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Amyloid
C. M. Roe, Babulal, G. M., Mishra, S., Gordon, B. A., Stout, S. H., Ott, B. R., Carr, D. B., Ances, B. M., Morris, J. C., and Benzinger, T. L. S., “Tau and Amyloid Positron Emission Tomography Imaging Predict Driving Performance Among Older Adults with and without Preclinical Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 2, pp. 509-513, 2018.
Amyloid beta-Peptides
T. Kondo, Asai, M., Tsukita, K., Kutoku, Y., Ohsawa, Y., Sunada, Y., Imamura, K., Egawa, N., Yahata, N., Okita, K., Takahashi, K., Asaka, I., Aoi, T., Watanabe, A., Watanabe, K., Kadoya, C., Nakano, R., Watanabe, D., Maruyama, K., Hori, O., Hibino, S., Choshi, T., Nakahata, T., Hioki, H., Kaneko, T., Naitoh, M., Yoshikawa, K., Yamawaki, S., Suzuki, S., Hata, R., Ueno, S. - I., Seki, T., Kobayashi, K., Toda, T., Murakami, K., Irie, K., Klein, W. L., Mori, H., Asada, T., Takahashi, R., Iwata, N., Yamanaka, S., and Inoue, H., “Modeling Alzheimer's disease with iPSCs reveals stress phenotypes associated with intracellular Aβ and differential drug responsiveness.”, Cell Stem Cell, vol. 12, no. 4, pp. 487-96, 2013.
T. Kondo, Asai, M., Tsukita, K., Kutoku, Y., Ohsawa, Y., Sunada, Y., Imamura, K., Egawa, N., Yahata, N., Okita, K., Takahashi, K., Asaka, I., Aoi, T., Watanabe, A., Watanabe, K., Kadoya, C., Nakano, R., Watanabe, D., Maruyama, K., Hori, O., Hibino, S., Choshi, T., Nakahata, T., Hioki, H., Kaneko, T., Naitoh, M., Yoshikawa, K., Yamawaki, S., Suzuki, S., Hata, R., Ueno, S. - I., Seki, T., Kobayashi, K., Toda, T., Murakami, K., Irie, K., Klein, W. L., Mori, H., Asada, T., Takahashi, R., Iwata, N., Yamanaka, S., and Inoue, H., “Modeling Alzheimer's disease with iPSCs reveals stress phenotypes associated with intracellular Aβ and differential drug responsiveness.”, Cell Stem Cell, vol. 12, no. 4, pp. 487-96, 2013.
R. Smith, Wibom, M., Olsson, T., Hägerström, D., Jögi, J., Rabinovici, G. D., and Hansson, O., “Posterior Accumulation of Tau and Concordant Hypometabolism in an Early-Onset Alzheimer's Disease Patient with Presenilin-1 Mutation.”, J Alzheimers Dis, vol. 51, no. 2, pp. 339-43, 2016.
J. Spiegel, Pirraglia, E., Osorio, R. S., Glodzik, L., Li, Y., Tsui, W., Louis, L. A. Saint, Randall, C., Butler, T., Xu, J., Zinkowski, R. P., Zetterberg, H., Fortea, J., Fossati, S., Wisniewski, T., Davies, P., Blennow, K., and de Leon, M. J., “Greater specificity for cerebrospinal fluid P-tau231 over P-tau181 in the differentiation of healthy controls from Alzheimer's disease.”, J Alzheimers Dis, vol. 49, no. 1, pp. 93-100, 2016.
S. V. Ovsepian and O'Leary, V. B., “Neuronal activity and amyloid plaque pathology: an update.”, J Alzheimers Dis, vol. 49, no. 1, pp. 13-9, 2016.
S. V. Ovsepian and O'Leary, V. B., “Neuronal activity and amyloid plaque pathology: an update.”, J Alzheimers Dis, vol. 49, no. 1, pp. 13-9, 2016.
M. Bjerke, Kern, S., Blennow, K., Zetterberg, H., Waern, M., Börjesson-Hanson, A., Östling, S., Kern, J., and Skoog, I., “Cerebrospinal Fluid Fatty Acid-Binding Protein 3 is Related to Dementia Development in a Population-Based Sample of Older Adult Women Followed for 8 Years.”, J Alzheimers Dis, vol. 49, no. 3, pp. 733-41, 2016.
A. Savastano, Klafki, H., Haußmann, U., Oberstein, T. Jan, Muller, P., Wirths, O., Wiltfang, J., and Bayer, T. A., “N-truncated Aβ2-X starting with position two in sporadic Alzheimer's disease cases and two Alzheimer mouse models.”, J Alzheimers Dis, vol. 49, no. 1, pp. 101-10, 2016.
J. Vallortigara, Whitfield, D., Quelch, W., Alghamdi, A., Howlett, D., Hortobágyi, T., Johnson, M., Attems, J., O'Brien, J. T., Thomas, A., Ballard, C. G., Aarsland, D., and Francis, P. T., “Decreased Levels of VAMP2 and Monomeric Alpha-Synuclein Correlate with Duration of Dementia.”, J Alzheimers Dis, vol. 50, no. 1, pp. 101-10, 2016.
S. Lim, Choi, J. Gyu, Moon, M., Kim, H. Geun, Lee, W., Bak, H. - R., Sung, H., Park, C. Hye, Kim, S. Yeou, and Oh, M. Sook, “An Optimized Combination of Ginger and Peony Root Effectively Inhibits Amyloid-β Accumulation and Amyloid-β-Mediated Pathology in AβPP/PS1 Double-Transgenic Mice.”, J Alzheimers Dis, vol. 50, no. 1, pp. 189-200, 2016.
K. E. Melah, Lu, S. Yuan- Fu, Hoscheidt, S. M., Alexander, A. L., Adluru, N., Destiche, D. J., Carlsson, C. M., Zetterberg, H., Blennow, K., Okonkwo, O. C., Gleason, C. E., N Dowling, M., Bratzke, L. C., Rowley, H. A., Sager, M. A., Asthana, S., Johnson, S. C., and Bendlin, B. B., “Cerebrospinal Fluid Markers of Alzheimer's Disease Pathology and Microglial Activation are Associated with Altered White Matter Microstructure in Asymptomatic Adults at Risk for Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 3, pp. 873-86, 2016.
C. Peters, Bascuñán, D., Opazo, C., and Aguayo, L. G., “Differential Membrane Toxicity of Amyloid-β Fragments by Pore Forming Mechanisms.”, J Alzheimers Dis, vol. 51, no. 3, pp. 689-99, 2016.
B. Ongali, Nicolakakis, N., Tong, X. - K., Aboulkassim, T., Imboden, H., and Hamel, E., “Enalapril Alone or Co-Administered with Losartan Rescues Cerebrovascular Dysfunction, but not Mnemonic Deficits or Amyloidosis in a Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1183-95, 2016.
N. Lelental, Brandner, S., Kofanova, O., Blennow, K., Zetterberg, H., Andreasson, U., Engelborghs, S., Mroczko, B., Gabryelewicz, T., Teunissen, C., Mollenhauer, B., Parnetti, L., Chiasserini, D., Molinuevo, J. Luis, Perret-Liaudet, A., Verbeek, M. M., Andreasen, N., Brosseron, F., Bahl, J. M. C., Herukka, S. - K., Hausner, L., Frölich, L., Labonte, A., Poirier, J., Miller, A. - M., Zilka, N., Kovacech, B., Urbani, A., Suardi, S., Oliveira, C., Baldeiras, I., Dubois, B., Rot, U., Lehmann, S., Skinningsrud, A., Betsou, F., Wiltfang, J., Gkatzima, O., Winblad, B., Buchfelder, M., Kornhuber, J., and Lewczuk, P., “Comparison of Different Matrices as Potential Quality Control Samples for Neurochemical Dementia Diagnostics.”, J Alzheimers Dis, vol. 52, no. 1, pp. 51-64, 2016.
M. Ángel Ontiveros-Torres, Labra-Barrios, M. Luisa, Díaz-Cintra, S., Aguilar-Vázquez, A. Ruth, Moreno-Campuzano, S., Flores-Rodríguez, P., Luna-Herrera, C., Mena, R., Perry, G., Florán-Garduño, B., Luna-Muñoz, J., and Luna-Arias, J. Pedro, “Fibrillar Amyloid-β Accumulation Triggers an Inflammatory Mechanism Leading to Hyperphosphorylation of the Carboxyl-Terminal End of Tau Polypeptide in the Hippocampal Formation of the 3×Tg-AD Transgenic Mouse.”, J Alzheimers Dis, vol. 52, no. 1, pp. 243-69, 2016.
S. L. C. Geijselaers, Aalten, P., Ramakers, I. H. G. B., De Deyn, P. Paul, Heijboer, A. C., Koek, H. L., OldeRikkert, M. G. M., Papma, J. M., Reesink, F. E., Smits, L. L., Stehouwer, C. D. A., Teunissen, C. E., Verhey, F. R. J., van der Flier, W. M., and Biessels, G. Jan, “Association of Cerebrospinal Fluid (CSF) Insulin with Cognitive Performance and CSF Biomarkers of Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 1, pp. 309-320, 2018.
N. Valech, Tort-Merino, A., Coll-Padrós, N., Olives, J., León, M., Rami, L., and Molinuevo, J. Luis, “Executive and Language Subjective Cognitive Decline Complaints Discriminate Preclinical Alzheimer's Disease from Normal Aging.”, J Alzheimers Dis, vol. 61, no. 2, pp. 689-703, 2018.
H. P Reddy, Manczak, M., Yin, X. L., Grady, M. Catherine, Mitchell, A., Tonk, S., Kuruva, C. Sekhar, Bhatti, J. Singh, Kandimalla, R., Vijayan, M., Kumar, S., Wang, R., Pradeepkiran, J. Adi, Ogunmokun, G., Thamarai, K., Quesada, K., Boles, A., and Reddy, A. P., “Protective Effects of Indian Spice Curcumin Against Amyloid-β in Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 3, pp. 843-866, 2018.
A. Arner, Rockenstein, E., Mante, M., Florio, J., Masliah, D., Salehi, B., Adame, A., Overk, C., Masliah, E., and Rissman, R. A., “Increased Vulnerability of the Hippocampus in Transgenic Mice Overexpressing APP and Triple Repeat Tau.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1201-1219, 2018.
G. Boström, Freyhult, E., Virhammar, J., Alcolea, D., Tumani, H., Otto, M., Brundin, R. - M., Kilander, L., Löwenmark, M., Giedraitis, V., Lleo, A., von Arnim, C. A. F., Kultima, K., and Ingelsson, M., “Different Inflammatory Signatures in Alzheimer's Disease and Frontotemporal Dementia Cerebrospinal Fluid.”, J Alzheimers Dis, vol. 81, no. 2, pp. 629-640, 2021.
M. Ettcheto, Busquets, O., Cano, A., Sánchez-López, E., Manzine, P. R., Espinosa-Jimenez, T., Verdaguer, E., Sureda, F. X., Olloquequi, J., Castro-Torres, R. D., Auladell, C., Folch, J., Casadesus, G., and Camins, A., “Pharmacological Strategies to Improve Dendritic Spines in Alzheimer's Disease.”, J Alzheimers Dis, vol. 82, no. s1, pp. S91-S107, 2021.
M. Abdelhamid, Zhou, C., Ohno, K., Kuhara, T., Taslima, F., Abdullah, M., Jung, C. - G., and Michikawa, M., “Probiotic Bifidobacterium breve Prevents Memory Impairment Through the Reduction of Both Amyloid-β Production and Microglia Activation in APP Knock-In Mouse.”, J Alzheimers Dis, vol. 85, no. 4, pp. 1555-1571, 2022.
H. Shinagawa, Ohuchi, K., Goto, Y., Hashimoto, K., Kijima, H., Maekawa, S., Kurita, H., and Inden, M., “Vacuolar Protein-Sorting Proteins Are Reduced Even Before Cognitive Decline in a Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 96, no. 3, pp. 1011-1017, 2023.
N. Ortí-Casañ, Wajant, H., H Kuiperij, B., Hooijsma, A., Tromp, L., Poortman, I. L., Tadema, N., de Lange, J. H. E., Verbeek, M. M., De Deyn, P. P., Naudé, P. J. W., and Eisel, U. L. M., “Activation of TNF Receptor 2 Improves Synaptic Plasticity and Enhances Amyloid-β Clearance in an Alzheimer's Disease Mouse Model with Humanized TNF Receptor 2.”, J Alzheimers Dis, vol. 94, no. 3, pp. 977-991, 2023.

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