Biblio
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“Sex Difference in Aerobic Exercise Efficacy to Improve Cognition in Older Adults with Vascular Cognitive Impairment: Secondary Analysis of a Randomized Controlled Trial.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1397-1410, 2017.
, “Short-Term Response is not Predictive of Long-Term Response to Acetylcholinesterase Inhibitors in Old Age Subjects with Alzheimer's Disease: A "Real World" Study.”, J Alzheimers Dis, vol. 56, no. 1, pp. 239-248, 2017.
, “Sleep-Wake Profile in Dementia with Lewy Bodies, Alzheimer's Disease, and Normal Aging.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1529-1536, 2017.
, “Social Cognition in the Frontal Variant of Alzheimer's Disease: A Case Study.”, J Alzheimers Dis, vol. 55, no. 2, pp. 459-463, 2017.
, “Social Representation of Dementia: An Analysis of 5,792 Consecutive Cases Evaluated in a Memory Clinic.”, J Alzheimers Dis, vol. 58, no. 4, pp. 1099-1108, 2017.
, “Soluble Oligomers Require a Ganglioside to Trigger Neuronal Calcium Overload.”, J Alzheimers Dis, vol. 60, no. 3, pp. 923-938, 2017.
, “Spatial Patterns of Longitudinal Gray Matter Change as Predictors of Concurrent Cognitive Decline in Amyloid Positive Healthy Subjects.”, J Alzheimers Dis, vol. 55, no. 1, pp. 343-358, 2017.
, “Tau Oligomers Associate with Inflammation in the Brain and Retina of Tauopathy Mice and in Neurodegenerative Diseases.”, J Alzheimers Dis, vol. 55, no. 3, pp. 1083-1099, 2017.
, “Trends, Predictors, and Outcomes of Healthcare Resources Used in Patients Hospitalized with Alzheimer's Disease with at Least One Procedure: The Nationwide Inpatient Sample.”, J Alzheimers Dis, vol. 57, no. 3, pp. 813-824, 2017.
, “Using Drugs as Molecular Probes: A Computational Chemical Biology Approach in Neurodegenerative Diseases.”, J Alzheimers Dis, vol. 56, no. 2, pp. 677-686, 2017.
, “Validation by Magnetic Resonance Imaging of the Diagnostic Potential of a Heptapeptide-Functionalized Imaging Probe Targeted to Amyloid-β and Able to Cross the Blood-Brain Barrier.”, J Alzheimers Dis, vol. 60, no. 4, pp. 1547-1565, 2017.
, “Visual Selective Attention Toward Novel Stimuli Predicts Cognitive Decline in Alzheimer's Disease Patients.”, J Alzheimers Dis, vol. 55, no. 4, pp. 1339-1349, 2017.
, “Young to Middle-Aged Dogs with High Amyloid-β Levels in Cerebrospinal Fluid are Impaired on Learning in Standard Cognition tests.”, J Alzheimers Dis, vol. 56, no. 2, pp. 763-774, 2017.
, “Administering Cognitive Tests Through Touch Screen Tablet Devices: Potential Issues.”, J Alzheimers Dis, vol. 54, no. 3, pp. 1169-1182, 2016.
, “Amyloid-β Increases Activity of Proteasomes Capped with 19S and 11S Regulators.”, J Alzheimers Dis, vol. 54, no. 2, pp. 763-76, 2016.
, “Amyloid-β Increases Activity of Proteasomes Capped with 19S and 11S Regulators.”, J Alzheimers Dis, vol. 54, no. 2, pp. 763-76, 2016.
, “Amyloid-β Reduces Exosome Release from Astrocytes by Enhancing JNK Phosphorylation.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1433-41, 2016.
, “Anemia and Mild Cognitive Impairment in the German General Population.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1031-42, 2016.
, “Anemia and Mild Cognitive Impairment in the German General Population.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1031-42, 2016.
, “Apathy and Attentional Biases in Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 3, pp. 837-46, 2016.
, “Assessment and Reporting of Driving Fitness in Patients with Dementia in Clinical Practice: Data from SveDem, the Swedish Dementia Registry.”, J Alzheimers Dis, vol. 53, no. 2, pp. 631-8, 2016.
, “Association of Plasma Aβ40 Peptides, But Not Aβ42, with Coronary Artery Disease and Diabetes Mellitus.”, J Alzheimers Dis, vol. 52, no. 1, pp. 161-9, 2016.
, “AZD3293: A Novel, Orally Active BACE1 Inhibitor with High Potency and Permeability and Markedly Slow Off-Rate Kinetics.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1109-23, 2016.
, “Cerebrospinal Fluid Alzheimer's Disease Biomarkers Across the Spectrum of Lewy Body Diseases: Results from a Large Multicenter Cohort.”, J Alzheimers Dis, vol. 54, no. 1, pp. 287-95, 2016.
, “The Cerebrospinal Fluid Neurogranin/BACE1 Ratio is a Potential Correlate of Cognitive Decline in Alzheimer's Disease.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1523-38, 2016.
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