Biblio
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“Delirium Severity Post-Surgery and its Relationship with Long-Term Cognitive Decline in a Cohort of Patients without Dementia.”, J Alzheimers Dis, vol. 61, no. 1, pp. 347-358, 2018.
, “Development of a High-Sensitivity Method for the Measurement of Human Nasal Aβ42, Tau, and Phosphorylated Tau.”, J Alzheimers Dis, vol. 62, no. 2, pp. 737-744, 2018.
, “The Diagnostic and Prognostic Value of a Dual-Tasking Paradigm in a Memory Clinic.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1189-1199, 2018.
, “Early and Persistent O-GlcNAc Protein Modification in the Streptozotocin Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 237-249, 2018.
, “Economic Burden, Mortality, and Institutionalization in Patients Newly Diagnosed with Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 185-193, 2018.
, “Endogenous Murine Amyloid-β Peptide Assembles into Aggregates in the Aged C57BL/6J Mouse Suggesting These Animals as a Model to Study Pathogenesis of Amyloid-β Plaque Formation.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1425-1450, 2018.
, “Endogenous Murine Amyloid-β Peptide Assembles into Aggregates in the Aged C57BL/6J Mouse Suggesting These Animals as a Model to Study Pathogenesis of Amyloid-β Plaque Formation.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1425-1450, 2018.
, “Executive and Language Subjective Cognitive Decline Complaints Discriminate Preclinical Alzheimer's Disease from Normal Aging.”, J Alzheimers Dis, vol. 61, no. 2, pp. 689-703, 2018.
, “High Caregiver Burden in Young Onset Dementia: What Factors Need Attention?”, J Alzheimers Dis, vol. 61, no. 2, pp. 537-543, 2018.
, “Humanin Exerts Neuroprotection During Cardiac Ischemia-Reperfusion Injury.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1343-1353, 2018.
, “Humanin Exerts Neuroprotection During Cardiac Ischemia-Reperfusion Injury.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1343-1353, 2018.
, “Humanin Exerts Neuroprotection During Cardiac Ischemia-Reperfusion Injury.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1343-1353, 2018.
, “Increased Foxo3a Nuclear Translocation and Activity is an Early Neuronal Response to βγ-Secretase-Mediated Processing of the Amyloid-β Protein Precursor: Utility of an AβPP-GAL4 Reporter Assay.”, J Alzheimers Dis, vol. 61, no. 2, pp. 673-688, 2018.
, “Increased Vulnerability of the Hippocampus in Transgenic Mice Overexpressing APP and Triple Repeat Tau.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1201-1219, 2018.
, “Interactions between Atrial Fibrillation, Cardiovascular Risk Factors, and ApoE Genotype in Promoting Cognitive Decline in Patients with Alzheimer's Disease: A Prospective Cohort Study.”, J Alzheimers Dis, vol. 62, no. 2, pp. 713-725, 2018.
, “Lentivirus-Mediated HDAC3 Inhibition Attenuates Oxidative Stress in APPswe/PS1dE9 Mice.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1411-1424, 2018.
, “A Link between Subjective Perceptions of Memory and Physical Function: Implications for Subjective Cognitive Decline.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1387-1398, 2018.
, “A Link between Subjective Perceptions of Memory and Physical Function: Implications for Subjective Cognitive Decline.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1387-1398, 2018.
, “Lipid Metabolism and Survival Across the Frontotemporal Dementia-Amyotrophic Lateral Sclerosis Spectrum: Relationships to Eating Behavior and Cognition.”, J Alzheimers Dis, vol. 61, no. 2, pp. 773-783, 2018.
, “Longitudinal Neuroimaging Analysis in Mild-Moderate Alzheimer's Disease Patients Treated with Plasma Exchange with 5% Human Albumin.”, J Alzheimers Dis, vol. 61, no. 1, pp. 321-332, 2018.
, “A Longitudinal Study of Total and Phosphorylated α-Synuclein with Other Biomarkers in Cerebrospinal Fluid of Alzheimer's Disease and Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1541-1553, 2018.
, “Low Prevalence of Cancer in Patients with Frontotemporal Lobar Degeneration.”, J Alzheimers Dis, vol. 62, no. 2, pp. 789-794, 2018.
, “Mental States in Moving Shapes: Distinct Cortical and Subcortical Contributions to Theory of Mind Impairments in Dementia.”, J Alzheimers Dis, vol. 61, no. 2, pp. 521-535, 2018.
, “Mesenchymal Stem Cell-Derived Extracellular Vesicles Suppresses iNOS Expression and Ameliorates Neural Impairment in Alzheimer's Disease Mice.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1005-1013, 2018.
, “Mesenchymal Stem Cell-Derived Extracellular Vesicles Suppresses iNOS Expression and Ameliorates Neural Impairment in Alzheimer's Disease Mice.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1005-1013, 2018.
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