Biblio
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“Cohort Effects in the Prevalence and Survival of People with Dementia in a Rural Area in Northern Sweden.”, J Alzheimers Dis, vol. 50, no. 2, pp. 387-96, 2016.
, “Cohort Study on Predictors of Need for Nursing Care in Alzheimer's Disease: An Analysis of Healthcare Data.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1365-1372, 2016.
, “Combination of Structural MRI and FDG-PET of the Brain Improves Diagnostic Accuracy in Newly Manifested Cognitive Impairment in Geriatric Inpatients.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1319-1331, 2016.
, “Community-Dwelling People Screened Positive for Dementia in Primary Care: A Comprehensive, Multivariate Descriptive Analysis Using Data from the DelpHi-Study.”, J Alzheimers Dis, vol. 52, no. 2, pp. 609-17, 2016.
, “Comparison of Different Matrices as Potential Quality Control Samples for Neurochemical Dementia Diagnostics.”, J Alzheimers Dis, vol. 52, no. 1, pp. 51-64, 2016.
, “Comparison of Different Matrices as Potential Quality Control Samples for Neurochemical Dementia Diagnostics.”, J Alzheimers Dis, vol. 52, no. 1, pp. 51-64, 2016.
, “Comparison of Prefrontal Atrophy and Episodic Memory Performance in Dysexecutive Alzheimer's Disease and Behavioral-Variant Frontotemporal Dementia.”, J Alzheimers Dis, vol. 51, no. 3, pp. 889-903, 2016.
, “Concanavalin agglutinin levels are decreased in peripheral blood of Alzheimer's disease patients.”, J Alzheimers Dis, vol. 49, no. 1, pp. 63-72, 2016.
, “Concanavalin agglutinin levels are decreased in peripheral blood of Alzheimer's disease patients.”, J Alzheimers Dis, vol. 49, no. 1, pp. 63-72, 2016.
, “Cortical Amyloid Burden Differences Across Empirically-Derived Mild Cognitive Impairment Subtypes and Interaction with APOE ɛ4 Genotype.”, J Alzheimers Dis, vol. 52, no. 3, pp. 849-61, 2016.
, “Cost Related to Dementia in the Young and the Impact of Etiological Subtype on Cost.”, J Alzheimers Dis, vol. 49, no. 2, pp. 277-85, 2016.
, “Cost Related to Dementia in the Young and the Impact of Etiological Subtype on Cost.”, J Alzheimers Dis, vol. 49, no. 2, pp. 277-85, 2016.
, “The Cytome Assay as a Tool to Investigate the Possible Association Between Exposure to Extremely Low Frequency Magnetic Fields and an Increased Risk for Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 3, pp. 741-9, 2016.
, “Decreased Levels of VAMP2 and Monomeric Alpha-Synuclein Correlate with Duration of Dementia.”, J Alzheimers Dis, vol. 50, no. 1, pp. 101-10, 2016.
, “Dementia Apraxia Test (DATE): A Brief Tool to Differentiate Behavioral Variant Frontotemporal Dementia from Alzheimer's Dementia Based on Apraxia Profiles.”, J Alzheimers Dis, vol. 49, no. 3, pp. 593-605, 2016.
, “Dementia Risk and Protective Factors Differ in the Context of Memory Trajectory Groups.”, J Alzheimers Dis, vol. 52, no. 3, pp. 1013-20, 2016.
, “A Depressive Endophenotype for Predicting Cognitive Decline among Mexican American Adults and Elders.”, J Alzheimers Dis, vol. 54, no. 1, pp. 201-6, 2016.
, “Detection of Alzheimer's Disease by Three-Dimensional Displacement Field Estimation in Structural Magnetic Resonance Imaging.”, J Alzheimers Dis, vol. 50, no. 1, pp. 233-48, 2016.
, “Diagnosing Dementia in the Clinical Setting: Can Amyloid PET Provide Additional Value Over Cerebrospinal Fluid?”, J Alzheimers Dis, vol. 54, no. 4, pp. 1297-1302, 2016.
, “Diagnosing Dementia in the Clinical Setting: Can Amyloid PET Provide Additional Value Over Cerebrospinal Fluid?”, J Alzheimers Dis, vol. 54, no. 4, pp. 1297-1302, 2016.
, “Diagnostic Accuracy of MRI and Additional [18F]FDG-PET for Behavioral Variant Frontotemporal Dementia in Patients with Late Onset Behavioral Changes.”, J Alzheimers Dis, vol. 53, no. 4, pp. 1287-97, 2016.
, “Differential Diagnosis of Dementia with High Levels of Cerebrospinal Fluid Tau Protein.”, J Alzheimers Dis, vol. 51, no. 3, pp. 905-13, 2016.
, “Differential Effects of APOE Genotypes on the Anterior and Posterior Subnetworks of Default Mode Network in Amnestic Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1409-1423, 2016.
, “Differential Regulation of N-Methyl-D-Aspartate Receptor Subunits is an Early Event in the Actions of Soluble Amyloid-β(1-40) Oligomers on Hippocampal Neurons.”, J Alzheimers Dis, vol. 51, no. 1, pp. 197-212, 2016.
, “Differential Risk of Incident Alzheimer's Disease Dementia in Stable Versus Unstable Patterns of Subjective Cognitive Decline.”, J Alzheimers Dis, vol. 54, no. 3, pp. 1135-1146, 2016.
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