Biblio
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“Serotonin Degeneration and Amyloid-β Deposition in Mild Cognitive Impairment: Relationship to Cognitive Deficits.”, J Alzheimers Dis, vol. 96, no. 1, pp. 215-227, 2023.
, “Serotonin Degeneration and Amyloid-β Deposition in Mild Cognitive Impairment: Relationship to Cognitive Deficits.”, J Alzheimers Dis, vol. 96, no. 1, pp. 215-227, 2023.
, “Serotonin Selective Reuptake Inhibitor Treatment Improves Cognition and Grey Matter Atrophy but not Amyloid Burden During Two-Year Follow-Up in Mild Cognitive Impairment and Alzheimer's Disease Patients with Depressive Symptoms.”, J Alzheimers Dis, vol. 65, no. 3, pp. 793-806, 2018.
, “Serum Adiponectin Levels, Neuroimaging, and Cognition in the Mayo Clinic Study of Aging.”, J Alzheimers Dis, vol. 53, no. 2, pp. 573-81, 2016.
, “Serum Levels of α-Klotho Are Correlated with Cerebrospinal Fluid Levels and Predict Measures of Cognitive Function.”, J Alzheimers Dis, vol. 86, no. 3, pp. 1471-1481, 2022.
, “Serum Levels of α-Klotho Are Correlated with Cerebrospinal Fluid Levels and Predict Measures of Cognitive Function.”, J Alzheimers Dis, vol. 86, no. 3, pp. 1471-1481, 2022.
, “Severity of Depression Impacts Imminent Conversion from Mild Cognitive Impairment to Alzheimer's Disease.”, J Alzheimers Dis, vol. 59, no. 4, pp. 1439-1448, 2017.
, “Sex-Dependent Associations of Serum Uric Acid with Brain Function During Aging.”, J Alzheimers Dis, vol. 60, no. 2, pp. 699-706, 2017.
, “Sex-Dependent Associations of Serum Uric Acid with Brain Function During Aging.”, J Alzheimers Dis, vol. 60, no. 2, pp. 699-706, 2017.
, “Sex-Dependent Differences in Spontaneous Autoimmunity in Adult 3xTg-AD Mice.”, J Alzheimers Dis, vol. 63, no. 3, pp. 1191-1205, 2018.
, “Sex-Related Reserve Hypothesis in Alzheimer's Disease: Changes in Cortical Thickness with a Five-Year Longitudinal Follow-Up.”, J Alzheimers Dis, vol. 65, no. 2, pp. 641-649, 2018.
, “Sex-Related Reserve Hypothesis in Alzheimer's Disease: Changes in Cortical Thickness with a Five-Year Longitudinal Follow-Up.”, J Alzheimers Dis, vol. 65, no. 2, pp. 641-649, 2018.
, “Sex-Related Reserve Hypothesis in Alzheimer's Disease: Changes in Cortical Thickness with a Five-Year Longitudinal Follow-Up.”, J Alzheimers Dis, vol. 65, no. 2, pp. 641-649, 2018.
, “Sex-specific effects of apolipoprotein E ε4 genotype on longitudinal hippocampal atrophy in amnestic mild cognitive impairment over a 2-year evaluation period.”, J Alzheimers Dis, p. 13872877241313066, 2025.
, “Sex-specific effects of apolipoprotein E ε4 genotype on longitudinal hippocampal atrophy in amnestic mild cognitive impairment over a 2-year evaluation period.”, J Alzheimers Dis, p. 13872877241313066, 2025.
, “Sex-specific effects of apolipoprotein E ε4 genotype on longitudinal hippocampal atrophy in amnestic mild cognitive impairment over a 2-year evaluation period.”, J Alzheimers Dis, p. 13872877241313066, 2025.
, “Sex-Specific Patterns of Body Mass Index Relationship with White Matter Connectivity.”, J Alzheimers Dis, vol. 86, no. 4, pp. 1831-1848, 2022.
, “Shortening of Saccades as a Possible Easy-to-Use Biomarker to Detect Risk of Alzheimer's Disease.”, J Alzheimers Dis, vol. 88, no. 2, pp. 609-618, 2022.
, “Shortening of Saccades as a Possible Easy-to-Use Biomarker to Detect Risk of Alzheimer's Disease.”, J Alzheimers Dis, vol. 88, no. 2, pp. 609-618, 2022.
, “Shortening of Saccades as a Possible Easy-to-Use Biomarker to Detect Risk of Alzheimer's Disease.”, J Alzheimers Dis, vol. 88, no. 2, pp. 609-618, 2022.
, “Shortening of Saccades as a Possible Easy-to-Use Biomarker to Detect Risk of Alzheimer's Disease.”, J Alzheimers Dis, vol. 88, no. 2, pp. 609-618, 2022.
, “Sideritis spp. Extracts Enhance Memory and Learning in Alzheimer's β-Amyloidosis Mouse Models and Aged C57Bl/6 Mice.”, J Alzheimers Dis, vol. 53, no. 3, pp. 967-80, 2016.
, “Simultaneous Aerobic Exercise and Memory Training Program in Older Adults with Subjective Memory Impairments.”, J Alzheimers Dis, vol. 62, no. 2, pp. 795-806, 2018.
, “Simultaneous Aerobic Exercise and Memory Training Program in Older Adults with Subjective Memory Impairments.”, J Alzheimers Dis, vol. 62, no. 2, pp. 795-806, 2018.
, “Site-Specific Cerebrospinal Fluid Tau Hyperphosphorylation in Response to Alzheimer's Disease Brain Pathology: Not All Tau Phospho-Sites are Hyperphosphorylated.”, J Alzheimers Dis, vol. 85, no. 1, pp. 415-429, 2021.
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