Biblio
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“Inhibition of Cholesterol Biosynthesis Reduces γ-Secretase Activity and Amyloid-β Generation.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1057-68, 2016.
, “Inhibition of Cholesterol Biosynthesis Reduces γ-Secretase Activity and Amyloid-β Generation.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1057-68, 2016.
, “Inhibition of Cholesterol Biosynthesis Reduces γ-Secretase Activity and Amyloid-β Generation.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1057-68, 2016.
, “Integrating Biomarkers for Underlying Alzheimer's Disease in Mild Cognitive Impairment in Daily Practice: Comparison of a Clinical Decision Support System with Individual Biomarkers.”, J Alzheimers Dis, vol. 50, no. 1, pp. 261-70, 2016.
, “Integrating Biomarkers for Underlying Alzheimer's Disease in Mild Cognitive Impairment in Daily Practice: Comparison of a Clinical Decision Support System with Individual Biomarkers.”, J Alzheimers Dis, vol. 50, no. 1, pp. 261-70, 2016.
, “Intranasal TAT-haFGF Improves Cognition and Amyloid-β Pathology in an AβPP/PS1 Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 4, pp. 985-90, 2016.
, “Iron Regulates Apolipoprotein E Expression and Secretion in Neurons and Astrocytes.”, J Alzheimers Dis, vol. 51, no. 2, pp. 471-87, 2016.
, “Iron Regulates Apolipoprotein E Expression and Secretion in Neurons and Astrocytes.”, J Alzheimers Dis, vol. 51, no. 2, pp. 471-87, 2016.
, “Iron Regulates Apolipoprotein E Expression and Secretion in Neurons and Astrocytes.”, J Alzheimers Dis, vol. 51, no. 2, pp. 471-87, 2016.
, “Iron Regulates Apolipoprotein E Expression and Secretion in Neurons and Astrocytes.”, J Alzheimers Dis, vol. 51, no. 2, pp. 471-87, 2016.
, “Japanese Care Location and Medical Procedures for People with Dementia in the Last Month of Life.”, J Alzheimers Dis, vol. 51, no. 3, pp. 747-55, 2016.
, “JNK: A Putative Link Between Insulin Signaling and VGLUT1 in Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 4, pp. 963-7, 2016.
, “Lack of evidence for a role of HHV-6 in the pathogenesis of Alzheimer's disease.”, J Alzheimers Dis, vol. 49, no. 1, pp. 229-35, 2016.
, “Lack of evidence for a role of HHV-6 in the pathogenesis of Alzheimer's disease.”, J Alzheimers Dis, vol. 49, no. 1, pp. 229-35, 2016.
, “The Language Profile of Behavioral Variant Frontotemporal Dementia.”, J Alzheimers Dis, vol. 50, no. 2, pp. 359-71, 2016.
, “The Language Profile of Behavioral Variant Frontotemporal Dementia.”, J Alzheimers Dis, vol. 50, no. 2, pp. 359-71, 2016.
, “A Late Life Risk Index for Severe Cognitive Impairment in Mexico.”, J Alzheimers Dis, vol. 52, no. 1, pp. 191-203, 2016.
, “The Latent Dementia Phenotype δ is Associated with Cerebrospinal Fluid Biomarkers of Alzheimer's Disease and Predicts Conversion to Dementia in Subjects with Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 49, no. 2, pp. 547-60, 2016.
, “Late-Onset Metachromatic Leukodystrophy with Early Onset Dementia Associated with a Novel Missense Mutation in the Arylsulfatase A Gene.”, J Alzheimers Dis, vol. 51, no. 3, pp. 683-7, 2016.
, “Lewy Bodies, Vascular Risk Factors, and Subcortical Arteriosclerotic Leukoencephalopathy, but not Alzheimer Pathology, are Associated with Development of Psychosis in Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 1, pp. 283-95, 2016.
, “Lifelong Reading Disorder and Mild Cognitive Impairment: Implications for Diagnosis.”, J Alzheimers Dis, vol. 50, no. 1, pp. 41-5, 2016.
, “Longitudinal Changes in Functional Brain Connectivity Predicts Conversion to Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 2, pp. 377-89, 2016.
, “Longitudinal Changes in Functional Brain Connectivity Predicts Conversion to Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 2, pp. 377-89, 2016.
, “Longitudinal Protein Changes in Blood Plasma Associated with the Rate of Cognitive Decline in Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1105-14, 2016.
, “Longitudinal Protein Changes in Blood Plasma Associated with the Rate of Cognitive Decline in Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1105-14, 2016.
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