Biblio

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Glutathione
A. Belkacemi and Ramassamy, C., “Anthocyanins Protect SK-N-SH Cells Against Acrolein-Induced Toxicity by Preserving the Cellular Redox State.”, J Alzheimers Dis, vol. 50, no. 4, pp. 981-98, 2016.
J. Paulo Alme Santos, Vizuete, A., Hansen, F., Biasibetti, R., and Gonçalves, C. - A., “Early and Persistent O-GlcNAc Protein Modification in the Streptozotocin Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 237-249, 2018.
Glutathione Disulfide
K. R. LeVault, Tischkau, S. A., and Brewer, G. J., “Circadian Disruption Reveals a Correlation of an Oxidative GSH/GSSG Redox Shift with Learning and Impaired Memory in an Alzheimer's Disease Mouse Model.”, J Alzheimers Dis, vol. 49, no. 2, pp. 301-16, 2016.
Glycation End Products, Advanced
J. Lynn, Park, M., Ogunwale, C., and Acquaah-Mensah, G. K., “A Tale of Two Diseases: Exploring Mechanisms Linking Diabetes Mellitus with Alzheimer's Disease.”, J Alzheimers Dis, vol. 85, no. 2, pp. 485-501, 2022.
Glycogen
A. Suzuki, Stern, S. A., Bozdagi, O., Huntley, G. W., Walker, R. H., Magistretti, P. J., and Alberini, C. M., “Astrocyte-neuron lactate transport is required for long-term memory formation.”, Cell, vol. 144, no. 5, pp. 810-23, 2011.
Glycogen Synthase Kinase 3
S. Hoon Choi, Kim, Y. Hye, Hebisch, M., Sliwinski, C., Lee, S., D'Avanzo, C., Chen, H., Hooli, B., Asselin, C., Muffat, J., Klee, J. B., Zhang, C., Wainger, B. J., Peitz, M., Kovacs, D. M., Woolf, C. J., Wagner, S. L., Tanzi, R. E., and Kim, D. Yeon, “A three-dimensional human neural cell culture model of Alzheimer's disease.”, Nature, vol. 515, no. 7526, pp. 274-8, 2014.
D. Chu, Tan, J., Xie, S., Jin, N., Yin, X., Gong, C. - X., Iqbal, K., and Liu, F., “GSK-3β is Dephosphorylated by PP2A in a Leu309 Methylation-Independent Manner.”, J Alzheimers Dis, vol. 49, no. 2, pp. 365-75, 2016.
X. - P. Hong, Chen, T., Yin, N. - N., Han, Y. - M., Yuan, F., Duan, Y. - J., Shen, F., Zhang, Y. - H., and Bin Chen, Z. -, “Puerarin Ameliorates D-Galactose Induced Enhanced Hippocampal Neurogenesis and Tau Hyperphosphorylation in Rat Brain.”, J Alzheimers Dis, vol. 51, no. 2, pp. 605-17, 2016.
A. Arner, Rockenstein, E., Mante, M., Florio, J., Masliah, D., Salehi, B., Adame, A., Overk, C., Masliah, E., and Rissman, R. A., “Increased Vulnerability of the Hippocampus in Transgenic Mice Overexpressing APP and Triple Repeat Tau.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1201-1219, 2018.
Glycogen Synthase Kinase 3 beta
D. Chu, Tan, J., Xie, S., Jin, N., Yin, X., Gong, C. - X., Iqbal, K., and Liu, F., “GSK-3β is Dephosphorylated by PP2A in a Leu309 Methylation-Independent Manner.”, J Alzheimers Dis, vol. 49, no. 2, pp. 365-75, 2016.
C. -cui Yang, Kuai, X. -xian, bin Gao, W. -, Yu, J. -chun, Wang, Q., Li, L., and Zhang, L., “Morroniside-Induced PP2A Activation Antagonizes Tau Hyperphosphorylation in a Cellular Model of Neurodegeneration.”, J Alzheimers Dis, vol. 51, no. 1, pp. 33-44, 2016.
C. Huang, Ng, O. Tsz- Wa, Ho, Y. - S., Irwin, M. Garnet, Chang, R. Chuen- Chun, and Wong, G. Tin- Chun, “Effect of Continuous Propofol Infusion in Rat on Tau Phosphorylation with or without Temperature Control.”, J Alzheimers Dis, vol. 51, no. 1, pp. 213-26, 2016.
H. - H. Li, Lin, S. - L., Huang, C. - N., Lu, F. - J., Chiu, P. - Y., Huang, W. - N., Lai, T. - J., and Lin, C. - L., “miR-302 Attenuates Amyloid-β-Induced Neurotoxicity through Activation of Akt Signaling.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1083-98, 2016.
Glycoproteins
M. Fà, Zhang, H., Staniszewski, A., Saeed, F., Shen, L. W., Schiefer, I. T., Siklos, M. I., Tapadar, S., Litosh, V. A., Libien, J., Petukhov, P. A., Teich, A. F., Thatcher, G. R. J., and Arancio, O., “Novel Selective Calpain 1 Inhibitors as Potential Therapeutics in Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 3, pp. 707-21, 2016.
R. Yu, Deochand, C., Krotow, A., Leão, R., Tong, M., Agarwal, A. R., Cadenas, E., and de la Monte, S. M., “Tobacco Smoke-Induced Brain White Matter Myelin Dysfunction: Potential Co-Factor Role of Smoking in Neurodegeneration.”, J Alzheimers Dis, vol. 50, no. 1, pp. 133-48, 2016.
Glycosides
C. -cui Yang, Kuai, X. -xian, bin Gao, W. -, Yu, J. -chun, Wang, Q., Li, L., and Zhang, L., “Morroniside-Induced PP2A Activation Antagonizes Tau Hyperphosphorylation in a Cellular Model of Neurodegeneration.”, J Alzheimers Dis, vol. 51, no. 1, pp. 33-44, 2016.
Glymphatic System
X. Guo, Zhang, G., Peng, Q., Huang, L., Zhang, Z., and Zhang, Z., “Emerging Roles of Meningeal Lymphatic Vessels in Alzheimer's Disease.”, J Alzheimers Dis, vol. 94, no. s1, pp. S355-S366, 2023.
Government Regulation
M. Pacheco-Herrero, Soto-Rojas, L. O., Reyes-Sabater, H., Garcés-Ramirez, L., López, Fde la Cruz, Villanueva-Fierro, I., and Luna-Muñoz, J., “Current Status and Challenges of Stem Cell Treatment for Alzheimer's Disease.”, J Alzheimers Dis, vol. 84, no. 3, pp. 917-935, 2021.
Gray Matter
N. - Y. Jung, Han, C. E., Kim, H. Jin, Yoo, S. Wook, Kim, H. - J., Kim, E. - J., Na, D. L., Lockhart, S. N., Jagust, W. J., Seong, J. - K., and Seo, S. Won, “Tract-Specific Correlates of Neuropsychological Deficits in Patients with Subcortical Vascular Cognitive Impairment.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1125-35, 2016.
E. Makovac, Serra, L., Spanò, B., Giulietti, G., Torso, M., Cercignani, M., Caltagirone, C., and Bozzali, M., “Different Patterns of Correlation between Grey and White Matter Integrity Account for Behavioral and Psychological Symptoms in Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 2, pp. 591-604, 2016.
B. N. Dugger, Whiteside, C. M., Maarouf, C. L., Walker, D. G., Beach, T. G., Sue, L. I., Garcia, A., Dunckley, T., Meechoovet, B., Reiman, E. M., and Roher, A. E., “The Presence of Select Tau Species in Human Peripheral Tissues and Their Relation to Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 2, pp. 345-56, 2016.
L. Serra, Cercignani, M., Mastropasqua, C., Torso, M., Spanò, B., Makovac, E., Viola, V., Giulietti, G., Marra, C., Caltagirone, C., and Bozzali, M., “Longitudinal Changes in Functional Brain Connectivity Predicts Conversion to Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 2, pp. 377-89, 2016.
P. J. Lao, Handen, B. L., Betthauser, T. J., Mihaila, I., Hartley, S. L., Cohen, A. D., Tudorascu, D. L., Bulova, P. D., Lopresti, B. J., Tumuluru, R. V., Murali, D., Mathis, C. A., Barnhart, T. E., Stone, C. K., Price, J. C., Devenny, D. A., Johnson, S. C., Klunk, W. E., and Christian, B. T., “Alzheimer-Like Pattern of Hypometabolism Emerges with Elevated Amyloid-β Burden in Down Syndrome.”, J Alzheimers Dis, vol. 61, no. 2, pp. 631-644, 2018.
A. Kimura, Yoshikura, N., Hayashi, Y., and Inuzuka, T., “Cerebrospinal Fluid C-C Motif Chemokine Ligand 2 Correlates with Brain Atrophy and Cognitive Impairment in Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 2, pp. 581-588, 2018.
A. Tucholka, Grau-Rivera, O., Falcon, C., Rami, L., Sánchez-Valle, R., Lladó, A., Gispert, J. Domingo, and Molinuevo, J. Luis, “Structural Connectivity Alterations Along the Alzheimer's Disease Continuum: Reproducibility Across Two Independent Samples and Correlation with Cerebrospinal Fluid Amyloid-β and Tau.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1575-1587, 2018.

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