Biblio
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“δ Scores are Exportable Across Cultural and Linguistic Boundaries.”, J Alzheimers Dis, vol. 49, no. 2, pp. 561-70, 2016.
, “δ Scores are Exportable Across Cultural and Linguistic Boundaries.”, J Alzheimers Dis, vol. 49, no. 2, pp. 561-70, 2016.
, “Why Is Amyloid-β PET Requested After Performing Cerebrospinal Fluid Biomarkers?”, J Alzheimers Dis, vol. 73, no. 2, pp. 559-569, 2019.
, “Voxel-Based Acetylcholinesterase PET Study in Early and Late Onset Alzheimer's Disease.”, J Alzheimers Dis, vol. 62, no. 4, pp. 1539-1548, 2018.
, “Visual Processing during Short-Term Memory Binding in Mild Alzheimer's Disease.”, J Alzheimers Dis, vol. 63, no. 1, pp. 185-194, 2018.
, “Visit-to-Visit Blood Pressure Variability and Alzheimer's Disease: Links and Risks.”, J Alzheimers Dis, vol. 59, no. 2, pp. 515-526, 2017.
, “Vestibular Loss Predicts Poorer Spatial Cognition in Patients with Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 3, pp. 995-1003, 2018.
, “Ventilatory Response to Hypercapnia Predicts Dementia with Lewy Bodies in Late-Onset Major Depressive Disorder.”, J Alzheimers Dis, vol. 50, no. 3, pp. 751-8, 2016.
, “Vascular Burden Score Impacts Cognition Independent of Amyloid PET and MRI Measures of Alzheimer's Disease and Vascular Brain Injury.”, J Alzheimers Dis, vol. 68, no. 1, pp. 187-196, 2019.
, “Vacuolar Protein-Sorting Proteins Are Reduced Even Before Cognitive Decline in a Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 96, no. 3, pp. 1011-1017, 2023.
, “Utility of Molecular and Structural Brain Imaging to Predict Progression from Mild Cognitive Impairment to Dementia.”, J Alzheimers Dis, vol. 60, no. 3, pp. 939-947, 2017.
, “Upregulated Expression of Heparanase and Heparanase 2 in the Brains of Alzheimer's Disease.”, J Alzheimers Dis, vol. 58, no. 1, pp. 185-192, 2017.
, “Underlying Biological Processes in Mild Cognitive Impairment: Amyloidosis Versus Neurodegeneration.”, J Alzheimers Dis, vol. 64, no. s1, pp. S647-S657, 2018.
, “Two-Year Changes in Physical Activity and Concurrent Changes in Cognitive Function in a Cohort of Adults with Metabolic Syndrome.”, J Alzheimers Dis, vol. 95, no. 3, pp. 887-899, 2023.
, “Two Year Outcomes, Cognitive and Behavioral Markers of Decline in Healthy, Cognitively Normal Older Persons with Global Deterioration Scale Stage 2 (Subjective Cognitive Decline with Impairment).”, J Alzheimers Dis, vol. 67, no. 2, pp. 685-705, 2019.
, “Two Novel Mutations in the First Transmembrane Domain of Presenilin1 Cause Young-Onset Alzheimer's Disease.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1035-1041, 2017.
, “Two Novel Mutations in the First Transmembrane Domain of Presenilin1 Cause Young-Onset Alzheimer's Disease.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1035-1041, 2017.
, “TREM1 mRNA Expression in Leukocytes and Cognitive Function in Japanese Patients with Alzheimer's Disease.”, J Alzheimers Dis, vol. 64, no. 4, pp. 1275-1284, 2018.
, “TREM1 mRNA Expression in Leukocytes and Cognitive Function in Japanese Patients with Alzheimer's Disease.”, J Alzheimers Dis, vol. 64, no. 4, pp. 1275-1284, 2018.
, “Traffic-Related Air Pollution as a Risk Factor for Dementia: No Clear Modifying Effects of APOEɛ4 in the Betula Cohort.”, J Alzheimers Dis, vol. 71, no. 3, pp. 733-740, 2019.
, “TOMM40 and APOE Gene Expression and Cognitive Decline in Japanese Alzheimer's Disease Subjects.”, J Alzheimers Dis, vol. 60, no. 3, pp. 1107-1117, 2017.
, “TOMM40 and APOE Gene Expression and Cognitive Decline in Japanese Alzheimer's Disease Subjects.”, J Alzheimers Dis, vol. 60, no. 3, pp. 1107-1117, 2017.
, “Tocotrienol-Rich Fraction Modulates Amyloid Pathology and Improves Cognitive Function in AβPP/PS1 Mice.”, J Alzheimers Dis, vol. 55, no. 2, pp. 597-612, 2017.
, “Three-Repeat Tau with Grain-Like Structures and Distribution in an 83-Year-Old Man.”, J Alzheimers Dis, vol. 58, no. 3, pp. 681-685, 2017.
, “Tau and Amyloid-β Cerebrospinal Fluid Biomarkers have Differential Relationships with Cognition in Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 47, no. 4, pp. 965-75, 2015.
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