Biblio
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“MAPT H1 Haplotype is Associated with Late-Onset Alzheimer's Disease Risk in APOEɛ4 Noncarriers: Results from the Dementia Genetics Spanish Consortium.”, J Alzheimers Dis, vol. 49, no. 2, pp. 343-52, 2016.
, “Measuring Information Processing Speed in Mild Cognitive Impairment: Clinical Versus Research Dichotomy.”, J Alzheimers Dis, vol. 51, no. 1, pp. 263-75, 2016.
, “Memory Binding Test Predicts Incident Amnestic Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1585-95, 2016.
, “Memory for Public Events in Mild Cognitive Impairment and Alzheimer's Disease: The Importance of Rehearsal.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1023-33, 2016.
, “Midlife Physical Activity and Cognition Later in Life: A Prospective Twin Study.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1303-1317, 2016.
, “Mindfulness in the Maintenance of Cognitive Capacities in Alzheimer's Disease: A Randomized Clinical Trial.”, J Alzheimers Dis, vol. 50, no. 1, pp. 217-32, 2016.
, “Mismatch Negativity and Ear Laterality in Alzheimer's Disease and in Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1405-10, 2016.
, “Molecular Signaling Mechanisms of Natural and Synthetic Retinoids for Inhibition of Pathogenesis in Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 2, pp. 335-52, 2016.
, “Molecular Signaling Mechanisms of Natural and Synthetic Retinoids for Inhibition of Pathogenesis in Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 2, pp. 335-52, 2016.
, “Monitoring Blood-Brain Barrier Integrity Following Amyloid-β Immunotherapy Using Gadolinium-Enhanced MRI in a PDAPP Mouse Model.”, J Alzheimers Dis, vol. 54, no. 2, pp. 723-35, 2016.
, “Monophosphoryl Lipid-A: A Promising Tool for Alzheimer's Disease Toll.”, J Alzheimers Dis, vol. 52, no. 4, pp. 1189-202, 2016.
, “Monophosphoryl Lipid-A: A Promising Tool for Alzheimer's Disease Toll.”, J Alzheimers Dis, vol. 52, no. 4, pp. 1189-202, 2016.
, “Monophosphoryl Lipid-A: A Promising Tool for Alzheimer's Disease Toll.”, J Alzheimers Dis, vol. 52, no. 4, pp. 1189-202, 2016.
, “Moving from the Dish to the Clinical Practice: A Decade of Lessons and Perspectives from the Pre-Clinical and Clinical Stem Cell Studies for Alzheimer's Disease.”, J Alzheimers Dis, vol. 53, no. 3, pp. 1209-30, 2016.
, “Multifunctional Effect of Human Serum Albumin Reduces Alzheimer's Disease Related Pathologies in the 3xTg Mouse Model.”, J Alzheimers Dis, vol. 50, no. 1, pp. 175-88, 2016.
, “Multifunctional Effect of Human Serum Albumin Reduces Alzheimer's Disease Related Pathologies in the 3xTg Mouse Model.”, J Alzheimers Dis, vol. 50, no. 1, pp. 175-88, 2016.
, “Music and Memory in Alzheimer's Disease and The Potential Underlying Mechanisms.”, J Alzheimers Dis, vol. 51, no. 4, pp. 949-59, 2016.
, “Netrin-1 Interrupts Amyloid-β Amplification, Increases sAβPPα in vitro and in vivo, and Improves Cognition in a Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 52, no. 1, pp. 223-42, 2016.
, “Neurocognitive Deficits Distinguishing Mild Dementia with Lewy Bodies from Mild Alzheimer's Disease are Associated with Parkinsonism.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1277-85, 2016.
, “Neuroimaging Correlates of Frontotemporal Dementia Associated with SQSTM1 Mutations.”, J Alzheimers Dis, vol. 53, no. 1, pp. 303-13, 2016.
, “Neuroimaging Correlates of Frontotemporal Dementia Associated with SQSTM1 Mutations.”, J Alzheimers Dis, vol. 53, no. 1, pp. 303-13, 2016.
, “Neurophysiological assessment of Alzheimer's disease individuals by a single electroencephalographic marker.”, J Alzheimers Dis, vol. 49, no. 1, pp. 159-77, 2016.
, “Neuropsychological Markers of Medial Perirhinal and Entorhinal Cortex Functioning are Impaired Twelve Years Preceding Diagnosis of Alzheimer's Dementia.”, J Alzheimers Dis, vol. 52, no. 2, pp. 573-80, 2016.
, “Non-Verbal Episodic Memory Deficits in Primary Progressive Aphasias are Highly Predictive of Underlying Amyloid Pathology.”, J Alzheimers Dis, vol. 51, no. 2, pp. 367-76, 2016.
, “Non-Verbal Episodic Memory Deficits in Primary Progressive Aphasias are Highly Predictive of Underlying Amyloid Pathology.”, J Alzheimers Dis, vol. 51, no. 2, pp. 367-76, 2016.
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