Biblio
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“PRNP P39L Variant is a Rare Cause of Frontotemporal Dementia in Italian Population.”, J Alzheimers Dis, vol. 50, no. 2, pp. 353-7, 2016.
, “PRNP P39L Variant is a Rare Cause of Frontotemporal Dementia in Italian Population.”, J Alzheimers Dis, vol. 50, no. 2, pp. 353-7, 2016.
, “Prognostic Significance of Mild Cognitive Impairment Subtypes for Dementia and Mortality: Data from the NEDICES Cohort.”, J Alzheimers Dis, vol. 50, no. 3, pp. 719-31, 2016.
, “Progranulin Protein Levels in Cerebrospinal Fluid in Primary Neurodegenerative Dementias.”, J Alzheimers Dis, vol. 50, no. 2, pp. 539-46, 2016.
, “Progranulin Protein Levels in Cerebrospinal Fluid in Primary Neurodegenerative Dementias.”, J Alzheimers Dis, vol. 50, no. 2, pp. 539-46, 2016.
, “Progranulin Protein Levels in Cerebrospinal Fluid in Primary Neurodegenerative Dementias.”, J Alzheimers Dis, vol. 50, no. 2, pp. 539-46, 2016.
, “Prolonged Visual Facial Grasp in Frontotemporal Dementia.”, J Alzheimers Dis, vol. 53, no. 1, pp. 327-35, 2016.
, “Promising Role of Neuromodulation in Predicting the Progression of Mild Cognitive Impairment to Dementia.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1375-88, 2016.
, “Promising Role of Neuromodulation in Predicting the Progression of Mild Cognitive Impairment to Dementia.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1375-88, 2016.
, “Protein Kinase Activity Decreases with Higher Braak Stages of Alzheimer's Disease Pathology.”, J Alzheimers Dis, vol. 49, no. 4, pp. 927-43, 2016.
, “Puerarin Ameliorates D-Galactose Induced Enhanced Hippocampal Neurogenesis and Tau Hyperphosphorylation in Rat Brain.”, J Alzheimers Dis, vol. 51, no. 2, pp. 605-17, 2016.
, “Quantitative phosphoproteomic analyses of the inferior parietal lobule from three different pathological stages of Alzheimer's disease.”, J Alzheimers Dis, vol. 49, no. 1, pp. 45-62, 2016.
, “Reduction of Amyloid-β Plasma Levels by Hemodialysis: An Anti-Amyloid Treatment Strategy?”, J Alzheimers Dis, vol. 50, no. 3, pp. 791-6, 2016.
, “Reduction of Blood Amyloid-β Oligomers in Alzheimer's Disease Transgenic Mice by c-Abl Kinase Inhibition.”, J Alzheimers Dis, vol. 54, no. 3, pp. 1193-1205, 2016.
, “Reelin in Alzheimer's Disease, Increased Levels but Impaired Signaling: When More is Less.”, J Alzheimers Dis, vol. 52, no. 2, pp. 403-16, 2016.
, “Relative Alterations in Blood-Based Levels of Sestrin in Alzheimer's Disease and Mild Cognitive Impairment Patients.”, J Alzheimers Dis, vol. 54, no. 3, pp. 1147-1155, 2016.
, “Reproductive history and risk of cognitive impairment in elderly women: a cross-sectional study in eastern China.”, J Alzheimers Dis, vol. 49, no. 1, pp. 139-47, 2016.
, “Reversal of LTP-Like Cortical Plasticity in Alzheimer's Disease Patients with Tau-Related Faster Clinical Progression.”, J Alzheimers Dis, vol. 50, no. 2, pp. 605-16, 2016.
, “Reversible Mild Cognitive Impairment: The Role of Comorbidities at Baseline Evaluation.”, J Alzheimers Dis, vol. 51, no. 1, pp. 57-67, 2016.
, “Reversible Mild Cognitive Impairment: The Role of Comorbidities at Baseline Evaluation.”, J Alzheimers Dis, vol. 51, no. 1, pp. 57-67, 2016.
, “Reversible Mild Cognitive Impairment: The Role of Comorbidities at Baseline Evaluation.”, J Alzheimers Dis, vol. 51, no. 1, pp. 57-67, 2016.
, “Revisiting the Heidenhain Variant of Creutzfeldt-Jakob Disease: Evidence for Prion Type Variability Influencing Clinical Course and Laboratory Findings.”, J Alzheimers Dis, vol. 50, no. 2, pp. 465-76, 2016.
, “The Role of Cerebrovascular Disease in Amyloid Deposition.”, J Alzheimers Dis, vol. 54, no. 3, pp. 1015-1026, 2016.
, “The Role of Cerebrovascular Disease in Amyloid Deposition.”, J Alzheimers Dis, vol. 54, no. 3, pp. 1015-1026, 2016.
, “The Role of Ethnicity in Alzheimer's Disease: Findings From The C-PATH Online Data Repository.”, J Alzheimers Dis, vol. 51, no. 2, pp. 515-23, 2016.
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