Biblio

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Journal Article
C. R. Jack, Knopman, D. S., Weigand, S. D., Wiste, H. J., Vemuri, P., Lowe, V., Kantarci, K., Gunter, J. L., Senjem, M. L., Ivnik, R. J., Roberts, R. O., Rocca, W. A., Boeve, B. F., and Petersen, R. C., “An operational approach to National Institute on Aging-Alzheimer's Association criteria for preclinical Alzheimer disease.”, Ann Neurol, vol. 71, no. 6, pp. 765-75, 2012.
J. Antonio Allué, Sarasa, L., Izco, M., Pérez-Grijalba, V., Fandos, N., Pascual-Lucas, M., Ogueta, S., Pesini, P., and Sarasa, M., “Outstanding Phenotypic Differences in the Profile of Amyloid-β between Tg2576 and APPswe/PS1dE9 Transgenic Mouse Models of Alzheimer's Disease.”, J Alzheimers Dis, vol. 53, no. 3, pp. 773-85, 2016.
M. Wezyk, Szybinska, A., Wojsiat, J., Szczerba, M., Day, K., Ronnholm, H., Kele, M., Berdynski, M., Peplonska, B., Fichna, J. Piotr, Ilkowski, J., Styczyńska, M., Barczak, A., Zboch, M., Filipek-Gliszczyńska, A., Bojakowski, K., Skrzypczak, M., Ginalski, K., Kabza, M., Makalowska, I., Barcikowska-Kotowicz, M., Wojda, U., Falk, A., and Zekanowski, C., “Overactive BRCA1 Affects Presenilin 1 in Induced Pluripotent Stem Cell-Derived Neurons in Alzheimer's Disease.”, J Alzheimers Dis, vol. 62, no. 1, pp. 175-202, 2018.
J. J. Iliff, Wang, M., Liao, Y., Plogg, B. A., Peng, W., Gundersen, G. A., Benveniste, H., G Vates, E., Deane, R., Goldman, S. A., Nagelhus, E. A., and Nedergaard, M., “A paravascular pathway facilitates CSF flow through the brain parenchyma and the clearance of interstitial solutes, including amyloid β.”, Sci Transl Med, vol. 4, no. 147, p. 147ra111, 2012.
B. Hanseeuw, Dricot, L., Lhommel, R., Quenon, L., and Ivanoiu, A., “Patients with Amyloid-Negative Mild Cognitive Impairment have Cortical Hypometabolism but the Hippocampus is Preserved.”, J Alzheimers Dis, vol. 53, no. 2, pp. 651-60, 2016.
M. Okuda, Fujita, Y., Hijikuro, I., Wada, M., Uemura, T., Kobayashi, Y., Waku, T., Tanaka, N., Nishimoto, T., Izumi, Y., Kume, T., Akaike, A., Takahashi, T., and Sugimoto, H., “PE859, A Novel Curcumin Derivative, Inhibits Amyloid-β and Tau Aggregation, and Ameliorates Cognitive Dysfunction in Senescence-Accelerated Mouse Prone 8.”, J Alzheimers Dis, vol. 59, no. 1, pp. 313-328, 2017.
R. S. Doody, Thomas, R. G., Farlow, M., Iwatsubo, T., Vellas, B., Joffe, S., Kieburtz, K., Raman, R., Sun, X., Aisen, P. S., Siemers, E., Liu-Seifert, H., and Mohs, R., “Phase 3 trials of solanezumab for mild-to-moderate Alzheimer's disease.”, N Engl J Med, vol. 370, no. 4, pp. 311-21, 2014.
S. Hilal, Akoudad, S., van Duijn, C. M., Niessen, W. J., Verbeek, M. M., Vanderstichele, H., Stoops, E., M Ikram, A., and Vernooij, M. W., “Plasma Amyloid-β Levels, Cerebral Small Vessel Disease, and Cognition: The Rotterdam Study.”, J Alzheimers Dis, vol. 60, no. 3, pp. 977-987, 2017.
T. Nakamura, Kawarabayashi, T., Ueda, T., Shimomura, S., Hoshino, M., Itoh, K., Ihara, K., Nakaji, S., Takatama, M., Ikeda, Y., and Shoji, M., “Plasma ApoE4 Levels Are Lower than ApoE2 and ApoE3 Levels, and Not Associated with Plasma Aβ 40/42 Ratio as a Biomarker of Amyloid-β Amyloidosis in Alzheimer's Disease.”, J Alzheimers Dis, vol. 93, no. 1, pp. 333-348, 2023.
T. Nakamura, Kawarabayashi, T., Ueda, T., Shimomura, S., Hoshino, M., Itoh, K., Ihara, K., Nakaji, S., Takatama, M., Ikeda, Y., and Shoji, M., “Plasma ApoE4 Levels Are Lower than ApoE2 and ApoE3 Levels, and Not Associated with Plasma Aβ 40/42 Ratio as a Biomarker of Amyloid-β Amyloidosis in Alzheimer's Disease.”, J Alzheimers Dis, vol. 93, no. 1, pp. 333-348, 2023.
T. Nakamura, Kawarabayashi, T., Ueda, T., Shimomura, S., Hoshino, M., Itoh, K., Ihara, K., Nakaji, S., Takatama, M., Ikeda, Y., and Shoji, M., “Plasma ApoE4 Levels Are Lower than ApoE2 and ApoE3 Levels, and Not Associated with Plasma Aβ 40/42 Ratio as a Biomarker of Amyloid-β Amyloidosis in Alzheimer's Disease.”, J Alzheimers Dis, vol. 93, no. 1, pp. 333-348, 2023.
P. Chatterjee, Doré, V., Pedrini, S., Krishnadas, N., Thota, R., Bourgeat, P., Ikonomovic, M. D., Rainey-Smith, S. R., Burnham, S. C., Fowler, C., Taddei, K., Mulligan, R., Ames, D., Masters, C. L., Fripp, J., Rowe, C. C., Martins, R. N., and Villemagne, V. L., “Plasma Glial Fibrillary Acidic Protein Is Associated with 18F-SMBT-1 PET: Two Putative Astrocyte Reactivity Biomarkers for Alzheimer's Disease.”, J Alzheimers Dis, vol. 92, no. 2, pp. 615-628, 2023.
A. Pilotto, Imarisio, A., Carrarini, C., Russo, M., Masciocchi, S., Gipponi, S., Cottini, E., Aarsland, D., Zetterberg, H., Ashton, N. J., Hye, A., Bonanni, L., and Padovani, A., “Plasma Neurofilament Light Chain Predicts Cognitive Progression in Prodromal and Clinical Dementia with Lewy Bodies.”, J Alzheimers Dis, vol. 82, no. 3, pp. 913-919, 2021.
M. Hashimoto, Ho, G., Sugama, S., Takenouchi, T., Waragai, M., Sugino, H., Inoue, S., and Masliah, E., “Possible Role of Activin in the Adiponectin Paradox-Induced Progress of Alzheimer's Disease.”, J Alzheimers Dis, vol. 81, no. 2, pp. 451-458, 2021.
D. M. Cairns, Itzhaki, R. F., and Kaplan, D. L., “Potential Involvement of Varicella Zoster Virus in Alzheimer's Disease via Reactivation of Quiescent Herpes Simplex Virus Type 1.”, J Alzheimers Dis, vol. 88, no. 3, pp. 1189-1200, 2022.
A. - V. Idland, Wyller, T. Bruun, Støen, R., Eri, L. Magne, Frihagen, F., Ræder, J., Chaudhry, F. Abbas, Hansson, O., Zetterberg, H., Blennow, K., Bogdanovic, N., Brækhus, A., and Watne, L. Otto, “Preclinical Amyloid-β and Axonal Degeneration Pathology in Delirium.”, J Alzheimers Dis, vol. 55, no. 1, pp. 371-379, 2017.
C. M. Stonnington, Chen, Y., Savage, C. R., Lee, W., Iii, R. J. Bauer, Sharieff, S., Thiyyagura, P., Alexander, G. E., Caselli, R. J., Locke, D. E. C., Reiman, E. M., and Chen, K., “Predicting Imminent Progression to Clinically Significant Memory Decline Using Volumetric MRI and FDG PET.”, J Alzheimers Dis, 2018.
K. Ota, Oishi, N., Ito, K., and Fukuyama, H., “Prediction of Alzheimer's Disease in Amnestic Mild Cognitive Impairment Subtypes: Stratification Based on Imaging Biomarkers.”, J Alzheimers Dis, vol. 52, no. 4, pp. 1385-401, 2016.
R. A. Stern, Tripodis, Y., Baugh, C. M., Fritts, N. G., Martin, B. M., Chaisson, C., Cantu, R. C., Joyce, J. A., Shah, S., Ikezu, T., Zhang, J., Gercel-Taylor, C., and Taylor, D. D., “Preliminary Study of Plasma Exosomal Tau as a Potential Biomarker for Chronic Traumatic Encephalopathy.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1099-109, 2016.
M. Nakajima, Miyajima, M., Ogino, I., Akiba, C., Kawamura, K., Kamohara, C., Fusegi, K., Harada, Y., Hara, T., Sugano, H., Tange, Y., Karagiozov, K., Kasuga, K., Ikeuchi, T., Tokuda, T., and Arai, H., “Preoperative Phosphorylated Tau Concentration in the Cerebrospinal Fluid Can Predict Cognitive Function Three Years after Shunt Surgery in Patients with Idiopathic Normal Pressure Hydrocephalus.”, J Alzheimers Dis, vol. 66, no. 1, pp. 319-331, 2018.
L. Fanning, Ryan-Atwood, T. E., J Bell, S., Meretoja, A., McNamara, K. P., Dārziņš, P., Wong, I. C. K., and Ilomäki, J., “Prevalence, Safety, and Effectiveness of Oral Anticoagulant Use in People with and without Dementia or Cognitive Impairment: A Systematic Review and Meta-Analysis.”, J Alzheimers Dis, vol. 65, no. 2, pp. 489-517, 2018.
N. Baazaoui and Iqbal, K., “Prevention of Amyloid-β and Tau Pathologies, Associated Neurodegeneration, and Cognitive Deficit by Early Treatment with a Neurotrophic Compound.”, J Alzheimers Dis, vol. 58, no. 1, pp. 215-230, 2017.
A. Trebbastoni, D'Antonio, F., de Lena, C., Onesti, E., John, B., and Inghilleri, M., “Primary Progressive Orofacial Apraxia: A Ten-Year Long Follow-Up Case Report.”, J Alzheimers Dis, vol. 54, no. 3, pp. 1039-1045, 2016.
N. Dermody, Hornberger, M., Piguet, O., Hodges, J. R., and Irish, M., “Prospective Memory Impairments in Alzheimer's Disease and Behavioral Variant Frontotemporal Dementia: Clinical and Neural Correlates.”, J Alzheimers Dis, vol. 50, no. 2, pp. 425-41, 2016.
F. Martínez-Sánchez, Meilán, J. José G., Carro, J., and Ivanova, O., “A Prototype for the Voice Analysis Diagnosis of Alzheimer's Disease.”, J Alzheimers Dis, vol. 64, no. 2, pp. 473-481, 2018.

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