Biblio
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“Stress-Induced Synaptic Dysfunction and Neurotransmitter Release in Alzheimer's Disease: Can Neurotransmitters and Neuromodulators be Potential Therapeutic Targets?”, J Alzheimers Dis, vol. 57, no. 4, pp. 1017-1039, 2017.
, “Stress-Induced Synaptic Dysfunction and Neurotransmitter Release in Alzheimer's Disease: Can Neurotransmitters and Neuromodulators be Potential Therapeutic Targets?”, J Alzheimers Dis, vol. 57, no. 4, pp. 1017-1039, 2017.
, “Stratification by Genetic and Demographic Characteristics Improves Diagnostic Accuracy of Cerebrospinal Fluid Biomarkers in Rapidly Progressive Dementia.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1385-1393, 2016.
, “Strategies for improving memory: a randomized trial of memory groups for older people, including those with mild cognitive impairment.”, J Alzheimers Dis, vol. 49, no. 1, pp. 31-43, 2016.
, “Stimulating the Activity of Amyloid-Beta Degrading Enzymes: A Novel Approach for the Therapeutic Manipulation of Amyloid-Beta Levels.”, J Alzheimers Dis, vol. 54, no. 3, pp. 891-895, 2016.
, “Stem Cell-Derived Neurons as Cellular Models of Sporadic Alzheimer's Disease.”, J Alzheimers Dis, vol. 67, no. 3, pp. 893-910, 2019.
, “Stem Cell Therapy for Alzheimer's Disease: A Review of Recent Clinical Trials.”, J Alzheimers Dis, vol. 54, no. 3, pp. 879-889, 2016.
, “Statins and Brain Health: Alzheimer's Disease and Cerebrovascular Disease Biomarkers in Older Adults.”, J Alzheimers Dis, vol. 65, no. 4, pp. 1345-1352, 2018.
, “Stages of Objective Memory Impairment Predict Alzheimer's Disease Neuropathology: Comparison with the Clinical Dementia Rating Scale-Sum of Boxes.”, J Alzheimers Dis, vol. 80, no. 1, pp. 185-195, 2021.
, “Stabilized Low-n Amyloid-β Oligomers Induce Robust Novel Object Recognition Deficits Associated with Inflammatory, Synaptic, and GABAergic Dysfunction in the Rat.”, J Alzheimers Dis, vol. 62, no. 1, pp. 213-226, 2018.
, “Spiritual Fitness: A New Dimension in Alzheimer's Disease Prevention.”, J Alzheimers Dis, vol. 80, no. 2, pp. 505-519, 2021.
, “Specific Triazine Herbicides Induce Amyloid-β42 Production.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1593-1605, 2016.
, “Specific Triazine Herbicides Induce Amyloid-β42 Production.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1593-1605, 2016.
, “Specific Neuropsychiatric Symptoms Are Associated with Faster Progression in Alzheimer's Disease: Results of the Prospective Dementia Registry (PRODEM-Austria).”, J Alzheimers Dis, vol. 73, no. 1, pp. 125-133, 2020.
, “Specific Binding of Cu(II) Ions to Amyloid-Beta Peptides Bound to Aggregation-Inhibiting Molecules or SDS Micelles Creates Complexes that Generate Radical Oxygen Species.”, J Alzheimers Dis, vol. 54, no. 3, pp. 971-982, 2016.
, “Spatial Patterns of Longitudinal Gray Matter Change as Predictors of Concurrent Cognitive Decline in Amyloid Positive Healthy Subjects.”, J Alzheimers Dis, vol. 55, no. 1, pp. 343-358, 2017.
, “SPARCL1 Accelerates Symptom Onset in Alzheimer's Disease and Influences Brain Structure and Function During Aging.”, J Alzheimers Dis, vol. 61, no. 1, pp. 401-414, 2018.
, “SPARCL1 Accelerates Symptom Onset in Alzheimer's Disease and Influences Brain Structure and Function During Aging.”, J Alzheimers Dis, vol. 61, no. 1, pp. 401-414, 2018.
, “Social Robot Interventions for People with Dementia: A Systematic Review on Effects and Quality of Reporting.”, J Alzheimers Dis, vol. 79, no. 2, pp. 773-792, 2021.
, “Small Vessel Disease Burden Predicts Incident Dementia and Poor Functional Outcome in Independent Outpatients.”, J Alzheimers Dis, vol. 101, no. 3, pp. 751-760, 2024.
, “Small RNA Sequencing in the Tg4-42 Mouse Model Suggests the Involvement of snoRNAs in the Etiology of Alzheimer's Disease.”, J Alzheimers Dis, vol. 87, no. 4, pp. 1671-1681, 2022.
, “Small RNA Sequencing in the Tg4-42 Mouse Model Suggests the Involvement of snoRNAs in the Etiology of Alzheimer's Disease.”, J Alzheimers Dis, vol. 87, no. 4, pp. 1671-1681, 2022.
, “Small RNA Sequencing in the Tg4-42 Mouse Model Suggests the Involvement of snoRNAs in the Etiology of Alzheimer's Disease.”, J Alzheimers Dis, vol. 87, no. 4, pp. 1671-1681, 2022.
, “A Small Molecule Inhibitor of Plasminogen Activator Inhibitor-1 Reduces Brain Amyloid-β Load and Improves Memory in an Animal Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 64, no. 2, pp. 447-457, 2018.
, “Small Molecule Amyloid-β Protein Precursor Processing Modulators Lower Amyloid-β Peptide Levels via cKit Signaling.”, J Alzheimers Dis, vol. 67, no. 3, pp. 1089-1106, 2019.
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