Biblio

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2017
M. Romeo, Stravalaci, M., Beeg, M., Rossi, A., Fiordaliso, F., Corbelli, A., Salmona, M., Gobbi, M., Cagnotto, A., and Diomede, L., “Humanin Specifically Interacts with Amyloid-β Oligomers and Counteracts Their in vivo Toxicity.”, J Alzheimers Dis, vol. 57, no. 3, pp. 857-871, 2017.
Y. Huang, Shen, W., Su, J., Cheng, B., Li, D., Liu, G., Zhou, W. - X., and Zhang, Y. - X., “Modulating the Balance of Synaptic and Extrasynaptic NMDA Receptors Shows Positive Effects against Amyloid-β-Induced Neurotoxicity.”, J Alzheimers Dis, vol. 57, no. 3, pp. 885-897, 2017.
G. Marwarha, Rostad, S., Lilek, J., Kleinjan, M., Schommer, J., and Ghribi, O., “Palmitate Increases β-site AβPP-Cleavage Enzyme 1 Activity and Amyloid-β Genesis by Evoking Endoplasmic Reticulum Stress and Subsequent C/EBP Homologous Protein Activation.”, J Alzheimers Dis, vol. 57, no. 3, pp. 907-925, 2017.
L. - S. Liu, Bai, X. - Q., Gao, Y., Wu, Q., Ren, Z., Li, Q., Pan, L. - H., He, N. - Y., Peng, J., and Tang, Z. - H., “PCSK9 Promotes oxLDL-Induced PC12 Cell Apoptosis Through the Bcl-2/Bax-Caspase 9/3 Signaling Pathway.”, J Alzheimers Dis, vol. 57, no. 3, pp. 723-734, 2017.
N. Iwahara, Hisahara, S., Kawamata, J., Matsumura, A., Yokokawa, K., Saito, T., Fujikura, M., Manabe, T., Suzuki, H., Matsushita, T., Suzuki, S., and Shimohama, S., “Role of Suppressor of Cytokine Signaling 3 (SOCS3) in Altering Activated Microglia Phenotype in APPswe/PS1dE9 Mice.”, J Alzheimers Dis, vol. 55, no. 3, pp. 1235-1247, 2017.
2018
C. D. Pereira, Martins, F., Wiltfang, J., Silva, O. A. B. da Cru, and Rebelo, S., “ABC Transporters Are Key Players in Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 2, pp. 463-485, 2018.
J. L. King, Wong, A. A., and Brown, R. E., “Age-Related Changes in the Spatial Frequency Threshold of Male and Female 3xTg-AD Mice Using OptoMotry.”, J Alzheimers Dis, vol. 62, no. 2, pp. 591-596, 2018.
D. Zádori, Veres, G., Szalárdy, L., Klivenyi, P., and Vecsei, L., “Alzheimer's Disease: Recent Concepts on the Relation of Mitochondrial Disturbances, Excitotoxicity, Neuroinflammation, and Kynurenines.”, J Alzheimers Dis, vol. 62, no. 2, pp. 523-547, 2018.
J. A. Snir, Suchy, M., Bindseil, G. A., Kovacs, M., Chronik, B. A., Hudson, R. H. E., Pasternak, S. H., and Bartha, R., “An Aspartyl Cathepsin Targeted PET Agent: Application in an Alzheimer's Disease Mouse Model.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1241-1252, 2018.
J. Cummings, Ritter, A., and Zhong, K., “Clinical Trials for Disease-Modifying Therapies in Alzheimer's Disease: A Primer, Lessons Learned, and a Blueprint for the Future.”, J Alzheimers Dis, vol. 64, no. s1, pp. S3-S22, 2018.
C. M. Huber, Yee, C., May, T., Dhanala, A., and Mitchell, C. S., “Cognitive Decline in Preclinical Alzheimer's Disease: Amyloid-Beta versus Tauopathy.”, J Alzheimers Dis, vol. 61, no. 1, pp. 265-281, 2018.
S. Gelman, Palma, J., Tombaugh, G., and Ghavami, A., “Differences in Synaptic Dysfunction Between rTg4510 and APP/PS1 Mouse Models of Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 195-208, 2018.
Y. Fan, Luo, R., Su, L. - Y., Xiang, Q., Yu, D., Xu, L., Chen, J. - Q., Bi, R., Wu, D. - D., Zheng, P., and Yao, Y. - G., “Does the Genetic Feature of the Chinese Tree Shrew (Tupaia belangeri chinensis) Support Its Potential as a Viable Model for Alzheimer's Disease Research?”, J Alzheimers Dis, vol. 61, no. 3, pp. 1015-1028, 2018.
J. Lu, Shu, R., and Zhu, Y., “Dysregulation and Dislocation of SFPQ Disturbed DNA Organization in Alzheimer's Disease and Frontotemporal Dementia.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1311-1321, 2018.
J. Paulo Alme Santos, Vizuete, A., Hansen, F., Biasibetti, R., and Gonçalves, C. - A., “Early and Persistent O-GlcNAc Protein Modification in the Streptozotocin Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 237-249, 2018.
B. Gurel, Cansev, M., Sevinc, C., Kelestemur, S., Ocalan, B., Cakir, A., Aydin, S., Kahveci, N., Ozansoy, M., Taskapilioglu, O., Ulus, I. Hakki, Başar, M. Karayel, Sahin, B., Tuzuner, M. Bora, and Baykal, A. Tarik, “Early Stage Alterations in CA1 Extracellular Region Proteins Indicate Dysregulation of IL6 and Iron Homeostasis in the 5XFAD Alzheimer's Disease Mouse Model.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1399-1410, 2018.
B. Ahlemeyer, Halupczok, S., Rodenberg-Frank, E., Valerius, K. - P., and Baumgart-Vogt, E., “Endogenous Murine Amyloid-β Peptide Assembles into Aggregates in the Aged C57BL/6J Mouse Suggesting These Animals as a Model to Study Pathogenesis of Amyloid-β Plaque Formation.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1425-1450, 2018.
E. Chiziane, Telemann, H., Krueger, M., Adler, J., Arnhold, J., Alia, A., and Flemmig, J., “Free Heme and Amyloid-β: A Fatal Liaison in Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 3, pp. 963-984, 2018.
W. Zhang, Guo, Y., Li, B., Zhang, Q., Liu, J. - H., Gu, G. - J., Wang, J. - H., Bao, R. - K., Chen, Y. - J., and Xu, J. - R., “GDF11 Rejuvenates Cerebrovascular Structure and Function in an Animal Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 62, no. 2, pp. 807-819, 2018.
J. Mehla, Lacoursiere, S., Stuart, E., McDonald, R. J., and Mohajerani, M. H., “Gradual Cerebral Hypoperfusion Impairs Fear Conditioning and Object Recognition Learning and Memory in Mice: Potential Roles of Neurodegeneration and Cholinergic Dysfunction.”, J Alzheimers Dis, vol. 61, no. 1, pp. 283-293, 2018.
S. Kumfu, Charununtakorn, S. T., Jaiwongkam, T., Chattipakorn, N., and Chattipakorn, S. C., “Humanin Exerts Neuroprotection During Cardiac Ischemia-Reperfusion Injury.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1343-1353, 2018.
T. Saksida, Koprivica, I., Vujičić, M., Stošić-Grujičić, S., Perović, M., Kanazir, S., and Stojanović, I., “Impaired IL-17 Production in Gut-Residing Immune Cells of 5xFAD Mice with Alzheimer's Disease Pathology.”, J Alzheimers Dis, vol. 61, no. 2, pp. 619-630, 2018.
A. Ishikawa, Tokunaga, M., Maeda, J., Minamihisamatsu, T., Shimojo, M., Takuwa, H., Ono, M., Ni, R., Hirano, S., Kuwabara, S., Ji, B., Zhang, M. - R., Aoki, I., Suhara, T., Higuchi, M., and Sahara, N., “In Vivo Visualization of Tau Accumulation, Microglial Activation, and Brain Atrophy in a Mouse Model of Tauopathy rTg4510.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1037-1052, 2018.
B. M. Law, Guest, A. L., Pullen, M. W. J., Perkinton, M. S., and Williams, R. J., “Increased Foxo3a Nuclear Translocation and Activity is an Early Neuronal Response to βγ-Secretase-Mediated Processing of the Amyloid-β Protein Precursor: Utility of an AβPP-GAL4 Reporter Assay.”, J Alzheimers Dis, vol. 61, no. 2, pp. 673-688, 2018.
A. Arner, Rockenstein, E., Mante, M., Florio, J., Masliah, D., Salehi, B., Adame, A., Overk, C., Masliah, E., and Rissman, R. A., “Increased Vulnerability of the Hippocampus in Transgenic Mice Overexpressing APP and Triple Repeat Tau.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1201-1219, 2018.

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