Biblio
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“Increased Levels of Extracellular Microvesicle Markers and Decreased Levels of Endocytic/Exocytic Proteins in the Alzheimer's Disease Brain.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1671-1686, 2016.
, “Increased Release of Apolipoprotein E in Extracellular Vesicles Following Amyloid-β Protofibril Exposure of Neuroglial Co-Cultures.”, J Alzheimers Dis, vol. 60, no. 1, pp. 305-321, 2017.
, “Increased Risk of Dementia in Subjective Cognitive Decline if CT Brain Changes are Present.”, J Alzheimers Dis, vol. 66, no. 2, pp. 483-495, 2018.
, “Influence of genotype on PCSK9-lipids association in cerebrospinal fluid and serum of patients in the Alzheimer's disease continuum.”, J Alzheimers Dis, vol. 102, no. 1, pp. 162-172, 2024.
, “An Insertion Within SIRPβ1 Shows a Dual Effect Over Alzheimer's Disease Cognitive Decline Altering the Microglial Response.”, J Alzheimers Dis, vol. 98, no. 2, pp. 601-618, 2024.
, “Integrated systems approach identifies genetic nodes and networks in late-onset Alzheimer's disease.”, Cell, vol. 153, no. 3, pp. 707-20, 2013.
, “Intelligent Assistive Technology for Alzheimer's Disease and Other Dementias: A Systematic Review.”, J Alzheimers Dis, vol. 56, no. 4, pp. 1301-1340, 2017.
, “Interaction between Cytomegalovirus and Herpes Simplex Virus Type 1 Associated with the Risk of Alzheimer's Disease Development.”, J Alzheimers Dis, vol. 61, no. 3, pp. 939-945, 2018.
, “Interaction between Cytomegalovirus and Herpes Simplex Virus Type 1 Associated with the Risk of Alzheimer's Disease Development.”, J Alzheimers Dis, vol. 61, no. 3, pp. 939-945, 2018.
, “An Intracellular Amyloid-β/AβPP Epitope Correlates with Neurodegeneration in those Neuronal Populations Early Involved in Alzheimer's Disease.”, J Alzheimers Dis, vol. 59, no. 3, pp. 1079-1096, 2017.
, “Late Onset Alzheimer's Disease Risk Variants in Cognitive Decline: The PATH Through Life Study.”, J Alzheimers Dis, vol. 57, no. 2, pp. 423-436, 2017.
, “Left Frontal Hub Connectivity during Memory Performance Supports Reserve in Aging and Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 59, no. 4, pp. 1381-1392, 2017.
, “Left Frontal Hub Connectivity during Memory Performance Supports Reserve in Aging and Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 59, no. 4, pp. 1381-1392, 2017.
, “Less Daily Computer Use is Related to Smaller Hippocampal Volumes in Cognitively Intact Elderly.”, J Alzheimers Dis, vol. 52, no. 2, pp. 713-7, 2016.
, “Linking Air Pollution Exposure to Blood-Based Metabolic Features in a Community-Based Aging Cohort with and without Dementia.”, J Alzheimers Dis, vol. 96, no. 3, pp. 1025-1040, 2023.
, “Living Alone with Alzheimer's Disease: Data from SveDem, the Swedish Dementia Registry.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1265-1272, 2017.
, “Living Alone with Dementia: Prevalence, Correlates and the Utilization of Health and Nursing Care Services.”, J Alzheimers Dis, vol. 52, no. 2, pp. 619-29, 2016.
, “Longitudinal Effects of Herpesviruses on Multiple Cognitive Outcomes in Healthy Elderly Adults.”, J Alzheimers Dis, vol. 94, no. 2, pp. 751-762, 2023.
, “Longitudinal Relationships between Caloric Expenditure and Gray Matter in the Cardiovascular Health Study.”, J Alzheimers Dis, vol. 52, no. 2, pp. 719-29, 2016.
, “Longitudinal Relationships between Caloric Expenditure and Gray Matter in the Cardiovascular Health Study.”, J Alzheimers Dis, vol. 52, no. 2, pp. 719-29, 2016.
, “A Longitudinal Study of Total and Phosphorylated α-Synuclein with Other Biomarkers in Cerebrospinal Fluid of Alzheimer's Disease and Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 61, no. 4, pp. 1541-1553, 2018.
, “Long-Term Cognitive Decline in Dementia with Lewy Bodies in a Large Multicenter, International Cohort.”, J Alzheimers Dis, vol. 57, no. 3, pp. 787-795, 2017.
, “Low Cognitive Awareness, but Not Complaint, is a Good Marker of Preclinical Alzheimer's Disease.”, J Alzheimers Dis, vol. 59, no. 2, pp. 753-762, 2017.
, “Low Serum Insulin-Like Growth Factor-I Predicts Cognitive Decline in Alzheimer's Disease.”, J Alzheimers Dis, vol. 52, no. 2, pp. 641-9, 2016.
, “MAPT H1 Haplotype is Associated with Late-Onset Alzheimer's Disease Risk in APOEɛ4 Noncarriers: Results from the Dementia Genetics Spanish Consortium.”, J Alzheimers Dis, vol. 49, no. 2, pp. 343-52, 2016.
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