Biblio
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“Antibodies to Multiple Receptors are Associated with Neuropsychiatric Symptoms and Mortality in Alzheimer's Disease: A Longitudinal Study.”, J Alzheimers Dis, vol. 64, no. 3, pp. 761-774, 2018.
, “Antibodies to Signaling Molecules and Receptors in Alzheimer's Disease are Associated with Psychomotor Slowing, Depression, and Poor Visuospatial Function.”, J Alzheimers Dis, vol. 59, no. 3, pp. 929-939, 2017.
, “Anticholinergic Drug Burden and Neurocognitive Performance in the Study of Latinos-Investigation of Neurocognitive Aging.”, J Alzheimers Dis, vol. 86, no. 1, pp. 53-65, 2022.
, “Anticholinergic Drug Burden and Neurocognitive Performance in the Study of Latinos-Investigation of Neurocognitive Aging.”, J Alzheimers Dis, vol. 86, no. 1, pp. 53-65, 2022.
, “Anticholinergic Exposure and Risk of Pneumonia in Persons with Alzheimer's Disease: A Nested Case-Control Study.”, J Alzheimers Dis, vol. 56, no. 1, pp. 119-128, 2017.
, “Antiepileptic Drug Use Is Associated with an Increased Risk of Pneumonia Among Community-Dwelling Persons with Alzheimer's Disease-Matched Cohort Study.”, J Alzheimers Dis, vol. 68, no. 1, pp. 127-136, 2019.
, “Antihypertensive Medication Class and the Risk of Dementia and Cognitive Decline in Older Adults: A Secondary Analysis of the Prospective HELIAD Cohort.”, J Alzheimers Dis, vol. 89, no. 2, pp. 709-719, 2022.
, “Antihypertensive Treatment is associated with MRI-Derived Markers of Neurodegeneration and Impaired Cognition: A Propensity-Weighted Cohort Study.”, J Alzheimers Dis, vol. 59, no. 3, pp. 1113-1122, 2017.
, “Anti-Tau Monoclonal Antibodies Derived from Soluble and Filamentous Tau Show Diverse Functional Properties in vitro and in vivo.”, J Alzheimers Dis, vol. 65, no. 1, pp. 265-281, 2018.
, “Aortic Stiffness is Associated with Increased Risk of Incident Dementia in Older Adults.”, J Alzheimers Dis, vol. 66, no. 1, pp. 297-306, 2018.
, “Apathy and Attentional Biases in Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 3, pp. 837-46, 2016.
, “The APOE Gene is Differentially Methylated in Alzheimer's Disease.”, J Alzheimers Dis, 2015.
, “APOE ε4 Modulation of Training Outcomes in Several Cognitive Domains in a Sample of Cognitively Intact Older Adults.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1201-1215, 2017.
, “APOE ε4 Modulation of Training Outcomes in Several Cognitive Domains in a Sample of Cognitively Intact Older Adults.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1201-1215, 2017.
, “APOE4 Copy Number-Dependent Proteomic Changes in the Cerebrospinal Fluid.”, J Alzheimers Dis, vol. 79, no. 2, pp. 511-530, 2021.
, “APOE4 Status is Related to Differences in Memory-Related Brain Function in Asymptomatic Older Adults with Family History of Alzheimer's Disease: Baseline Analysis of the PREVENT-AD Task Functional MRI Dataset.”, J Alzheimers Dis, vol. 76, no. 1, pp. 97-119, 2020.
, “APOEɛ4 Allele Moderates the Association Between Basal Forebrain Nuclei Volumes and Allocentric Navigation in Older Adults Without Dementia.”, J Alzheimers Dis, vol. 86, no. 1, pp. 155-171, 2022.
, “APOEɛ4 Allele Moderates the Association Between Basal Forebrain Nuclei Volumes and Allocentric Navigation in Older Adults Without Dementia.”, J Alzheimers Dis, vol. 86, no. 1, pp. 155-171, 2022.
, “APOEɛ4 Genotype, Amyloid, and Clinical Disease Progression in Cognitively Normal Older Adults.”, J Alzheimers Dis, vol. 57, no. 2, 2017.
, “APOEɛ4 Genotype, Amyloid, and Clinical Disease Progression in Cognitively Normal Older Adults.”, J Alzheimers Dis, vol. 57, no. 2, 2017.
, “APOEɛ4-TOMM40L Haplotype Increases the Risk of Mild Cognitive Impairment Conversion to Alzheimer's Disease.”, J Alzheimers Dis, vol. 78, no. 2, pp. 587-601, 2020.
, “Apolipoprotein E Isoforms Differentially Regulate Alzheimer's Disease and Amyloid-β-Induced Inflammatory Response in vivo and in vitro.”, J Alzheimers Dis, vol. 57, no. 4, pp. 1265-1279, 2017.
, “Apolipoprotein E Isoforms Differentially Regulate Alzheimer's Disease and Amyloid-β-Induced Inflammatory Response in vivo and in vitro.”, J Alzheimers Dis, vol. 57, no. 4, pp. 1265-1279, 2017.
, “Apolipoprotein E Isoforms Differentially Regulate Alzheimer's Disease and Amyloid-β-Induced Inflammatory Response in vivo and in vitro.”, J Alzheimers Dis, vol. 57, no. 4, pp. 1265-1279, 2017.
, “Apolipoprotein E4 Elicits Lysosomal Cathepsin D Release, Decreased Thioredoxin-1 Levels, and Apoptosis.”, J Alzheimers Dis, vol. 56, no. 2, pp. 601-617, 2017.
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