Biblio
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Author Title Type Year Filters: First Letter Of Last Name is W [Clear All Filters]
“5-Hydroxymethylcytosine Signatures in Circulating Cell-Free DNA as Diagnostic Biomarkers for Late-Onset Alzheimer's Disease.”, J Alzheimers Dis, vol. 85, no. 2, pp. 573-585, 2022.
, “An epigenetic blockade of cognitive functions in the neurodegenerating brain.”, Nature, vol. 483, no. 7388, pp. 222-6, 2012.
, “Alterations in Acrolein Metabolism Contribute to Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 2, pp. 571-580, 2018.
, “Automatic Subtyping of Individuals with Primary Progressive Aphasia.”, J Alzheimers Dis, vol. 79, no. 3, pp. 1185-1194, 2021.
, “Alterations in Acrolein Metabolism Contribute to Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 2, pp. 571-580, 2018.
, “Gx-50 Inhibits Neuroinflammation via α7 nAChR Activation of the JAK2/STAT3 and PI3K/AKT Pathways.”, J Alzheimers Dis, vol. 50, no. 3, pp. 859-71, 2016.
, “Gx-50 Inhibits Neuroinflammation via α7 nAChR Activation of the JAK2/STAT3 and PI3K/AKT Pathways.”, J Alzheimers Dis, vol. 50, no. 3, pp. 859-71, 2016.
, “Neuronal basis of age-related working memory decline.”, Nature, vol. 476, no. 7359, pp. 210-3, 2011.
, “Neuronal basis of age-related working memory decline.”, Nature, vol. 476, no. 7359, pp. 210-3, 2011.
, “The Latent Dementia Phenotype δ is Associated with Cerebrospinal Fluid Biomarkers of Alzheimer's Disease and Predicts Conversion to Dementia in Subjects with Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 49, no. 2, pp. 547-60, 2016.
, “The Latent Dementia Phenotype δ is Associated with Cerebrospinal Fluid Biomarkers of Alzheimer's Disease and Predicts Conversion to Dementia in Subjects with Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 49, no. 2, pp. 547-60, 2016.
, “The Latent Dementia Phenotype δ is Associated with Cerebrospinal Fluid Biomarkers of Alzheimer's Disease and Predicts Conversion to Dementia in Subjects with Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 49, no. 2, pp. 547-60, 2016.
, “Moderate-to-High Intensity Physical Exercise in Patients with Alzheimer's Disease: A Randomized Controlled Trial.”, J Alzheimers Dis, vol. 50, no. 2, pp. 443-53, 2016.
, “Moderate-to-High Intensity Physical Exercise in Patients with Alzheimer's Disease: A Randomized Controlled Trial.”, J Alzheimers Dis, vol. 50, no. 2, pp. 443-53, 2016.
, “Ten-year effects of the advanced cognitive training for independent and vital elderly cognitive training trial on cognition and everyday functioning in older adults.”, J Am Geriatr Soc, vol. 62, no. 1, pp. 16-24, 2014.
, “Economic Analysis of Formal Care, Informal Care, and Productivity Losses in Primary Care Patients who Screened Positive for Dementia in Germany.”, J Alzheimers Dis, vol. 50, no. 1, pp. 47-59, 2016.
, “What Drives Country Differences in Cost of Alzheimer's Disease? An Explanation from Resource Use in the GERAS Study.”, J Alzheimers Dis, vol. 57, no. 3, pp. 797-812, 2017.
, “The Rationale and Design of the Reducing Pathology in Alzheimer's Disease through Angiotensin TaRgeting (RADAR) Trial.”, J Alzheimers Dis, vol. 61, no. 2, pp. 803-814, 2018.
, “Association Between Serum Vitamins and the Risk of Alzheimer's Disease in Chinese Population.”, J Alzheimers Dis, vol. 85, no. 2, pp. 829-836, 2022.
, “Association Between Serum Vitamins and the Risk of Alzheimer's Disease in Chinese Population.”, J Alzheimers Dis, vol. 85, no. 2, pp. 829-836, 2022.
, “Association Between Serum Vitamins and the Risk of Alzheimer's Disease in Chinese Population.”, J Alzheimers Dis, vol. 85, no. 2, pp. 829-836, 2022.
, “Increase of α-Secretase ADAM10 in Platelets Along Cognitively Healthy Aging.”, J Alzheimers Dis, vol. 50, no. 3, pp. 817-26, 2016.
, “Increase of α-Secretase ADAM10 in Platelets Along Cognitively Healthy Aging.”, J Alzheimers Dis, vol. 50, no. 3, pp. 817-26, 2016.
, “"I Don't Think Of It As An Illness": Illness Representations in Mild to Moderate Dementia.”, J Alzheimers Dis, vol. 51, no. 1, pp. 139-50, 2016.
, “Integrated systems approach identifies genetic nodes and networks in late-onset Alzheimer's disease.”, Cell, vol. 153, no. 3, pp. 707-20, 2013.
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