Biblio
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“Human apoE isoforms differentially regulate brain amyloid-β peptide clearance.”, Sci Transl Med, vol. 3, no. 89, p. 89ra57, 2011.
, “How Do Persons with Young and Late Onset Dementia Die?”, J Alzheimers Dis, vol. 81, no. 2, pp. 843-852, 2021.
, “How Do Persons with Young and Late Onset Dementia Die?”, J Alzheimers Dis, vol. 81, no. 2, pp. 843-852, 2021.
, “How Do Persons with Young and Late Onset Dementia Die?”, J Alzheimers Dis, vol. 81, no. 2, pp. 843-852, 2021.
, “Home-Based Exercise Program Improves Balance and Fear of Falling in Community-Dwelling Older Adults with Mild Alzheimer's Disease: A Pilot Study.”, J Alzheimers Dis, vol. 59, no. 2, pp. 565-574, 2017.
, “HMGCS2-Induced Autophagic Degradation of Tau Involves Ketone Body and ANKRD24.”, J Alzheimers Dis, vol. 91, no. 1, pp. 407-426, 2022.
, “Histogram-Based Feature Extraction from Individual Gray Matter Similarity-Matrix for Alzheimer's Disease Classification.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1571-1582, 2017.
, “Hippocampal Lipid Homeostasis in APP/PS1 Mice is Modulated by a Complex Interplay Between Dietary DHA and Estrogens: Relevance for Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 2, pp. 459-81, 2016.
, “High-Throughput Screening for Identification of Blood-Brain Barrier Integrity Enhancers: A Drug Repurposing Opportunity to Rectify Vascular Amyloid Toxicity.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1499-516, 2016.
, “Higher Body Mass Index Is Associated with Lower Cortical Amyloid-β Burden in Cognitively Normal Individuals in Late-Life.”, J Alzheimers Dis, vol. 69, no. 3, pp. 817-827, 2019.
, “Higher Blood Vitamin C Levels are Associated with Reduction of Apolipoprotein E E4-related Risks of Cognitive Decline in Women: The Nakajima Study.”, J Alzheimers Dis, vol. 63, no. 4, pp. 1289-1297, 2018.
, “A High-Cholesterol Diet Increases 27-Hydroxycholesterol and Modifies Estrogen Receptor Expression and Neurodegeneration in Rabbit Hippocampus.”, J Alzheimers Dis, vol. 56, no. 1, pp. 185-196, 2017.
, “High Soluble Amyloid-β42 Predicts Normal Cognition in Amyloid-Positive Individuals with Alzheimer's Disease-Causing Mutations.”, J Alzheimers Dis, vol. 90, no. 1, pp. 333-348, 2022.
, “High Soluble Amyloid-β42 Predicts Normal Cognition in Amyloid-Positive Individuals with Alzheimer's Disease-Causing Mutations.”, J Alzheimers Dis, vol. 90, no. 1, pp. 333-348, 2022.
, “High Sensitivity Cardiac Troponin T and Cognitive Function in the Oldest Old: The Leiden 85-Plus Study.”, J Alzheimers Dis, vol. 60, no. 1, pp. 235-242, 2017.
, “Hierarchical Distribution of the Tau Cytoskeletal Pathology in the Thalamus of Alzheimer's Disease Patients.”, J Alzheimers Dis, vol. 49, no. 4, pp. 905-15, 2016.
, “Hierarchical Distribution of the Tau Cytoskeletal Pathology in the Thalamus of Alzheimer's Disease Patients.”, J Alzheimers Dis, vol. 49, no. 4, pp. 905-15, 2016.
, “Hedonic Assessment of Odors: A Comparison of Two Sensory Scales for Use with Alzheimer's Disease Patients and Elderly Individuals.”, J Alzheimers Dis, vol. 61, no. 3, pp. 929-938, 2018.
, “Hedonic Assessment of Odors: A Comparison of Two Sensory Scales for Use with Alzheimer's Disease Patients and Elderly Individuals.”, J Alzheimers Dis, vol. 61, no. 3, pp. 929-938, 2018.
, “Health-Related Quality of Life in Patients with Alzheimer's Disease in Different German Health Care Settings.”, J Alzheimers Dis, vol. 51, no. 2, pp. 545-61, 2016.
, “Health-Related Quality of Life in Patients with Alzheimer's Disease in Different German Health Care Settings.”, J Alzheimers Dis, vol. 51, no. 2, pp. 545-61, 2016.
, “Halting of Caspase Activity Protects Tau from MC1-Conformational Change and Aggregation.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1521-1538, 2016.
, “Greater specificity for cerebrospinal fluid P-tau231 over P-tau181 in the differentiation of healthy controls from Alzheimer's disease.”, J Alzheimers Dis, vol. 49, no. 1, pp. 93-100, 2016.
, “Greater specificity for cerebrospinal fluid P-tau231 over P-tau181 in the differentiation of healthy controls from Alzheimer's disease.”, J Alzheimers Dis, vol. 49, no. 1, pp. 93-100, 2016.
, “Greater Regional Cortical Thickness is Associated with Selective Vulnerability to Atrophy in Alzheimer's Disease, Independent of Amyloid Load and APOE Genotype.”, J Alzheimers Dis, vol. 69, no. 1, pp. 145-156, 2019.
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