Biblio
“Endogenous Murine Amyloid-β Peptide Assembles into Aggregates in the Aged C57BL/6J Mouse Suggesting These Animals as a Model to Study Pathogenesis of Amyloid-β Plaque Formation.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1425-1450, 2018.
, “Erythromyeloid-Derived TREM2: A Major Determinant of Alzheimer's Disease Pathology in Down Syndrome.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1143-1162, 2018.
, “Free Heme and Amyloid-β: A Fatal Liaison in Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 3, pp. 963-984, 2018.
, “Gradual Cerebral Hypoperfusion Impairs Fear Conditioning and Object Recognition Learning and Memory in Mice: Potential Roles of Neurodegeneration and Cholinergic Dysfunction.”, J Alzheimers Dis, vol. 61, no. 1, pp. 283-293, 2018.
, “Humanin Exerts Neuroprotection During Cardiac Ischemia-Reperfusion Injury.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1343-1353, 2018.
, “The Impact of APOE ɛ4 in Alzheimer's Disease Differs According to Age.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1377-1385, 2018.
, “In vivo Depiction of α7 Nicotinic Receptor Loss for Cognitive Decline in Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1355-1365, 2018.
, “In Vivo Visualization of Tau Accumulation, Microglial Activation, and Brain Atrophy in a Mouse Model of Tauopathy rTg4510.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1037-1052, 2018.
, “A Long Journey into Aging, Brain Aging, and Alzheimer's Disease Following the Oxidative Stress Tracks.”, J Alzheimers Dis, vol. 62, no. 3, pp. 1319-1335, 2018.
, “Longitudinal Changes in Serum Glucose Levels are Associated with Metabolic Changes in Alzheimer's Disease Related Brain Regions.”, J Alzheimers Dis, vol. 62, no. 2, pp. 833-840, 2018.
, “Mental States in Moving Shapes: Distinct Cortical and Subcortical Contributions to Theory of Mind Impairments in Dementia.”, J Alzheimers Dis, vol. 61, no. 2, pp. 521-535, 2018.
, “Mesenchymal Stem Cell-Derived Extracellular Vesicles Suppresses iNOS Expression and Ameliorates Neural Impairment in Alzheimer's Disease Mice.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1005-1013, 2018.
, “Mitochondrial Translocase of the Outer Membrane Alterations May Underlie Dysfunctional Oxidative Phosphorylation in Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 2, pp. 793-801, 2018.
, “A Novel Antibody Targeting Tau Phosphorylated at Serine 235 Detects Neurofibrillary Tangles.”, J Alzheimers Dis, vol. 61, no. 3, pp. 899-905, 2018.
, “Prediction of Alzheimer's Dementia in Patients with Amnestic Mild Cognitive Impairment in Clinical Routine: Incremental Value of Biomarkers of Neurodegeneration and Brain Amyloidosis Added Stepwise to Cognitive Status.”, J Alzheimers Dis, vol. 61, no. 1, pp. 373-388, 2018.
, “Progression of Alzheimer's Disease-Related Pathology and Cell Counts in a Patient with Idiopathic Normal Pressure Hydrocephalus.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1451-1462, 2018.
, “Rationale and Design of the Mechanistic Potential of Antihypertensives in Preclinical Alzheimer's (HEART) Trial.”, J Alzheimers Dis, vol. 61, no. 2, pp. 815-824, 2018.
, “The Rationale and Design of the Reducing Pathology in Alzheimer's Disease through Angiotensin TaRgeting (RADAR) Trial.”, J Alzheimers Dis, vol. 61, no. 2, pp. 803-814, 2018.
, “The Relevance of Iron in the Pathogenesis of Multiple System Atrophy: A Viewpoint.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1253-1273, 2018.
, “Revolution of Alzheimer Precision Neurology. Passageway of Systems Biology and Neurophysiology.”, J Alzheimers Dis, vol. 64, no. s1, pp. S47-S105, 2018.
, “Roles of Mitochondrial 17β-Hydroxysteroid Dehydrogenase Type 10 in Alzheimer's Disease.”, J Alzheimers Dis, vol. 62, no. 2, pp. 665-673, 2018.
, “SPARCL1 Accelerates Symptom Onset in Alzheimer's Disease and Influences Brain Structure and Function During Aging.”, J Alzheimers Dis, vol. 61, no. 1, pp. 401-414, 2018.
, “Synthetic Fragment of Receptor for Advanced Glycation End Products Prevents Memory Loss and Protects Brain Neurons in Olfactory Bulbectomized Mice.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1061-1076, 2018.
, “Tauopathies: Mechanisms and Therapeutic Strategies.”, J Alzheimers Dis, vol. 61, no. 2, pp. 487-508, 2018.
, “VEGFR1 and VEGFR2 in Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 2, pp. 741-752, 2018.
,