Biblio
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“Region-Specific Vulnerability to Oxidative Stress, Neuroinflammation, and Tau Hyperphosphorylation in Experimental Diabetes Mellitus Mice.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1209-24, 2016.
, “Region-Specific Vulnerability to Oxidative Stress, Neuroinflammation, and Tau Hyperphosphorylation in Experimental Diabetes Mellitus Mice.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1209-24, 2016.
, “Regular Benzodiazepine and Z-Substance Use and Risk of Dementia: An Analysis of German Claims Data.”, J Alzheimers Dis, vol. 54, no. 2, pp. 801-8, 2016.
, “Reliability and Validity of a Novel Internet-Based Battery to Assess Mood and Cognitive Function in the Elderly.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1359-1364, 2016.
, “Reliability and Validity of a Novel Internet-Based Battery to Assess Mood and Cognitive Function in the Elderly.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1359-1364, 2016.
, “Resting-State Cardiac Workload is Related to Both Increased Neocortical Aggregation of Amyloid-β and Relative Impairments in Spatial Working Memory in Pre-Clinical Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 1, pp. 127-31, 2016.
, “Resting-State Cardiac Workload is Related to Both Increased Neocortical Aggregation of Amyloid-β and Relative Impairments in Spatial Working Memory in Pre-Clinical Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 1, pp. 127-31, 2016.
, “Reversal of ApoE4-Driven Brain Pathology by Vascular Endothelial Growth Factor Treatment.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1443-58, 2016.
, “Reversal of LTP-Like Cortical Plasticity in Alzheimer's Disease Patients with Tau-Related Faster Clinical Progression.”, J Alzheimers Dis, vol. 50, no. 2, pp. 605-16, 2016.
, “Reversible Mild Cognitive Impairment: The Role of Comorbidities at Baseline Evaluation.”, J Alzheimers Dis, vol. 51, no. 1, pp. 57-67, 2016.
, “Reversible Mild Cognitive Impairment: The Role of Comorbidities at Baseline Evaluation.”, J Alzheimers Dis, vol. 51, no. 1, pp. 57-67, 2016.
, “Salidroside, a Bioactive Compound of Rhodiola Rosea, Ameliorates Memory and Emotional Behavior in Adult Mice.”, J Alzheimers Dis, vol. 52, no. 1, pp. 65-75, 2016.
, “Screening for Mild Cognitive Impairment: Comparison of "MCI Specific" Screening Instruments.”, J Alzheimers Dis, vol. 51, no. 2, pp. 619-29, 2016.
, “Searching for Primary Predictors of Conversion from Mild Cognitive Impairment to Alzheimer's Disease: A Multivariate Follow-Up Study.”, J Alzheimers Dis, vol. 52, no. 1, pp. 133-43, 2016.
, “Searching for Primary Predictors of Conversion from Mild Cognitive Impairment to Alzheimer's Disease: A Multivariate Follow-Up Study.”, J Alzheimers Dis, vol. 52, no. 1, pp. 133-43, 2016.
, “Serotonin 2A Receptor Inverse Agonist as a Treatment for Parkinson's Disease Psychosis: A Systematic Review and Meta-analysis of Serotonin 2A Receptor Negative Modulators.”, J Alzheimers Dis, vol. 50, no. 3, pp. 733-40, 2016.
, “Serum Adiponectin Levels, Neuroimaging, and Cognition in the Mayo Clinic Study of Aging.”, J Alzheimers Dis, vol. 53, no. 2, pp. 573-81, 2016.
, “Severe Brain Metabolic Decreases Associated with REM Sleep Behavior Disorder in Dementia with Lewy Bodies.”, J Alzheimers Dis, vol. 52, no. 3, pp. 989-97, 2016.
, “Severe Brain Metabolic Decreases Associated with REM Sleep Behavior Disorder in Dementia with Lewy Bodies.”, J Alzheimers Dis, vol. 52, no. 3, pp. 989-97, 2016.
, “So Close Yet So Far: Executive Contribution to Memory Processing in Behavioral Variant Frontotemporal Dementia.”, J Alzheimers Dis, vol. 54, no. 3, pp. 1005-1014, 2016.
, “The Societal Cost of Dementia in Singapore: Results from the WiSE Study.”, J Alzheimers Dis, vol. 51, no. 2, pp. 439-49, 2016.
, “Spatial Memory Impairment is Associated with Intraneural Amyloid-β Immunoreactivity and Dysfunctional Arc Expression in the Hippocampal-CA3 Region of a Transgenic Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 1, pp. 69-79, 2016.
, “Spatial Navigation in Preclinical Alzheimer's Disease.”, J Alzheimers Dis, vol. 52, no. 1, pp. 77-90, 2016.
, “Specific Features of Subjective Cognitive Decline Predict Faster Conversion to Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 52, no. 1, pp. 271-81, 2016.
, “Specific Features of Subjective Cognitive Decline Predict Faster Conversion to Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 52, no. 1, pp. 271-81, 2016.
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