Biblio
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“The Role of Single-Subject Brain Metabolic Patterns in the Early Differential Diagnosis of Primary Progressive Aphasias and in Prediction of Progression to Dementia.”, J Alzheimers Dis, vol. 55, no. 1, pp. 183-197, 2017.
, “Role of Suppressor of Cytokine Signaling 3 (SOCS3) in Altering Activated Microglia Phenotype in APPswe/PS1dE9 Mice.”, J Alzheimers Dis, vol. 55, no. 3, pp. 1235-1247, 2017.
, “Sample Size Estimation for Alzheimer's Disease Trials from Japanese ADNI Serial Magnetic Resonance Imaging.”, J Alzheimers Dis, vol. 56, no. 1, pp. 75-88, 2017.
, “Skin Autofluorescence Examination as a Diagnostic Tool for Mild Cognitive Impairment in Healthy People.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1481-1487, 2017.
, “Skin Autofluorescence Examination as a Diagnostic Tool for Mild Cognitive Impairment in Healthy People.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1481-1487, 2017.
, “Smoking, APOE Genotype, and Cognitive Decline: The Rotterdam Study.”, J Alzheimers Dis, vol. 57, no. 4, pp. 1191-1195, 2017.
, “Synaptic Compensation as a Probable Cause of Prolonged Mild Cognitive Impairment in Alzheimer's Disease: Implications from a Transgenic Mouse Model of the Disease.”, J Alzheimers Dis, vol. 56, no. 4, pp. 1385-1401, 2017.
, “Tocotrienol-Rich Fraction Modulates Amyloid Pathology and Improves Cognitive Function in AβPP/PS1 Mice.”, J Alzheimers Dis, vol. 55, no. 2, pp. 597-612, 2017.
, “TOMM40 and APOE Gene Expression and Cognitive Decline in Japanese Alzheimer's Disease Subjects.”, J Alzheimers Dis, vol. 60, no. 3, pp. 1107-1117, 2017.
, “Violation of Laws in Frontotemporal Dementia: A Multicenter Study in Japan.”, J Alzheimers Dis, vol. 57, no. 4, pp. 1221-1227, 2017.
, “Vitamin D and the Risk of Dementia: The Rotterdam Study.”, J Alzheimers Dis, vol. 60, no. 3, pp. 989-997, 2017.
, “Vitamin D and the Risk of Dementia: The Rotterdam Study.”, J Alzheimers Dis, vol. 60, no. 3, pp. 989-997, 2017.
, “Antemortem-Postmortem Correlation of Florbetapir (18F) PET Amyloid Imaging with Quantitative Biochemical Measures of Aβ42 but not Aβ40.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1509-1516, 2018.
, “Assessing Mild Behavioral Impairment with the Mild Behavioral Impairment-Checklist in People with Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 66, no. 1, pp. 83-95, 2018.
, “Assessment of the Genetic Architecture of Alzheimer's Disease Risk in Rate of Memory Decline.”, J Alzheimers Dis, vol. 62, no. 2, pp. 745-756, 2018.
, “Association between α-Klotho and Deep White Matter Lesions in the Brain: A Pilot Case Control Study Using Brain MRI.”, J Alzheimers Dis, vol. 61, no. 1, pp. 145-155, 2018.
, “Association between α-Klotho and Deep White Matter Lesions in the Brain: A Pilot Case Control Study Using Brain MRI.”, J Alzheimers Dis, vol. 61, no. 1, pp. 145-155, 2018.
, “Association between α-Klotho and Deep White Matter Lesions in the Brain: A Pilot Case Control Study Using Brain MRI.”, J Alzheimers Dis, vol. 61, no. 1, pp. 145-155, 2018.
, “Automated Multi-Atlas Segmentation of Hippocampal and Extrahippocampal Subregions in Alzheimer's Disease at 3T and 7T: What Atlas Composition Works Best?”, J Alzheimers Dis, vol. 63, no. 1, pp. 217-225, 2018.
, “Bone-Marrow-Derived Microglia-Like Cells Ameliorate Brain Amyloid Pathology and Cognitive Impairment in a Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 64, no. 2, pp. 563-585, 2018.
, “Calpain I Activation Causes GLUT3 Proteolysis and Downregulation of O-GlcNAcylation in Alzheimer's Disease Brain.”, J Alzheimers Dis, vol. 62, no. 4, pp. 1737-1746, 2018.
, “Cardiovascular Response to Mental Stress in Mild Cognitive Impairment and its Association with Cerebral Perfusion.”, J Alzheimers Dis, 2018.
, “Cerebrospinal Fluid C-C Motif Chemokine Ligand 2 Correlates with Brain Atrophy and Cognitive Impairment in Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 2, pp. 581-588, 2018.
, “Cerebrospinal Fluid Orexin Levels and Nocturnal Sleep Disruption in Alzheimer's Disease Patients Showing Neuropsychiatric Symptoms.”, J Alzheimers Dis, vol. 66, no. 3, pp. 993-999, 2018.
, “Change of Amyloid-β 1-42 Toxic Conformer Ratio After Cerebrospinal Fluid Diversion Predicts Long-Term Cognitive Outcome in Patients with Idiopathic Normal Pressure Hydrocephalus.”, J Alzheimers Dis, vol. 63, no. 3, pp. 989-1002, 2018.
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