Biblio
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“Physical Activity and Hippocampal Sub-Region Structure in Older Adults with Memory Complaints.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1089-1096, 2018.
, “Prevalence of Delirium in a Population of Elderly Outpatients with Dementia: A Retrospective Study.”, J Alzheimers Dis, vol. 61, no. 1, pp. 251-257, 2018.
, “Profiling of Specific Gene Expression Pathways in Peripheral Cells from Prodromal Alzheimer's Disease Patients.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1289-1294, 2018.
, “Progression of Alzheimer's Disease-Related Pathology and Cell Counts in a Patient with Idiopathic Normal Pressure Hydrocephalus.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1451-1462, 2018.
, “Rationale and Design of the Mechanistic Potential of Antihypertensives in Preclinical Alzheimer's (HEART) Trial.”, J Alzheimers Dis, vol. 61, no. 2, pp. 815-824, 2018.
, “Reduced Gray Matter Volume of the Thalamus and Hippocampal Region in Elderly Healthy Adults with no Impact of APOE ɛ4: A Longitudinal Voxel-Based Morphometry Study.”, J Alzheimers Dis, vol. 62, no. 2, pp. 757-771, 2018.
, “Role of ASK1/p38 Cascade in a Mouse Model of Alzheimer's Disease and Brain Aging.”, J Alzheimers Dis, vol. 61, no. 1, pp. 259-263, 2018.
, “Role of Genetics and Epigenetics in the Pathogenesis of Alzheimer's Disease and Frontotemporal Dementia.”, J Alzheimers Dis, vol. 62, no. 3, pp. 913-932, 2018.
, “Serum C-Peptide, Visfatin, Resistin, and Ghrelin are Altered in Sporadic and GRN-Associated Frontotemporal Lobar Degeneration.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1053-1060, 2018.
, “SORL1 Variants in Familial Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1275-1281, 2018.
, “SORL1 Variants in Familial Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1275-1281, 2018.
, “Structural Connectivity Alterations Along the Alzheimer's Disease Continuum: Reproducibility Across Two Independent Samples and Correlation with Cerebrospinal Fluid Amyloid-β and Tau.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1575-1587, 2018.
, “VEGFR1 and VEGFR2 in Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 2, pp. 741-752, 2018.
, “When Art Meets Gardens: Does It Enhance the Benefits? The Nancy Hypothesis of Care for Persons with Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 3, pp. 885-898, 2018.
, “Combined Dual-Task Gait Training and Aerobic Exercise to Improve Cognition, Mobility, and Vascular Health in Community-Dwelling Older Adults at Risk for Future Cognitive Decline1.”, J Alzheimers Dis, vol. 57, no. 3, pp. 747-763, 2017.
, “Exercise Training and Functional Connectivity Changes in Mild Cognitive Impairment and Healthy Elders.”, J Alzheimers Dis, vol. 57, no. 3, pp. 845-856, 2017.
, “Humanin Specifically Interacts with Amyloid-β Oligomers and Counteracts Their in vivo Toxicity.”, J Alzheimers Dis, vol. 57, no. 3, pp. 857-871, 2017.
, “Palmitate Increases β-site AβPP-Cleavage Enzyme 1 Activity and Amyloid-β Genesis by Evoking Endoplasmic Reticulum Stress and Subsequent C/EBP Homologous Protein Activation.”, J Alzheimers Dis, vol. 57, no. 3, pp. 907-925, 2017.
, “Anemia and Mild Cognitive Impairment in the German General Population.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1031-42, 2016.
, “Anthocyanins Protect SK-N-SH Cells Against Acrolein-Induced Toxicity by Preserving the Cellular Redox State.”, J Alzheimers Dis, vol. 50, no. 4, pp. 981-98, 2016.
, “Apolipoprotein ɛ4 is Associated with Dementia and Cognitive Impairment Predominantly Due to Alzheimer's Disease and Not with Vascular Cognitive Impairment: A Singapore-Based Cohort.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1111-8, 2016.
, “Apolipoprotein ɛ4 is Associated with Dementia and Cognitive Impairment Predominantly Due to Alzheimer's Disease and Not with Vascular Cognitive Impairment: A Singapore-Based Cohort.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1111-8, 2016.
, “Association Between Serum Ceruloplasmin Specific Activity and Risk of Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1181-9, 2016.
, “Association Between Serum Ceruloplasmin Specific Activity and Risk of Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1181-9, 2016.
, “Association of Butyrylcholinesterase-K Allele and Apolipoprotein E ɛ4 Allele with Cognitive Decline in Dementia with Lewy Bodies and Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 2, pp. 567-76, 2016.
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