Biblio
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“Genetic Interaction with Plasma Lipids on Alzheimer's Disease in the Framingham Heart Study.”, J Alzheimers Dis, vol. 66, no. 3, pp. 1275-1282, 2018.
, “Genetic Risk as a Marker of Amyloid-β and Tau Burden in Cerebrospinal Fluid.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1417-1427, 2017.
, “Genetic Risk for Age-Related Cognitive Impairment Does Not Predict Cognitive Performance in Middle Age.”, J Alzheimers Dis, vol. 64, no. 2, pp. 459-471, 2018.
, “Genetic Stratification to Identify Risk Groups for Alzheimer's Disease.”, J Alzheimers Dis, vol. 57, no. 1, pp. 275-283, 2017.
, “Genome-Wide Methylation of Mild Cognitive Impairment in Mexican Americans Highlights Genes Involved in Synaptic Transport, Alzheimer's Disease-Precursor Phenotypes, and Metabolic Morbidities.”, J Alzheimers Dis, vol. 72, no. 3, pp. 733-749, 2019.
, “Glia Maturation Factor and Mast Cell-Dependent Expression of Inflammatory Mediators and Proteinase Activated Receptor-2 in Neuroinflammation.”, J Alzheimers Dis, vol. 66, no. 3, pp. 1117-1129, 2018.
, “Glial Fibrillary Acidic Protein in Serum is Increased in Alzheimer's Disease and Correlates with Cognitive Impairment.”, J Alzheimers Dis, vol. 67, no. 2, pp. 481-488, 2019.
, “Glial Fibrillary Acidic Protein in Serum is Increased in Alzheimer's Disease and Correlates with Cognitive Impairment.”, J Alzheimers Dis, vol. 67, no. 2, pp. 481-488, 2019.
, “Global Metabolic Shifts in Age and Alzheimer's Disease Mouse Brains Pivot at NAD +/NADH Redox Sites.”, J Alzheimers Dis, vol. 71, no. 1, pp. 119-140, 2019.
, “Glucometabolic Changes Are Associated with Structural Gray Matter Alterations in Prodromal Dementia.”, J Alzheimers Dis, vol. 89, no. 4, pp. 1293-1302, 2022.
, “Greater Regional Cortical Thickness is Associated with Selective Vulnerability to Atrophy in Alzheimer's Disease, Independent of Amyloid Load and APOE Genotype.”, J Alzheimers Dis, vol. 69, no. 1, pp. 145-156, 2019.
, “Greater specificity for cerebrospinal fluid P-tau231 over P-tau181 in the differentiation of healthy controls from Alzheimer's disease.”, J Alzheimers Dis, vol. 49, no. 1, pp. 93-100, 2016.
, “Greater specificity for cerebrospinal fluid P-tau231 over P-tau181 in the differentiation of healthy controls from Alzheimer's disease.”, J Alzheimers Dis, vol. 49, no. 1, pp. 93-100, 2016.
, “Halting of Caspase Activity Protects Tau from MC1-Conformational Change and Aggregation.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1521-1538, 2016.
, “Health-Related Quality of Life in Patients with Alzheimer's Disease in Different German Health Care Settings.”, J Alzheimers Dis, vol. 51, no. 2, pp. 545-61, 2016.
, “Health-Related Quality of Life in Patients with Alzheimer's Disease in Different German Health Care Settings.”, J Alzheimers Dis, vol. 51, no. 2, pp. 545-61, 2016.
, “Hedonic Assessment of Odors: A Comparison of Two Sensory Scales for Use with Alzheimer's Disease Patients and Elderly Individuals.”, J Alzheimers Dis, vol. 61, no. 3, pp. 929-938, 2018.
, “Hedonic Assessment of Odors: A Comparison of Two Sensory Scales for Use with Alzheimer's Disease Patients and Elderly Individuals.”, J Alzheimers Dis, vol. 61, no. 3, pp. 929-938, 2018.
, “Hierarchical Distribution of the Tau Cytoskeletal Pathology in the Thalamus of Alzheimer's Disease Patients.”, J Alzheimers Dis, vol. 49, no. 4, pp. 905-15, 2016.
, “Hierarchical Distribution of the Tau Cytoskeletal Pathology in the Thalamus of Alzheimer's Disease Patients.”, J Alzheimers Dis, vol. 49, no. 4, pp. 905-15, 2016.
, “High Sensitivity Cardiac Troponin T and Cognitive Function in the Oldest Old: The Leiden 85-Plus Study.”, J Alzheimers Dis, vol. 60, no. 1, pp. 235-242, 2017.
, “High Soluble Amyloid-β42 Predicts Normal Cognition in Amyloid-Positive Individuals with Alzheimer's Disease-Causing Mutations.”, J Alzheimers Dis, vol. 90, no. 1, pp. 333-348, 2022.
, “High Soluble Amyloid-β42 Predicts Normal Cognition in Amyloid-Positive Individuals with Alzheimer's Disease-Causing Mutations.”, J Alzheimers Dis, vol. 90, no. 1, pp. 333-348, 2022.
, “A High-Cholesterol Diet Increases 27-Hydroxycholesterol and Modifies Estrogen Receptor Expression and Neurodegeneration in Rabbit Hippocampus.”, J Alzheimers Dis, vol. 56, no. 1, pp. 185-196, 2017.
, “Higher Blood Vitamin C Levels are Associated with Reduction of Apolipoprotein E E4-related Risks of Cognitive Decline in Women: The Nakajima Study.”, J Alzheimers Dis, vol. 63, no. 4, pp. 1289-1297, 2018.
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