 2018.
, “Circulating Plasma microRNAs are Altered with Amyloidosis in a Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 66, no. 2, pp. 835-852, 2018.
, “Circulating Progenitor Cells Correlate with Memory, Posterior Cortical Thickness, and Hippocampal Perfusion.”, J Alzheimers Dis, vol. 61, no. 1, pp. 91-101, 2018.
, “Clinical Subtypes of Dementia with Lewy Bodies Based on the Initial Clinical Presentation.”, J Alzheimers Dis, vol. 64, no. 2, pp. 505-513, 2018.
, “Clinical Trials for Disease-Modifying Therapies in Alzheimer's Disease: A Primer, Lessons Learned, and a Blueprint for the Future.”, J Alzheimers Dis, vol. 64, no. s1, pp. S3-S22, 2018.
, “Clinical-Neuropathological Correlations of Alzheimer's Disease and Related Dementias in Latino Volunteers.”, J Alzheimers Dis, vol. 66, no. 4, pp. 1539-1548, 2018.
, “Clinic-Based Validation of Cerebrospinal Fluid Biomarkers with Florbetapir PET for Diagnosis of Dementia.”, J Alzheimers Dis, vol. 61, no. 1, pp. 135-143, 2018.
, “Co-Expression of Glia Maturation Factor and Apolipoprotein E4 in Alzheimer's Disease Brain.”, J Alzheimers Dis, vol. 61, no. 2, pp. 553-560, 2018.
, “Co-Expression of Glia Maturation Factor and Apolipoprotein E4 in Alzheimer's Disease Brain.”, J Alzheimers Dis, vol. 61, no. 2, pp. 553-560, 2018.
, “Combining Cognitive, Genetic, and Structural Neuroimaging Markers to Identify Individuals with Increased Dementia Risk.”, J Alzheimers Dis, vol. 64, no. 2, pp. 533-542, 2018.
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