Biblio
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“Familial Presenilin Mutations and Sporadic Alzheimer's Disease Pathology: Is the Assumption of Biochemical Equivalence Justified?”, J Alzheimers Dis, vol. 50, no. 3, pp. 645-58, 2016.
, “FDG-PET and Neuropsychiatric Symptoms among Cognitively Normal Elderly Persons: The Mayo Clinic Study of Aging.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1609-16, 2016.
, “FDG-PET and Neuropsychiatric Symptoms among Cognitively Normal Elderly Persons: The Mayo Clinic Study of Aging.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1609-16, 2016.
, “Fibrillar Amyloid-β Accumulation Triggers an Inflammatory Mechanism Leading to Hyperphosphorylation of the Carboxyl-Terminal End of Tau Polypeptide in the Hippocampal Formation of the 3×Tg-AD Transgenic Mouse.”, J Alzheimers Dis, vol. 52, no. 1, pp. 243-69, 2016.
, “Fibrillar Amyloid-β Accumulation Triggers an Inflammatory Mechanism Leading to Hyperphosphorylation of the Carboxyl-Terminal End of Tau Polypeptide in the Hippocampal Formation of the 3×Tg-AD Transgenic Mouse.”, J Alzheimers Dis, vol. 52, no. 1, pp. 243-69, 2016.
, “Fine Particulate Matter, Residential Proximity to Major Roads, and Markers of Small Vessel Disease in a Memory Study Population.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1315-23, 2016.
, “Fine Particulate Matter, Residential Proximity to Major Roads, and Markers of Small Vessel Disease in a Memory Study Population.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1315-23, 2016.
, “Fine Particulate Matter, Residential Proximity to Major Roads, and Markers of Small Vessel Disease in a Memory Study Population.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1315-23, 2016.
, “First Symptoms and Neurocognitive Correlates of Behavioral Variant Frontotemporal Dementia.”, J Alzheimers Dis, vol. 54, no. 3, pp. 957-970, 2016.
, “First Symptoms and Neurocognitive Correlates of Behavioral Variant Frontotemporal Dementia.”, J Alzheimers Dis, vol. 54, no. 3, pp. 957-970, 2016.
, “The Folate-Vitamin B12 Interaction, Low Hemoglobin, and the Mortality Risk from Alzheimer's Disease.”, J Alzheimers Dis, vol. 52, no. 2, pp. 705-12, 2016.
, “The Folate-Vitamin B12 Interaction, Low Hemoglobin, and the Mortality Risk from Alzheimer's Disease.”, J Alzheimers Dis, vol. 52, no. 2, pp. 705-12, 2016.
, “FTIR and Raman Spectroscopy Applied to Dementia Diagnosis Through Analysis of Biological Fluids.”, J Alzheimers Dis, vol. 52, no. 3, pp. 801-12, 2016.
, “Functional Connectivity of Ventral and Dorsal Visual Streams in Posterior Cortical Atrophy.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1119-30, 2016.
, “Functional Connectivity of Ventral and Dorsal Visual Streams in Posterior Cortical Atrophy.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1119-30, 2016.
, “Genetic Counseling and Testing for Alzheimer's Disease and Frontotemporal Lobar Degeneration: An Italian Consensus Protocol.”, J Alzheimers Dis, vol. 51, no. 1, pp. 277-91, 2016.
, “Genetic Influences on Plasma Homocysteine Levels in African Americans and Yoruba Nigerians.”, J Alzheimers Dis, vol. 49, no. 4, pp. 991-1003, 2016.
, “The Genetic Variability of UCP4 Affects the Individual Susceptibility to Late-Onset Alzheimer's Disease and Modifies the Disease's Risk in APOE-ɛ4 Carriers.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1265-74, 2016.
, “The Genetic Variability of UCP4 Affects the Individual Susceptibility to Late-Onset Alzheimer's Disease and Modifies the Disease's Risk in APOE-ɛ4 Carriers.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1265-74, 2016.
, “A Genetic Variant of the Sortilin 1 Gene is Associated with Reduced Risk of Alzheimer's Disease.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1353-63, 2016.
, “Geographical Variation in Antipsychotic Drug Use in Elderly Patients with Dementia: A Nationwide Study.”, J Alzheimers Dis, vol. 54, no. 3, pp. 1183-1192, 2016.
, “Greater than the Sum of Its Parts: δ can be Constructed from Item Level Data.”, J Alzheimers Dis, vol. 49, no. 2, pp. 571-9, 2016.
, “Greater than the Sum of Its Parts: δ can be Constructed from Item Level Data.”, J Alzheimers Dis, vol. 49, no. 2, pp. 571-9, 2016.
, “Gypenoside XVII Enhances Lysosome Biogenesis and Autophagy Flux and Accelerates Autophagic Clearance of Amyloid-β through TFEB Activation.”, J Alzheimers Dis, vol. 52, no. 3, pp. 1135-50, 2016.
, “Halting of Caspase Activity Protects Tau from MC1-Conformational Change and Aggregation.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1521-1538, 2016.
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