Biblio
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Author Title [ Type] Year Filters: First Letter Of Keyword is G [Clear All Filters]
“Progression of Alzheimer's Disease-Related Pathology and Cell Counts in a Patient with Idiopathic Normal Pressure Hydrocephalus.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1451-1462, 2018.
, “Profiling of Specific Gene Expression Pathways in Peripheral Cells from Prodromal Alzheimer's Disease Patients.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1289-1294, 2018.
, “PRNP P39L Variant is a Rare Cause of Frontotemporal Dementia in Italian Population.”, J Alzheimers Dis, vol. 50, no. 2, pp. 353-7, 2016.
, “Prion Protein-Hemin Interaction Upregulates Hemoglobin Synthesis: Implications for Cerebral Hemorrhage and Sporadic Creutzfeldt-Jakob Disease.”, J Alzheimers Dis, vol. 51, no. 1, pp. 107-21, 2016.
, “Prevalence of Delirium in a Population of Elderly Outpatients with Dementia: A Retrospective Study.”, J Alzheimers Dis, vol. 61, no. 1, pp. 251-257, 2018.
, “The Presence of Select Tau Species in Human Peripheral Tissues and Their Relation to Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 2, pp. 345-56, 2016.
, “Predictive factors for disease progression in patients with early-onset Alzheimer's disease.”, J Alzheimers Dis, vol. 49, no. 1, pp. 85-91, 2016.
, “Plasma Glial Fibrillary Acidic Protein Is Associated with 18F-SMBT-1 PET: Two Putative Astrocyte Reactivity Biomarkers for Alzheimer's Disease.”, J Alzheimers Dis, vol. 92, no. 2, pp. 615-628, 2023.
, “Physical Activity and Hippocampal Sub-Region Structure in Older Adults with Memory Complaints.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1089-1096, 2018.
, “Pharmacological Strategies to Improve Dendritic Spines in Alzheimer's Disease.”, J Alzheimers Dis, vol. 82, no. s1, pp. S91-S107, 2021.
, “A Pathway Based Classification Method for Analyzing Gene Expression for Alzheimer's Disease Diagnosis.”, J Alzheimers Dis, vol. 49, no. 3, pp. 659-69, 2016.
, “A Pathway Based Classification Method for Analyzing Gene Expression for Alzheimer's Disease Diagnosis.”, J Alzheimers Dis, vol. 49, no. 3, pp. 659-69, 2016.
, “Palmitate Increases β-site AβPP-Cleavage Enzyme 1 Activity and Amyloid-β Genesis by Evoking Endoplasmic Reticulum Stress and Subsequent C/EBP Homologous Protein Activation.”, J Alzheimers Dis, vol. 57, no. 3, pp. 907-925, 2017.
, “Overexpression of Metallothionein-1 Modulates the Phenotype of the Tg2576 Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 1, pp. 81-95, 2016.
, “The Oral Iron Chelator, Deferasirox, Reverses the Age-Dependent Alterations in Iron and Amyloid-β Homeostasis in Rat Brain: Implications in the Therapy of Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 3, pp. 681-93, 2016.
, “An Optimized Combination of Ginger and Peony Root Effectively Inhibits Amyloid-β Accumulation and Amyloid-β-Mediated Pathology in AβPP/PS1 Double-Transgenic Mice.”, J Alzheimers Dis, vol. 50, no. 1, pp. 189-200, 2016.
, “An Optimized Combination of Ginger and Peony Root Effectively Inhibits Amyloid-β Accumulation and Amyloid-β-Mediated Pathology in AβPP/PS1 Double-Transgenic Mice.”, J Alzheimers Dis, vol. 50, no. 1, pp. 189-200, 2016.
, “Optimization of Statistical Single Subject Analysis of Brain FDG PET for the Prognosis of Mild Cognitive Impairment-to-Alzheimer's Disease Conversion.”, J Alzheimers Dis, vol. 49, no. 4, pp. 945-59, 2016.
, “A Novel Strategy for Alzheimer's Disease Based on the Regulatory Effect of Amyloid-β on Gut Flora.”, J Alzheimers Dis, vol. 94, no. s1, pp. S227-S239, 2023.
, “Novel Selective Calpain 1 Inhibitors as Potential Therapeutics in Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 3, pp. 707-21, 2016.
, “Neuronal basis of age-related working memory decline.”, Nature, vol. 476, no. 7359, pp. 210-3, 2011.
, “Neuroinflammation, hyperphosphorylated tau, diffuse amyloid plaques, and down-regulation of the cellular prion protein in air pollution exposed children and young adults.”, J Alzheimers Dis, vol. 28, no. 1, pp. 93-107, 2012.
, “A Network of Genetic Effects on Non-Demented Cognitive Aging: Alzheimer's Genetic Risk (CLU + CR1 + PICALM) Intensifies Cognitive Aging Genetic Risk (COMT + BDNF) Selectively for APOEɛ4 Carriers.”, J Alzheimers Dis, vol. 62, no. 2, pp. 887-900, 2018.
, “The Need to Separate Chronic Traumatic Encephalopathy Neuropathology from Clinical Features.”, J Alzheimers Dis, vol. 61, no. 1, pp. 17-28, 2018.
, “Mutation Screening of the CHCHD2 Gene for Alzheimer's Disease and Frontotemporal Dementia in Chinese Mainland Population.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1283-1288, 2018.
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