Biblio

Export 238 results:
[ Author(Desc)] Title Type Year
Filters: First Letter Of Keyword is G  [Clear All Filters]
A B C D E F G H I J K L M N O P Q R S T U V W X Y Z 
Y
C. -cui Yang, Kuai, X. -xian, bin Gao, W. -, Yu, J. -chun, Wang, Q., Li, L., and Zhang, L., Morroniside-Induced PP2A Activation Antagonizes Tau Hyperphosphorylation in a Cellular Model of Neurodegeneration., J Alzheimers Dis, vol. 51, no. 1, pp. 33-44, 2016.
C. -cui Yang, Kuai, X. -xian, bin Gao, W. -, Yu, J. -chun, Wang, Q., Li, L., and Zhang, L., Morroniside-Induced PP2A Activation Antagonizes Tau Hyperphosphorylation in a Cellular Model of Neurodegeneration., J Alzheimers Dis, vol. 51, no. 1, pp. 33-44, 2016.
Q. Ye, Bai, F., and Zhang, Z., Shared Genetic Risk Factors for Late-Life Depression and Alzheimer's Disease., J Alzheimers Dis, vol. 52, no. 1, pp. 1-15, 2016.
T. June Yoo, Anti-Inflammatory Gene Therapy Improves Spatial Memory Performance in a Mouse Model of Alzheimer's Disease., J Alzheimers Dis, vol. 85, no. 3, pp. 1001-1008, 2022.
B. Yoon, Shim, Y. S., Park, H. - K., Park, S. Ah, Choi, S. Hye, and Yang, D. Won, Predictive factors for disease progression in patients with early-onset Alzheimer's disease., J Alzheimers Dis, vol. 49, no. 1, pp. 85-91, 2016.
R. Yu, Deochand, C., Krotow, A., Leão, R., Tong, M., Agarwal, A. R., Cadenas, E., and de la Monte, S. M., Tobacco Smoke-Induced Brain White Matter Myelin Dysfunction: Potential Co-Factor Role of Smoking in Neurodegeneration., J Alzheimers Dis, vol. 50, no. 1, pp. 133-48, 2016.
R. Yu, Deochand, C., Krotow, A., Leão, R., Tong, M., Agarwal, A. R., Cadenas, E., and de la Monte, S. M., Tobacco Smoke-Induced Brain White Matter Myelin Dysfunction: Potential Co-Factor Role of Smoking in Neurodegeneration., J Alzheimers Dis, vol. 50, no. 1, pp. 133-48, 2016.
R. Yu, Deochand, C., Krotow, A., Leão, R., Tong, M., Agarwal, A. R., Cadenas, E., and de la Monte, S. M., Tobacco Smoke-Induced Brain White Matter Myelin Dysfunction: Potential Co-Factor Role of Smoking in Neurodegeneration., J Alzheimers Dis, vol. 50, no. 1, pp. 133-48, 2016.
Z
D. Zádori, Veres, G., Szalárdy, L., Klivenyi, P., and Vecsei, L., Alzheimer's Disease: Recent Concepts on the Relation of Mitochondrial Disturbances, Excitotoxicity, Neuroinflammation, and Kynurenines., J Alzheimers Dis, vol. 62, no. 2, pp. 523-547, 2018.
R. Zanardini, Benussi, L., Fostinelli, S., Saraceno, C., Ciani, M., Borroni, B., Padovani, A., Binetti, G., and Ghidoni, R., Serum C-Peptide, Visfatin, Resistin, and Ghrelin are Altered in Sporadic and GRN-Associated Frontotemporal Lobar Degeneration., J Alzheimers Dis, vol. 61, no. 3, pp. 1053-1060, 2018.
W. Zhang, Guo, Y., Li, B., Zhang, Q., Liu, J. - H., Gu, G. - J., Wang, J. - H., Bao, R. - K., Chen, Y. - J., and Xu, J. - R., GDF11 Rejuvenates Cerebrovascular Structure and Function in an Animal Model of Alzheimer's Disease., J Alzheimers Dis, vol. 62, no. 2, pp. 807-819, 2018.
B. Zhang, Gaiteri, C., Bodea, L. - G., Wang, Z., McElwee, J., Podtelezhnikov, A. A., Zhang, C., Xie, T., Tran, L., Dobrin, R., Fluder, E., Clurman, B., Melquist, S., Narayanan, M., Suver, C., Shah, H., Mahajan, M., Gillis, T., Mysore, J., MacDonald, M. E., Lamb, J. R., Bennett, D. A., Molony, C., Stone, D. J., Gudnason, V., Myers, A. J., Schadt, E. E., Neumann, H., Zhu, J., and Emilsson, V., Integrated systems approach identifies genetic nodes and networks in late-onset Alzheimer's disease., Cell, vol. 153, no. 3, pp. 707-20, 2013.
M. D. Zwan, Villemagne, V. L., Doré, V., Buckley, R., Bourgeat, P., Veljanoski, R., Salvado, O., Williams, R., Margison, L., Rembach, A., S Macaulay, L., Martins, R., Ames, D., van der Flier, W. M., Ellis, K. A., Scheltens, P., Masters, C. L., and Rowe, C. C., Subjective Memory Complaints in APOEɛ4 Carriers are Associated with High Amyloid-β Burden., J Alzheimers Dis, vol. 49, no. 4, pp. 1115-22, 2016.

Pages