Biblio
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Author [ Title] Type Year Filters: First Letter Of Last Name is B [Clear All Filters]
“Anthocyanins Protect SK-N-SH Cells Against Acrolein-Induced Toxicity by Preserving the Cellular Redox State.”, J Alzheimers Dis, vol. 50, no. 4, pp. 981-98, 2016.
, “The Anti-Amyloid Monoclonal Antibody Lecanemab: 16 Cautionary Notes.”, J Alzheimers Dis, vol. 94, no. 2, pp. 497-507, 2023.
, “Anti-amyloid β autoantibodies in cerebral amyloid angiopathy-related inflammation: implications for amyloid-modifying therapies.”, Ann Neurol, vol. 73, no. 4, pp. 449-58, 2013.
, “Anti-amyloid β autoantibodies in cerebral amyloid angiopathy-related inflammation: implications for amyloid-modifying therapies.”, Ann Neurol, vol. 73, no. 4, pp. 449-58, 2013.
, “Anti-Aβ Antibody Aducanumab Regulates the Proteome of Senile Plaques and Closely Surrounding Tissue in a Transgenic Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 79, no. 1, pp. 249-265, 2021.
, “Anti-Aβ Antibody Aducanumab Regulates the Proteome of Senile Plaques and Closely Surrounding Tissue in a Transgenic Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 79, no. 1, pp. 249-265, 2021.
, “Antihypertensive Treatment is associated with MRI-Derived Markers of Neurodegeneration and Impaired Cognition: A Propensity-Weighted Cohort Study.”, J Alzheimers Dis, vol. 59, no. 3, pp. 1113-1122, 2017.
, “Antihypertensive Treatment is associated with MRI-Derived Markers of Neurodegeneration and Impaired Cognition: A Propensity-Weighted Cohort Study.”, J Alzheimers Dis, vol. 59, no. 3, pp. 1113-1122, 2017.
, “The Anti-Prion Antibody 15B3 Detects Toxic Amyloid-β Oligomers.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1485-97, 2016.
, “The Anti-Prion Antibody 15B3 Detects Toxic Amyloid-β Oligomers.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1485-97, 2016.
, “The Anti-Prion Antibody 15B3 Detects Toxic Amyloid-β Oligomers.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1485-97, 2016.
, “Anti-Tau Monoclonal Antibodies Derived from Soluble and Filamentous Tau Show Diverse Functional Properties in vitro and in vivo.”, J Alzheimers Dis, vol. 65, no. 1, pp. 265-281, 2018.
, “Anti-Tau Monoclonal Antibodies Derived from Soluble and Filamentous Tau Show Diverse Functional Properties in vitro and in vivo.”, J Alzheimers Dis, vol. 65, no. 1, pp. 265-281, 2018.
, “Anti-Viral Properties of Amyloid-β Peptides.”, J Alzheimers Dis, vol. 54, no. 3, pp. 859-878, 2016.
, “Aortic Valve Calcification and the Risk of dementia: A Population-Based Study.”, J Alzheimers Dis, vol. 55, no. 3, pp. 893-897, 2017.
, “Aphasia in Progressive Supranuclear Palsy: As Severe as Progressive Non-Fluent Aphasia.”, J Alzheimers Dis, vol. 61, no. 2, pp. 705-715, 2018.
, “Aphasia in Progressive Supranuclear Palsy: As Severe as Progressive Non-Fluent Aphasia.”, J Alzheimers Dis, vol. 61, no. 2, pp. 705-715, 2018.
, “APOE Effect on Amyloid-β PET Spatial Distribution, Deposition Rate, and Cut-Points.”, J Alzheimers Dis, vol. 69, no. 3, pp. 783-793, 2019.
, “The APOE Gene is Differentially Methylated in Alzheimer's Disease.”, J Alzheimers Dis, 2015.
, “APOE ɛ4 Carriers Show Delayed Recovery of Verbal Memory and Smaller Entorhinal Volume in the First Year After Ischemic Stroke.”, J Alzheimers Dis, vol. 71, no. 1, pp. 245-259, 2019.
, “APOE ɛ4 Carriers Show Delayed Recovery of Verbal Memory and Smaller Entorhinal Volume in the First Year After Ischemic Stroke.”, J Alzheimers Dis, vol. 71, no. 1, pp. 245-259, 2019.
, “APOE ɛ4 Carriers Show Delayed Recovery of Verbal Memory and Smaller Entorhinal Volume in the First Year After Ischemic Stroke.”, J Alzheimers Dis, vol. 71, no. 1, pp. 245-259, 2019.
, “APOE ε4 Modulation of Training Outcomes in Several Cognitive Domains in a Sample of Cognitively Intact Older Adults.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1201-1215, 2017.
, “APOE4 Copy Number-Dependent Proteomic Changes in the Cerebrospinal Fluid.”, J Alzheimers Dis, vol. 79, no. 2, pp. 511-530, 2021.
, “APOE4 Copy Number-Dependent Proteomic Changes in the Cerebrospinal Fluid.”, J Alzheimers Dis, vol. 79, no. 2, pp. 511-530, 2021.
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