Biblio
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Author Title [ Type] Year Filters: Keyword is Alzheimer Disease [Clear All Filters]
“tRNA-Derived Fragments in Alzheimer's Disease: Implications for New Disease Biomarkers and Neuropathological Mechanisms.”, J Alzheimers Dis, vol. 79, no. 2, pp. 793-806, 2021.
, “Two Phase 2 Multiple Ascending-Dose Studies of Vanutide Cridificar (ACC-001) and QS-21 Adjuvant in Mild-to-Moderate Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1131-43, 2016.
, “Two phase 3 trials of bapineuzumab in mild-to-moderate Alzheimer's disease.”, N Engl J Med, vol. 370, no. 4, pp. 322-33, 2014.
, “Ubisol-Q10 (a Nanomicellar Water-Soluble Formulation of CoQ10) Treatment Inhibits Alzheimer-Type Behavioral and Pathological Symptoms in a Double Transgenic Mouse (TgAPEswe, PSEN1dE9) Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 221-236, 2018.
, “The Uniform Data Set, Czech Version: Normative Data in Older Adults from an International Perspective.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1233-1240, 2018.
, “Unlocking Modifiable Risk Factors for Alzheimer's Disease: Does the Oral Microbiome Hold Some of the Keys?”, J Alzheimers Dis, vol. 92, no. 4, pp. 1111-1129, 2023.
, “Unraveling Alzheimer's: Making Sense of the Relationship between Diabetes and Alzheimer's Disease1.”, J Alzheimers Dis, vol. 51, no. 4, pp. 961-77, 2016.
, “Unstable Body Mass Index and Progression to Probable Alzheimer's Disease Dementia in Patients with Amnestic Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 49, no. 2, pp. 483-91, 2016.
, “Untangling the Role of TREM2 in Conjugation with Microglia in Neuronal Dysfunction: A Hypothesis on a Novel Pathway in the Pathophysiology of Alzheimer's Disease.”, J Alzheimers Dis, vol. 94, no. s1, pp. S319-S333, 2023.
, “Upregulation of Cortical A2A Adenosine Receptors Is Reflected in Platelets of Patients with Alzheimer's Disease.”, J Alzheimers Dis, vol. 80, no. 3, pp. 1105-1117, 2021.
, “Usefulness of an Integrated Analysis of Different Memory Tasks to Predict the Progression from Mild Cognitive Impairment to Alzheimer's Disease: The Episodic Memory Score (EMS).”, J Alzheimers Dis, vol. 50, no. 1, pp. 61-70, 2016.
, “Vacuolar Protein-Sorting Proteins Are Reduced Even Before Cognitive Decline in a Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 96, no. 3, pp. 1011-1017, 2023.
, “Validation of the Delayed Matching-to-Sample Task 48 (DMS48) in Elderly Chinese.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1611-1618, 2018.
, “Validation of the Spanish version of the Baylor Profound Mental Status Examination.”, J Alzheimers Dis, vol. 49, no. 1, pp. 73-8, 2016.
, “The Vanderbilt Memory & Aging Project: Study Design and Baseline Cohort Overview.”, J Alzheimers Dis, vol. 52, no. 2, pp. 539-59, 2016.
, “Vascular risk factors promote conversion from mild cognitive impairment to Alzheimer disease.”, Neurology, vol. 76, no. 17, pp. 1485-91, 2011.
, “VEGFR1 and VEGFR2 in Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 2, pp. 741-752, 2018.
, “Vestibular Loss Predicts Poorer Spatial Cognition in Patients with Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 3, pp. 995-1003, 2018.
, “Volumetric and shape analysis of the thalamus and striatum in amnestic mild cognitive impairment.”, J Alzheimers Dis, vol. 49, no. 1, pp. 237-49, 2016.
, “What are the Most Frequently Impaired Markers of Neurodegeneration in ADNI Subjects?”, J Alzheimers Dis, vol. 51, no. 3, pp. 793-800, 2016.
, “What Drives Country Differences in Cost of Alzheimer's Disease? An Explanation from Resource Use in the GERAS Study.”, J Alzheimers Dis, vol. 57, no. 3, pp. 797-812, 2017.
, “When Art Meets Gardens: Does It Enhance the Benefits? The Nancy Hypothesis of Care for Persons with Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 3, pp. 885-898, 2018.
, “White Matter Abnormalities Track Disease Progression in PSEN1 Autosomal Dominant Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 3, pp. 827-35, 2016.
, “White Matter Changes are Associated with Ventricular Expansion in Aging, Mild Cognitive Impairment, and Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 2, pp. 329-42, 2016.
, “α-Lipoic Acid Has the Potential to Normalize Copper Metabolism, Which Is Dysregulated in Alzheimer's Disease.”, J Alzheimers Dis, vol. 85, no. 2, pp. 715-728, 2022.
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