Biblio
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“Multi-Site Cross-Site Inter-Rater and Test-Retest Reliability and Construct Validity of the MarkVCID White Matter Hyperintensity Growth and Regression Protocol.”, J Alzheimers Dis, vol. 96, no. 2, pp. 683-693, 2023.
, “The Multi-Target Drug M30 Shows Pro-Cognitive and Anti-Inflammatory Effects in a Rat Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 47, no. 2, pp. 373-83, 2015.
, “Music and Dementia: Individual Differences in Response to Personalized Playlists.”, J Alzheimers Dis, vol. 64, no. 3, pp. 933-941, 2018.
, “Music Playlists for People with Dementia: Trialing A Guide for Caregivers.”, J Alzheimers Dis, vol. 77, no. 1, pp. 219-226, 2020.
, “Mutation Screening of the CHCHD2 Gene for Alzheimer's Disease and Frontotemporal Dementia in Chinese Mainland Population.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1283-1288, 2018.
, “National Institute on Aging-Alzheimer's Association guidelines for the neuropathologic assessment of Alzheimer's disease.”, Alzheimers Dement, vol. 8, no. 1, pp. 1-13, 2012.
, “National Institute on Aging-Alzheimer's Association guidelines for the neuropathologic assessment of Alzheimer's disease.”, Alzheimers Dement, vol. 8, no. 1, pp. 1-13, 2012.
, “National Institutes of Health State-of-the-Science Conference statement: preventing alzheimer disease and cognitive decline.”, Ann Intern Med, vol. 153, no. 3, pp. 176-81, 2010.
, “National Institutes of Health State-of-the-Science Conference statement: preventing alzheimer disease and cognitive decline.”, Ann Intern Med, vol. 153, no. 3, pp. 176-81, 2010.
, “Netrin-1 Interrupts Amyloid-β Amplification, Increases sAβPPα in vitro and in vivo, and Improves Cognition in a Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 52, no. 1, pp. 223-42, 2016.
, “Network Hyperexcitability in Early-Stage Alzheimer's Disease: Evaluation of Functional Connectivity Biomarkers in a Computational Disease Model.”, J Alzheimers Dis, vol. 99, no. 4, pp. 1333-1348, 2024.
, “A Network of Genetic Effects on Non-Demented Cognitive Aging: Alzheimer's Genetic Risk (CLU + CR1 + PICALM) Intensifies Cognitive Aging Genetic Risk (COMT + BDNF) Selectively for APOEɛ4 Carriers.”, J Alzheimers Dis, vol. 62, no. 2, pp. 887-900, 2018.
, “Network-Based Substrate of Cognitive Reserve in Alzheimer's Disease.”, J Alzheimers Dis, vol. 55, no. 1, pp. 421-430, 2017.
, “Neural Correlates of Self-Reference Effect in Early Alzheimer's Disease.”, J Alzheimers Dis, vol. 56, no. 2, pp. 717-731, 2017.
, “Neural Correlates of Self-Reference Effect in Early Alzheimer's Disease.”, J Alzheimers Dis, vol. 56, no. 2, pp. 717-731, 2017.
, “Neuroanatomical Comparison of the "Word" and "Picture" Versions of the Free and Cued Selective Reminding Test in Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 2, pp. 589-600, 2018.
, “Neurocognitive and Behavioral Indexes for Identifying the Amnestic Subtypes of Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 60, no. 2, pp. 633-649, 2017.
, “Neuroimaging Correlates of Frontotemporal Dementia Associated with SQSTM1 Mutations.”, J Alzheimers Dis, vol. 53, no. 1, pp. 303-13, 2016.
, “Neuroimaging in Vascular Cognitive Impairment and Dementia: A Systematic Review.”, J Alzheimers Dis, vol. 73, no. 4, pp. 1279-1294, 2020.
, “Neuro-Immuno-Gene- and Genome-Editing-Therapy for Alzheimer's Disease: Are We There Yet?”, J Alzheimers Dis, vol. 65, no. 2, pp. 321-344, 2018.
, “Neuroinflammation, hyperphosphorylated tau, diffuse amyloid plaques, and down-regulation of the cellular prion protein in air pollution exposed children and young adults.”, J Alzheimers Dis, vol. 28, no. 1, pp. 93-107, 2012.
, “Neuroinflammation, hyperphosphorylated tau, diffuse amyloid plaques, and down-regulation of the cellular prion protein in air pollution exposed children and young adults.”, J Alzheimers Dis, vol. 28, no. 1, pp. 93-107, 2012.
, “Neuroinflammation, hyperphosphorylated tau, diffuse amyloid plaques, and down-regulation of the cellular prion protein in air pollution exposed children and young adults.”, J Alzheimers Dis, vol. 28, no. 1, pp. 93-107, 2012.
, “Neuronal Exosome-Derived Human Tau is Toxic to Recipient Mouse Neurons in vivo.”, J Alzheimers Dis, vol. 67, no. 2, pp. 541-553, 2019.
, “Neurons Derived from Induced Pluripotent Stem Cells of Patients with Down Syndrome Reproduce Early Stages of Alzheimer's Disease Type Pathology in vitro.”, J Alzheimers Dis, vol. 56, no. 2, pp. 835-847, 2017.
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