Biblio

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I
A. Ishikawa, Tokunaga, M., Maeda, J., Minamihisamatsu, T., Shimojo, M., Takuwa, H., Ono, M., Ni, R., Hirano, S., Kuwabara, S., Ji, B., Zhang, M. - R., Aoki, I., Suhara, T., Higuchi, M., and Sahara, N., “In Vivo Visualization of Tau Accumulation, Microglial Activation, and Brain Atrophy in a Mouse Model of Tauopathy rTg4510.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1037-1052, 2018.
A. Ishikawa, Tokunaga, M., Maeda, J., Minamihisamatsu, T., Shimojo, M., Takuwa, H., Ono, M., Ni, R., Hirano, S., Kuwabara, S., Ji, B., Zhang, M. - R., Aoki, I., Suhara, T., Higuchi, M., and Sahara, N., “In Vivo Visualization of Tau Accumulation, Microglial Activation, and Brain Atrophy in a Mouse Model of Tauopathy rTg4510.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1037-1052, 2018.
G. L. Iverson, C Keene, D., Perry, G., and Castellani, R. J., “The Need to Separate Chronic Traumatic Encephalopathy Neuropathology from Clinical Features.”, J Alzheimers Dis, vol. 61, no. 1, pp. 17-28, 2018.
N. Iwahara, Hisahara, S., Kawamata, J., Matsumura, A., Yokokawa, K., Saito, T., Fujikura, M., Manabe, T., Suzuki, H., Matsushita, T., Suzuki, S., and Shimohama, S., “Role of Suppressor of Cytokine Signaling 3 (SOCS3) in Altering Activated Microglia Phenotype in APPswe/PS1dE9 Mice.”, J Alzheimers Dis, vol. 55, no. 3, pp. 1235-1247, 2017.
J
C. R. Jack, Knopman, D. S., Jagust, W. J., Petersen, R. C., Weiner, M. W., Aisen, P. S., Shaw, L. M., Vemuri, P., Wiste, H. J., Weigand, S. D., Lesnick, T. G., Pankratz, V. S., Donohue, M. C., and Trojanowski, J. Q., “Tracking pathophysiological processes in Alzheimer's disease: an updated hypothetical model of dynamic biomarkers.”, Lancet Neurol, vol. 12, no. 2, pp. 207-16, 2013.
C. R. Jack, Vemuri, P., Wiste, H. J., Weigand, S. D., Aisen, P. S., Trojanowski, J. Q., Shaw, L. M., Bernstein, M. A., Petersen, R. C., Weiner, M. W., and Knopman, D. S., “Evidence for ordering of Alzheimer disease biomarkers.”, Arch Neurol, vol. 68, no. 12, pp. 1526-35, 2011.
C. R. Jack, Knopman, D. S., Weigand, S. D., Wiste, H. J., Vemuri, P., Lowe, V., Kantarci, K., Gunter, J. L., Senjem, M. L., Ivnik, R. J., Roberts, R. O., Rocca, W. A., Boeve, B. F., and Petersen, R. C., “An operational approach to National Institute on Aging-Alzheimer's Association criteria for preclinical Alzheimer disease.”, Ann Neurol, vol. 71, no. 6, pp. 765-75, 2012.
J. Alexander Jarholm, Bjørnerud, A., Dalaker, T. Olene, Akhavi, M. Sadat, Kirsebom, B. Eivind, Pålhaugen, L., Nordengen, K., Grøntvedt, G. Rolfseng, Nakling, A., Kalheim, L. F., Almdahl, I. S., Tecelao, S., Fladby, T., and Selnes, P., “Medial Temporal Lobe Atrophy in Predementia Alzheimer's Disease: A Longitudinal Multi-Site Study Comparing Staging and A/T/N in a Clinical Research Cohort.”, J Alzheimers Dis, vol. 94, no. 1, pp. 259-279, 2023.
M. Jin, Shepardson, N., Yang, T., Chen, G., Walsh, D., and Selkoe, D. J., “Soluble amyloid beta-protein dimers isolated from Alzheimer cortex directly induce Tau hyperphosphorylation and neuritic degeneration.”, Proc Natl Acad Sci U S A, vol. 108, no. 14, pp. 5819-24, 2011.
M. Jucker and Walker, L. C., “Pathogenic protein seeding in Alzheimer disease and other neurodegenerative disorders.”, Ann Neurol, vol. 70, no. 4, pp. 532-40, 2011.
P. Jul, Volbracht, C., de Jong, I. E. M., Helboe, L., Elvang, A. Brandt, and Pedersen, J. Torleif, “Hyperactivity with Agitative-Like Behavior in a Mouse Tauopathy Model.”, J Alzheimers Dis, vol. 49, no. 3, pp. 783-95, 2016.
P. Jul, Volbracht, C., de Jong, I. E. M., Helboe, L., Elvang, A. Brandt, and Pedersen, J. Torleif, “Hyperactivity with Agitative-Like Behavior in a Mouse Tauopathy Model.”, J Alzheimers Dis, vol. 49, no. 3, pp. 783-95, 2016.
P. Jul, Volbracht, C., de Jong, I. E. M., Helboe, L., Elvang, A. Brandt, and Pedersen, J. Torleif, “Hyperactivity with Agitative-Like Behavior in a Mouse Tauopathy Model.”, J Alzheimers Dis, vol. 49, no. 3, pp. 783-95, 2016.
K
T. K. Karikari, “Blood Tests for Alzheimer's Disease: Increasing Efforts to Expand and Diversify Research Participation Is Critical for Widespread Validation and Acceptance.”, J Alzheimers Dis, vol. 90, no. 3, pp. 967-974, 2022.
H. Jin Kim, Cha, J., Lee, J. - M., Shin, J. Soo, Jung, N. - Y., Kim, Y. Jin, Choe, Y. Seong, Lee, K. Han, Kim, S. Tae, Kim, J. Seung, Lee, J. Hong, Na, D. L., and Seo, S. Won, “Distinctive Resting State Network Disruptions Among Alzheimer's Disease, Subcortical Vascular Dementia, and Mixed Dementia Patients.”, J Alzheimers Dis, vol. 50, no. 3, pp. 709-18, 2016.
Y. Kim, Kim, C., Jang, H. Young, and Mook-Jung, I., “Inhibition of Cholesterol Biosynthesis Reduces γ-Secretase Activity and Amyloid-β Generation.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1057-68, 2016.
H. Jin Kim, Cho, H., Werring, D. J., Jang, Y. Kyoung, Kim, Y. Jin, San Lee, J., Lee, J., Jun, S., Park, S., Ryu, Y. Hoon, Choi, J. Yong, Cho, Y. Seok, Moon, S. Hwan, Na, D. L., Lyoo, C. Hyoung, and Seo, S. Won, “18F-AV-1451 PET Imaging in Three Patients with Probable Cerebral Amyloid Angiopathy.”, J Alzheimers Dis, vol. 57, no. 3, pp. 711-716, 2017.
Y. Kim, Kim, C., Jang, H. Young, and Mook-Jung, I., “Inhibition of Cholesterol Biosynthesis Reduces γ-Secretase Activity and Amyloid-β Generation.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1057-68, 2016.
A. Kimura, Yoshikura, N., Hayashi, Y., and Inuzuka, T., “Cerebrospinal Fluid C-C Motif Chemokine Ligand 2 Correlates with Brain Atrophy and Cognitive Impairment in Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 2, pp. 581-588, 2018.
A. Kimura, Yoshikura, N., Hayashi, Y., and Inuzuka, T., “Cerebrospinal Fluid C-C Motif Chemokine Ligand 2 Correlates with Brain Atrophy and Cognitive Impairment in Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 2, pp. 581-588, 2018.
J. L. King, Wong, A. A., and Brown, R. E., “Age-Related Changes in the Spatial Frequency Threshold of Male and Female 3xTg-AD Mice Using OptoMotry.”, J Alzheimers Dis, vol. 62, no. 2, pp. 591-596, 2018.
N. Y. Kirson, J Andrews, S., Desai, U., King, S. B., Schonfeld, S., Birnbaum, H. G., Ball, D. E., and Kahle-Wrobleski, K., “Patient Characteristics and Outcomes Associated with Receiving an Earlier Versus Later Diagnosis of Probable Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 295-307, 2018.
G. Koch, Di Lorenzo, F., Del Olmo, M. Fernandez, Bonní, S., Ponzo, V., Caltagirone, C., Bozzali, M., and Martorana, A., “Reversal of LTP-Like Cortical Plasticity in Alzheimer's Disease Patients with Tau-Related Faster Clinical Progression.”, J Alzheimers Dis, vol. 50, no. 2, pp. 605-16, 2016.
G. Koch, Di Lorenzo, F., Del Olmo, M. Fernandez, Bonní, S., Ponzo, V., Caltagirone, C., Bozzali, M., and Martorana, A., “Reversal of LTP-Like Cortical Plasticity in Alzheimer's Disease Patients with Tau-Related Faster Clinical Progression.”, J Alzheimers Dis, vol. 50, no. 2, pp. 605-16, 2016.
A. Koppara, Wolfsgruber, S., Kleineidam, L., Schmidtke, K., Frölich, L., Kurz, A., Schulz, S., Hampel, H., Heuser, I., Peters, O., Reischies, F. M., Jahn, H., Luckhaus, C., Hüll, M., Gertz, H. - J., Schröder, J., Pantel, J., Rienhoff, O., Rüther, E., Henn, F., Wiltfang, J., Maier, W., Jessen, F., Kornhuber, J., and Wagner, M., “The Latent Dementia Phenotype δ is Associated with Cerebrospinal Fluid Biomarkers of Alzheimer's Disease and Predicts Conversion to Dementia in Subjects with Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 49, no. 2, pp. 547-60, 2016.

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