Biblio
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“Alzheimer's Disease with Epileptiform EEG Activity: Abnormal Cortical Sources of Resting State Delta Rhythms in Patients with Amnesic Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 88, no. 3, pp. 903-931, 2022.
, “Alzheimer's Disease with Epileptiform EEG Activity: Abnormal Cortical Sources of Resting State Delta Rhythms in Patients with Amnesic Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 88, no. 3, pp. 903-931, 2022.
, “Alzheimer's Disease-Associated Cerebrospinal Fluid (CSF) Biomarkers do not Correlate with CSF Volumes or CSF Production Rate.”, J Alzheimers Dis, vol. 58, no. 3, pp. 821-828, 2017.
, “Alzheimer's Disease-Like Neurodegeneration in Porphyromonas gingivalis Infected Neurons with Persistent Expression of Active Gingipains.”, J Alzheimers Dis, vol. 75, no. 4, pp. 1361-1376, 2020.
, “Alzheimer's Disease-Related Neuropathology Among Patients with Medication Treated Type 2 Diabetes in a Community-Based Autopsy Cohort.”, J Alzheimers Dis, vol. 83, no. 3, pp. 1303-1312, 2021.
, “Amylin Treatment Reduces Neuroinflammation and Ameliorates Abnormal Patterns of Gene Expression in the Cerebral Cortex of an Alzheimer's Disease Mouse Model.”, J Alzheimers Dis, vol. 56, no. 1, pp. 47-61, 2017.
, “Amyloid and FDG PET of Successful Cognitive Aging: Global and Cingulate-Specific Differences.”, J Alzheimers Dis, vol. 66, no. 1, pp. 307-318, 2018.
, “Amyloid Burden in Obstructive Sleep Apnea.”, J Alzheimers Dis, vol. 59, no. 1, pp. 21-29, 2017.
, “Amyloid Hypothesis: The Emperor's New Clothes?”, J Alzheimers Dis, vol. 78, no. 4, pp. 1363-1366, 2020.
, “The Amyloid, Tau, and Neurodegeneration (A/T/N) Classification Applied to a Clinical Research Cohort with Long-Term Follow-Up.”, J Alzheimers Dis, vol. 74, no. 3, pp. 829-837, 2020.
, “The Amyloid, Tau, and Neurodegeneration (A/T/N) Classification Applied to a Clinical Research Cohort with Long-Term Follow-Up.”, J Alzheimers Dis, vol. 74, no. 3, pp. 829-837, 2020.
, “Amyloidogenic Nanoplaques in Blood Serum of Patients with Alzheimer's Disease Revealed by Time-Resolved Thioflavin T Fluorescence Intensity Fluctuation Analysis.”, J Alzheimers Dis, vol. 68, no. 2, pp. 571-582, 2019.
, “Amyloid-β Causes Mitochondrial Dysfunction via a Ca2+-Driven Upregulation of Oxidative Phosphorylation and Superoxide Production in Cerebrovascular Endothelial Cells.”, J Alzheimers Dis, vol. 75, no. 1, pp. 119-138, 2020.
, “Amyloid-β in Alzheimer's Disease: A Study of Citation Practices of the Amyloid Cascade Hypothesis Between 1992 and 2019.”, J Alzheimers Dis, vol. 74, no. 4, pp. 1309-1317, 2020.
, “Amyloid-β Increases Activity of Proteasomes Capped with 19S and 11S Regulators.”, J Alzheimers Dis, vol. 54, no. 2, pp. 763-76, 2016.
, “The Amyloid-β Oligomer Hypothesis: Beginning of the Third Decade.”, J Alzheimers Dis, vol. 64, no. s1, pp. S567-S610, 2018.
, “Amyloid-β/Drug Interactions from Computer Simulations and Cell-Based Assays.”, J Alzheimers Dis, vol. 64, no. s1, pp. S659-S672, 2018.
, “Analysis of Cerebrospinal Fluid and [11C]PIB PET Biomarkers for Alzheimer's Disease with Updated Protocols.”, J Alzheimers Dis, vol. 52, no. 4, pp. 1403-13, 2016.
, “Analysis of the Inhibitory Elements in the p5 Peptide Fragment of the CDK5 Activator, p35, CDKR1 Protein.”, J Alzheimers Dis, 2015.
, “Analysis of the Inhibitory Elements in the p5 Peptide Fragment of the CDK5 Activator, p35, CDKR1 Protein.”, J Alzheimers Dis, 2015.
, “Analysis of the MIRIAD Data Shows Sex Differences in Hippocampal Atrophy Progression.”, J Alzheimers Dis, vol. 50, no. 3, pp. 847-57, 2016.
, “Anemia and Mild Cognitive Impairment in the German General Population.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1031-42, 2016.
, “Ankle-Brachial Index but Neither Intima Media Thickness Nor Coronary Artery Calcification is Associated With Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 47, no. 2, pp. 433-42, 2015.
, “ANMerge: A Comprehensive and Accessible Alzheimer's Disease Patient-Level Dataset.”, J Alzheimers Dis, vol. 79, no. 1, pp. 423-431, 2021.
, “Antemortem-Postmortem Correlation of Florbetapir (18F) PET Amyloid Imaging with Quantitative Biochemical Measures of Aβ42 but not Aβ40.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1509-1516, 2018.
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