Biblio
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Author [ Title] Type Year Filters: First Letter Of Last Name is I [Clear All Filters]
“A Multi-Cohort Study of ApoE ɛ4 and Amyloid-β Effects on the Hippocampus in Alzheimer's Disease.”, J Alzheimers Dis, vol. 56, no. 3, pp. 1159-1174, 2017.
, “A Multi-Cohort Study of ApoE ɛ4 and Amyloid-β Effects on the Hippocampus in Alzheimer's Disease.”, J Alzheimers Dis, vol. 56, no. 3, pp. 1159-1174, 2017.
, “Multiple Mechanisms Linking Type 2 Diabetes and Alzheimer's Disease: Testosterone as a Modifier.”, J Alzheimers Dis, vol. 59, no. 2, pp. 445-466, 2017.
, “The Need to Separate Chronic Traumatic Encephalopathy Neuropathology from Clinical Features.”, J Alzheimers Dis, vol. 61, no. 1, pp. 17-28, 2018.
, “Neural Basis of Apathy in Patients with Amnestic Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1403-1416, 2017.
, “Neural Correlates of Sleep Disturbance in Alzheimer's Disease: Role of the Precuneus in Sleep Disturbance.”, J Alzheimers Dis, vol. 63, no. 3, pp. 957-964, 2018.
, “Neurofibrillary Tangles of Aβx-40 in Alzheimer's Disease Brains.”, J Alzheimers Dis, vol. 58, no. 3, pp. 661-667, 2017.
, “Neuroimaging Feature Terminology: A Controlled Terminology for the Annotation of Brain Imaging Features.”, J Alzheimers Dis, vol. 59, no. 4, pp. 1153-1169, 2017.
, “Neuro-Immuno-Gene- and Genome-Editing-Therapy for Alzheimer's Disease: Are We There Yet?”, J Alzheimers Dis, vol. 65, no. 2, pp. 321-344, 2018.
, “Neuroprotective Effects of Ferruginol, Jatrophone, and Junicedric Acid Against Amyloid-β Injury in Hippocampal Neurons.”, J Alzheimers Dis, vol. 63, no. 2, pp. 705-723, 2018.
, “Neuropsychiatric Symptoms and Cognitive Impairment: Understanding the Importance of Co-Morbid Symptoms.”, J Alzheimers Dis, vol. 59, no. 1, pp. 141-153, 2017.
, “Neuropsychological Features of Microbleeds and Cortical Microinfarct Detected by High Resolution Magnetic Resonance Imaging.”, J Alzheimers Dis, vol. 53, no. 1, pp. 315-25, 2016.
, “Neurotrophic Treatment Initiated During Early Postnatal Development Prevents the Alzheimer-Like Behavior and Synaptic Dysfunction.”, J Alzheimers Dis, vol. 82, no. 2, pp. 631-646, 2021.
, “New BACE1 Chimeric Peptide Inhibitors Selectively Prevent AβPPβ Cleavage Decreasing Amyloid-β Production and Accumulation in Alzheimer's Disease Models.”, J Alzheimers Dis, vol. 76, no. 4, pp. 1317-1337, 2020.
, “New Insights into the Spontaneous Human Alzheimer's Disease-Like Model Octodon degus: Unraveling Amyloid-β Peptide Aggregation and Age-Related Amyloid Pathology.”, J Alzheimers Dis, vol. 66, no. 3, pp. 1145-1163, 2018.
, “A New Serum Biomarker Set to Detect Mild Cognitive Impairment and Alzheimer's Disease by Peptidome Technology.”, J Alzheimers Dis, vol. 73, no. 1, pp. 217-227, 2020.
, “A New Serum Biomarker Set to Detect Mild Cognitive Impairment and Alzheimer's Disease by Peptidome Technology.”, J Alzheimers Dis, vol. 73, no. 1, pp. 217-227, 2020.
, “A Novel Cognitive Function Scale Using Functional Near-Infrared Spectroscopy for Evaluating Cognitive Dysfunction.”, J Alzheimers Dis, vol. 81, no. 4, pp. 1579-1588, 2021.
, “Novel GRN Mutations in Alzheimer's Disease and Frontotemporal Lobar Degeneration.”, J Alzheimers Dis, vol. 62, no. 4, pp. 1683-1689, 2018.
, “A Novel Therapeutic Approach to Treat Alzheimer's Disease by Neurotrophic Support During the Period of Synaptic Compensation.”, J Alzheimers Dis, vol. 62, no. 3, pp. 1211-1218, 2018.
, “NP03, a Microdose Lithium Formulation, Blunts Early Amyloid Post-Plaque Neuropathology in McGill-R-Thy1-APP Alzheimer-Like Transgenic Rats.”, J Alzheimers Dis, vol. 73, no. 2, pp. 723-739, 2019.
, “Nutrition: Review on the Possible Treatment for Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 3, pp. 867-883, 2018.
, “Nutritional Status of Patients with Alzheimer's Disease and Their Caregivers.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1619-1627, 2016.
, “Of Mice and Men: Comparative Analysis of Neuro-Inflammatory Mechanisms in Human and Mouse Using Cause-and-Effect Models.”, J Alzheimers Dis, vol. 59, no. 3, pp. 1045-1055, 2017.
, “Older Patients with Alzheimer's Disease-Related Cortical Atrophy Who Develop Post-Operative Delirium May Be at Increased Risk of Long-Term Cognitive Decline After Surgery.”, J Alzheimers Dis, vol. 75, no. 1, pp. 187-199, 2020.
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