Biblio
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“Exercise Training and Functional Connectivity Changes in Mild Cognitive Impairment and Healthy Elders.”, J Alzheimers Dis, vol. 57, no. 3, pp. 845-856, 2017.
, “Family History of Alzheimer's Disease is Associated with Impaired Perceptual Discrimination of Novel Objects.”, J Alzheimers Dis, vol. 57, no. 3, pp. 735-745, 2017.
, “Humanin Specifically Interacts with Amyloid-β Oligomers and Counteracts Their in vivo Toxicity.”, J Alzheimers Dis, vol. 57, no. 3, pp. 857-871, 2017.
, “Humanin Specifically Interacts with Amyloid-β Oligomers and Counteracts Their in vivo Toxicity.”, J Alzheimers Dis, vol. 57, no. 3, pp. 857-871, 2017.
, “Humanin Specifically Interacts with Amyloid-β Oligomers and Counteracts Their in vivo Toxicity.”, J Alzheimers Dis, vol. 57, no. 3, pp. 857-871, 2017.
, “Long-Term Cognitive Decline in Dementia with Lewy Bodies in a Large Multicenter, International Cohort.”, J Alzheimers Dis, vol. 57, no. 3, pp. 787-795, 2017.
, “Long-Term Cognitive Decline in Dementia with Lewy Bodies in a Large Multicenter, International Cohort.”, J Alzheimers Dis, vol. 57, no. 3, pp. 787-795, 2017.
, “Modeling the Relationships Among Late-Life Body Mass Index, Cerebrovascular Disease, and Alzheimer's Disease Neuropathology in an Autopsy Sample of 1,421 Subjects from the National Alzheimer's Coordinating Center Data Set.”, J Alzheimers Dis, vol. 57, no. 3, pp. 953-968, 2017.
, “Modulating the Balance of Synaptic and Extrasynaptic NMDA Receptors Shows Positive Effects against Amyloid-β-Induced Neurotoxicity.”, J Alzheimers Dis, vol. 57, no. 3, pp. 885-897, 2017.
, “Palmitate Increases β-site AβPP-Cleavage Enzyme 1 Activity and Amyloid-β Genesis by Evoking Endoplasmic Reticulum Stress and Subsequent C/EBP Homologous Protein Activation.”, J Alzheimers Dis, vol. 57, no. 3, pp. 907-925, 2017.
, “PCSK9 Promotes oxLDL-Induced PC12 Cell Apoptosis Through the Bcl-2/Bax-Caspase 9/3 Signaling Pathway.”, J Alzheimers Dis, vol. 57, no. 3, pp. 723-734, 2017.
, “PCSK9 Promotes oxLDL-Induced PC12 Cell Apoptosis Through the Bcl-2/Bax-Caspase 9/3 Signaling Pathway.”, J Alzheimers Dis, vol. 57, no. 3, pp. 723-734, 2017.
, “The Progression of Alzheimer's Disease: Are Fast Decliners Really Fast? A Four-Year Follow-Up.”, J Alzheimers Dis, vol. 57, no. 3, pp. 775-786, 2017.
, “Role of Suppressor of Cytokine Signaling 3 (SOCS3) in Altering Activated Microglia Phenotype in APPswe/PS1dE9 Mice.”, J Alzheimers Dis, vol. 55, no. 3, pp. 1235-1247, 2017.
, “What Drives Country Differences in Cost of Alzheimer's Disease? An Explanation from Resource Use in the GERAS Study.”, J Alzheimers Dis, vol. 57, no. 3, pp. 797-812, 2017.
, “What Drives Country Differences in Cost of Alzheimer's Disease? An Explanation from Resource Use in the GERAS Study.”, J Alzheimers Dis, vol. 57, no. 3, pp. 797-812, 2017.
, “What Drives Country Differences in Cost of Alzheimer's Disease? An Explanation from Resource Use in the GERAS Study.”, J Alzheimers Dis, vol. 57, no. 3, pp. 797-812, 2017.
, “Aberrant Spontaneous Brain Activity in Patients with Mild Cognitive Impairment and concomitant Lacunar Infarction: A Resting-State Functional MRI Study.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1243-54, 2016.
, “Accurate Prediction of Conversion to Alzheimer's Disease using Imaging, Genetic, and Neuropsychological Biomarkers.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1143-59, 2016.
, “Activities of Daily Living and Depressive Symptoms in Patients with Subjective Cognitive Decline, Mild Cognitive Impairment, and Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1043-50, 2016.
, “Age and its association with low insulin and high amyloid-β peptides in blood.”, J Alzheimers Dis, vol. 49, no. 1, pp. 129-37, 2016.
, “Age and its association with low insulin and high amyloid-β peptides in blood.”, J Alzheimers Dis, vol. 49, no. 1, pp. 129-37, 2016.
, “Age-Dependent Regulation of the Blood-Brain Barrier Influx/Efflux Equilibrium of Amyloid-β Peptide in a Mouse Model of Alzheimer's Disease (3xTg-AD).”, J Alzheimers Dis, vol. 49, no. 2, pp. 287-300, 2016.
, “Age-Dependent Regulation of the Blood-Brain Barrier Influx/Efflux Equilibrium of Amyloid-β Peptide in a Mouse Model of Alzheimer's Disease (3xTg-AD).”, J Alzheimers Dis, vol. 49, no. 2, pp. 287-300, 2016.
, “Aging as a Precipitating Factor in Chronic Restraint Stress-Induced Tau Aggregation Pathology, and the Protective Effects of Rosmarinic Acid.”, J Alzheimers Dis, vol. 49, no. 3, pp. 829-44, 2016.
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