Biblio
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“A Pathway Based Classification Method for Analyzing Gene Expression for Alzheimer's Disease Diagnosis.”, J Alzheimers Dis, vol. 49, no. 3, pp. 659-69, 2016.
, “Patients that have Undergone Hemodialysis Exhibit Lower Amyloid Deposition in the Brain: Evidence Supporting a Therapeutic Strategy for Alzheimer's Disease by Removal of Blood Amyloid.”, J Alzheimers Dis, vol. 51, no. 4, pp. 997-1002, 2016.
, “Patterns of Brain Iron Accumulation in Vascular Dementia and Alzheimer's Dementia Using Quantitative Susceptibility Mapping Imaging.”, J Alzheimers Dis, vol. 51, no. 3, pp. 737-45, 2016.
, “PDE5 Exists in Human Neurons and is a Viable Therapeutic Target for Neurologic Disease.”, J Alzheimers Dis, vol. 52, no. 1, pp. 295-302, 2016.
, “Performance of Hippocampus Volumetry with FSL-FIRST for Prediction of Alzheimer's Disease Dementia in at Risk Subjects with Amnestic Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 51, no. 3, pp. 867-73, 2016.
, “Performance on Specific Cognitive Domains and Cause of Death: A Prospective Population-Based Study in Non-Demented Older Adults (NEDICES).”, J Alzheimers Dis, vol. 51, no. 2, pp. 533-44, 2016.
, “Performance on Specific Cognitive Domains and Cause of Death: A Prospective Population-Based Study in Non-Demented Older Adults (NEDICES).”, J Alzheimers Dis, vol. 51, no. 2, pp. 533-44, 2016.
, “Performance on Specific Cognitive Domains and Cause of Death: A Prospective Population-Based Study in Non-Demented Older Adults (NEDICES).”, J Alzheimers Dis, vol. 51, no. 2, pp. 533-44, 2016.
, “Platelet Membrane β-Secretase Activity in Mild Cognitive Impairment and Conversion to Dementia: a Longitudinal Study.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1095-103, 2016.
, “Platelet Membrane β-Secretase Activity in Mild Cognitive Impairment and Conversion to Dementia: a Longitudinal Study.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1095-103, 2016.
, “Platelet Membrane β-Secretase Activity in Mild Cognitive Impairment and Conversion to Dementia: a Longitudinal Study.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1095-103, 2016.
, “Post Hoc Analyses of ApoE Genotype-Defined Subgroups in Clinical Trials.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1205-15, 2016.
, “Post Hoc Analyses of ApoE Genotype-Defined Subgroups in Clinical Trials.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1205-15, 2016.
, “Prediction of Progressive Mild Cognitive Impairment by Multi-Modal Neuroimaging Biomarkers.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1045-56, 2016.
, “Predictive Value of Cerebrospinal Fluid Visinin-Like Protein-1 Levels for Alzheimer's Disease Early Detection and Differential Diagnosis in Patients with Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 50, no. 3, pp. 765-78, 2016.
, “Predictors of Response to Cholinesterase Inhibitors Treatment of Alzheimer's Disease: Date Mining from the TREDEM Registry.”, J Alzheimers Dis, vol. 50, no. 4, pp. 969-79, 2016.
, “Predictors of Response to Cholinesterase Inhibitors Treatment of Alzheimer's Disease: Date Mining from the TREDEM Registry.”, J Alzheimers Dis, vol. 50, no. 4, pp. 969-79, 2016.
, “Preliminary Study of Plasma Exosomal Tau as a Potential Biomarker for Chronic Traumatic Encephalopathy.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1099-109, 2016.
, “Preliminary Study of Plasma Exosomal Tau as a Potential Biomarker for Chronic Traumatic Encephalopathy.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1099-109, 2016.
, “The Presence of Select Tau Species in Human Peripheral Tissues and Their Relation to Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 2, pp. 345-56, 2016.
, “Prevalence and Risk Factor of Cognitive Impairment were Different between Urban and Rural Population: A Community-Based Study.”, J Alzheimers Dis, vol. 49, no. 4, pp. 917-25, 2016.
, “Prevalence and Risk Factor of Cognitive Impairment were Different between Urban and Rural Population: A Community-Based Study.”, J Alzheimers Dis, vol. 49, no. 4, pp. 917-25, 2016.
, “Prevention Trials in Alzheimer's Disease: Current Status and Future Perspectives.”, J Alzheimers Dis, vol. 50, no. 4, pp. 927-45, 2016.
, “Prion Protein-Hemin Interaction Upregulates Hemoglobin Synthesis: Implications for Cerebral Hemorrhage and Sporadic Creutzfeldt-Jakob Disease.”, J Alzheimers Dis, vol. 51, no. 1, pp. 107-21, 2016.
, “Prion Protein-Hemin Interaction Upregulates Hemoglobin Synthesis: Implications for Cerebral Hemorrhage and Sporadic Creutzfeldt-Jakob Disease.”, J Alzheimers Dis, vol. 51, no. 1, pp. 107-21, 2016.
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