Biblio
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Author Title [ Type] Year Filters: First Letter Of Last Name is A [Clear All Filters]
“Amyloid-β Increases Activity of Proteasomes Capped with 19S and 11S Regulators.”, J Alzheimers Dis, vol. 54, no. 2, pp. 763-76, 2016.
, “Amyloid-β Reduces Exosome Release from Astrocytes by Enhancing JNK Phosphorylation.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1433-41, 2016.
, “Analysis of Cerebrospinal Fluid and [11C]PIB PET Biomarkers for Alzheimer's Disease with Updated Protocols.”, J Alzheimers Dis, vol. 52, no. 4, pp. 1403-13, 2016.
, “Analysis of the Inhibitory Elements in the p5 Peptide Fragment of the CDK5 Activator, p35, CDKR1 Protein.”, J Alzheimers Dis, 2015.
, “Analysis of the MIRIAD Data Shows Sex Differences in Hippocampal Atrophy Progression.”, J Alzheimers Dis, vol. 50, no. 3, pp. 847-57, 2016.
, “Angiotensin II Blood Levels Are Associated with Smaller Hippocampal and Cortical Volumes in Cognitively Normal Older Adults.”, J Alzheimers Dis, vol. 75, no. 2, pp. 521-529, 2020.
, “ANMerge: A Comprehensive and Accessible Alzheimer's Disease Patient-Level Dataset.”, J Alzheimers Dis, vol. 79, no. 1, pp. 423-431, 2021.
, “The Anti-Amyloid Monoclonal Antibody Lecanemab: 16 Cautionary Notes.”, J Alzheimers Dis, vol. 94, no. 2, pp. 497-507, 2023.
, “Anti-Aβ Antibody Aducanumab Regulates the Proteome of Senile Plaques and Closely Surrounding Tissue in a Transgenic Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 79, no. 1, pp. 249-265, 2021.
, “Antibodies to Multiple Receptors are Associated with Neuropsychiatric Symptoms and Mortality in Alzheimer's Disease: A Longitudinal Study.”, J Alzheimers Dis, vol. 64, no. 3, pp. 761-774, 2018.
, “Antibodies to Signaling Molecules and Receptors in Alzheimer's Disease are Associated with Psychomotor Slowing, Depression, and Poor Visuospatial Function.”, J Alzheimers Dis, vol. 59, no. 3, pp. 929-939, 2017.
, “Anti-Correlated Cerebrospinal Fluid Biomarker Trajectories in Preclinical Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1085-97, 2016.
, “Antihypertensive Treatment is associated with MRI-Derived Markers of Neurodegeneration and Impaired Cognition: A Propensity-Weighted Cohort Study.”, J Alzheimers Dis, vol. 59, no. 3, pp. 1113-1122, 2017.
, “APOE Effect on Amyloid-β PET Spatial Distribution, Deposition Rate, and Cut-Points.”, J Alzheimers Dis, vol. 69, no. 3, pp. 783-793, 2019.
, “APOE4 Copy Number-Dependent Proteomic Changes in the Cerebrospinal Fluid.”, J Alzheimers Dis, vol. 79, no. 2, pp. 511-530, 2021.
, “APOEɛ4 Genotype, Amyloid, and Clinical Disease Progression in Cognitively Normal Older Adults.”, J Alzheimers Dis, vol. 57, no. 2, 2017.
, “APOEɛ4 Genotype, Amyloid, and Clinical Disease Progression in Cognitively Normal Older Adults.”, J Alzheimers Dis, vol. 57, no. 2, 2017.
, “APOEɛ4-TOMM40L Haplotype Increases the Risk of Mild Cognitive Impairment Conversion to Alzheimer's Disease.”, J Alzheimers Dis, vol. 78, no. 2, pp. 587-601, 2020.
, “Apolipoprotein ɛ4-Associated Protection Against Pediatric Enteric Infections is a Survival Advantage in Pre-Industrial Populations.”, J Alzheimers Dis, vol. 93, no. 3, pp. 907-918, 2023.
, “APP Mutations in Cerebral Amyloid Angiopathy with or without Cortical Calcifications: Report of Three Families and a Literature Review.”, J Alzheimers Dis, vol. 56, no. 1, pp. 37-46, 2017.
, “Application of the IWG-2 Diagnostic Criteria for Alzheimer's Disease to the ADNI.”, J Alzheimers Dis, vol. 51, no. 1, pp. 227-36, 2016.
, “Assessing Mild Cognitive Impairment Progression using a Spherical Brain Mapping of Magnetic Resonance Imaging.”, J Alzheimers Dis, vol. 65, no. 3, pp. 713-729, 2018.
, “Assessing the Progression of Alzheimer's Disease in Real-World Settings in Three European Countries.”, J Alzheimers Dis, vol. 80, no. 2, pp. 749-759, 2021.
, “Assessing Working Memory in Mild Cognitive Impairment with Serial Order Recall.”, J Alzheimers Dis, vol. 61, no. 3, pp. 917-928, 2018.
, “Assessment of Driving Safety in Older Adults with Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 57, no. 4, pp. 1197-1205, 2017.
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